Zobrazeno 1 - 10
of 37
pro vyhledávání: '"Sture Sjöblad"'
Autor:
Erkki Pesonen, Sture Sjöblad, Michal Odermarsky, Seppo Ylä-Herttuala, Petru Liuba, Sture Andersson
Publikováno v:
European Journal of Clinical Investigation. 38:381-388
BACKGROUND: Optimal glucose control in juvenile type 1 diabetes mellitus is necessary but not sufficient to reduce the burden of cardiovascular events in later life. This emphasizes the importance of searching for other possible risk factors associat
Publikováno v:
Acta Paediatrica. 64:123-131
Five glycosidases, alpha-fucosidase, alpha-galactosidase, alpha-glucosidase, beta-mannosidase and N-acetyl-alpha-glucosaminidase were studied in human skin fibroblast cultures. The pH-dependency, kinetic properties of the enzymes and results of isoel
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 293:H3175-H3179
Expression of human leukocyte antigen (HLA) class II molecules on islet endothelial cells is a central vascular event in the pathogenesis of Type 1 diabetes. Previous studies demonstrated the ability of other vascular endothelial cells to express HLA
Autor:
Timo Otonkoski, Klaus Brusgaard, Henrik Thybo Christesen, Khalid Hussain, Bendt Brock Jacobsen, Jan Alm, Claus Fenger, Claus Hovendal, Sture Sjöblad, Lars S. Rasmussen
Publikováno v:
Christesen, H T, Brusgaard, K, Alm, J, Sjoblad, S, Hussain, K, Fenger, C, Rasmussen, L, Hovendal, C, Otonkoski, T & Brock-Jacobsen, B 2007, ' Rapid Genetic Analysis in Congenital Hyperinsulinism ', Hormone Research, vol. 67, no. 4, pp. 184-188 . https://doi.org/10.1159/000097063
Backgound: In severe, medically unresponsive congenital hyperinsulinism (CHI), the histological differentiation of focal versus diffuse disease is vital, since the surgical management is completely different. Genetic analysis may help in the differen
Autor:
J. Neiderud, Sture Sjöblad, Sten A. Ivarsson, Bengt Lindberg, Helena Elding-Larsson, K. Larsson, Karin Kockum, Barbro Lernmark, Corrado M. Cilio, Elisabeth Cederwall, Åke Lernmark
Publikováno v:
Diabetes/Metabolism Research and Reviews. 20:429-437
The mechanisms by which gestational infections, blood incompatibility, birth weight, mother's age and other prenatal or neonatal events increase the risk for type 1 diabetes are not understood. Studies so far have been retrospective, and there is a l
Publikováno v:
Pediatric Diabetes. 3:149-154
Results: IAA were detected at a significantly lower frequency (43%; p GAD 65 – islet cell antibodies – protein 0.001) than ICA (86%), GADA (72%), and IA-2 A (80%). In agreement, tyrosine phosphatase IA-2 IAA measurements only marginally increased
Autor:
Sten-Anders Ivarsson, Gunilla Thernlund, Bruno Hägglöf, Sture Sjöblad, Gisela Dahlquist, B Lernmark, Johnny Ludvigsson
Publikováno v:
Acta Paediatrica. 85:947-953
The initial psychological reactions at the onset of insulin-dependent diabetes mellitus (IDDM) in a population-based sample of 76 children were studied with staff observations and a self-report questionnaire for children 12 years of age and more. You
Autor:
Sten A. Ivarsson, Gundlla Thernlund, Bruno Hägglöf, Sture Sjöblad, Johnny Ludvigsson, Gisela Dahlquist
Publikováno v:
Nordic Journal of Psychiatry. 50:199-206
The initial parental reactions at the onset of insulin-dependent diabetes mellitus (IDDM) in children were described by means of a systematized clinical assessment for the staff and a self-report questionnaire for parents in a population-based sample
Publikováno v:
Diabetes Care. 27:2733-2735
Despite improvements in therapy for children with type 1 diabetes, the prevalence of cardiovascular morbidity in adulthood due to accelerated atherosclerosis remains significant (1). Similar to other cardiovascular risk factors, the diabetic state fa
Autor:
Kjell Hansson, Sten A. Ivarsson, Bruno Hägglöf, Sture Sjöblad, Gisela Dahlquist, Gunilla Thernlund, Johnny Ludvigsson
Publikováno v:
Diabetes Care. 18:1323-1329
OBJECTIVE The purpose of the study was to determine whether psychosocial stress during different life periods could be a risk factor in the etiology/pathogenesis of insulin-dependent diabetes mellitus (IDDM) in children. RESEARCH DESIGN AND METHODS I