Zobrazeno 1 - 10
of 189
pro vyhledávání: '"Stuart M Allan"'
Autor:
Isobel C. Mouat, Li Zhu, Alperen Aslan, Barry W. McColl, Stuart M. Allan, Craig J. Smith, Marion S. Buckwalter, Laura McCulloch
Publikováno v:
Journal of Inflammation, Vol 21, Iss 1, Pp 1-10 (2024)
Abstract Background Fatigue is a common complication of stroke that has a significant impact on quality of life. The biological mechanisms that underly post-stroke fatigue are currently unclear, however, reactivation of latent viruses and their impac
Externí odkaz:
https://doaj.org/article/00035330a4e44ca69fd858152c523075
Publikováno v:
Free Neuropathology, Vol 1, Pp 16-16 (2020)
Externí odkaz:
https://doaj.org/article/8607d52f9fb8477c90cd8904860af372
Autor:
Patrick Strangward, Michael J Haley, Tovah N Shaw, Jean-Marc Schwartz, Rachel Greig, Aleksandr Mironov, J Brian de Souza, Sheena M Cruickshank, Alister G Craig, Danny A Milner, Stuart M Allan, Kevin N Couper
Publikováno v:
PLoS Pathogens, Vol 13, Iss 3, p e1006267 (2017)
The murine model of experimental cerebral malaria (ECM) has been utilised extensively in recent years to study the pathogenesis of human cerebral malaria (HCM). However, it has been proposed that the aetiologies of ECM and HCM are distinct, and, cons
Externí odkaz:
https://doaj.org/article/b7f72bd026ef44539bd0bcf64d00434f
Autor:
Kieron South, Lucy Roberts, Anna Gray, Nadim Luka, Patrick Strangward, Graham Coutts, Craig J. Smith, Ingo Schiessl, Stuart M. Allan
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 178, Iss , Pp 117166- (2024)
Recent investigation of a constitutively active ADAMTS13 variant (caADAMTS13) in murine models of acute ischaemic stroke (AIS) have revealed a potential anti-inflammatory mechanism of action contributing to its protective effect. However, it remains
Externí odkaz:
https://doaj.org/article/6b075d79a3a24b2ba65f6c046add9823
Autor:
Tara A. Gleeson, Christina Kaiser, Catherine B. Lawrence, David Brough, Stuart M. Allan, Jack P. Green
Publikováno v:
Disease Models & Mechanisms, Vol 17, Iss 7 (2024)
Externí odkaz:
https://doaj.org/article/35abc22ec05f4cda92c605362d68cc25
Publikováno v:
PLoS ONE, Vol 9, Iss 6, p e99226 (2014)
Alzheimer's disease (AD) is not normally diagnosed until later in life, although evidence suggests that the disease starts at a much earlier age. Risk factors for AD, such as diabetes, hypertension and obesity, are known to have their affects during
Externí odkaz:
https://doaj.org/article/91f055ede04b4f5685d1868c7bb7d15c
Autor:
Adam eDenes, JESUS MIGUEL PRADILLO, Caroline eDrake, Hannah eBuggey, Nancy J Rothwell, Stuart M Allan
Publikováno v:
Frontiers in Neuroscience, Vol 7 (2014)
Acute brain injury results in peripheral inflammatory changes, although the impact of these processes on neuronal death and neuroinflammation is currently unclear. To facilitate the translation of experimental studies to clinical benefit, it is vital
Externí odkaz:
https://doaj.org/article/29d316adf8b048ed8fa1ec85c5ac4fd1
Autor:
Oluwaseun A. Sobowale, Isabel C. Hostettler, Teddy Y. Wu, Calvin Heal, Duncan Wilson, Darshan G. Shah, Daniel Strbian, Jukka Putaala, Turgut Tatlisumak, Andy Vail, Gagan Sharma, Stephen M. Davis, David J. Werring, Atte Meretoja, Stuart M. Allan, Adrian R. Parry-Jones
Publikováno v:
Frontiers in Neurology, Vol 15 (2024)
BackgroundThe relationship between baseline perihematomal edema (PHE) and inflammation, and their impact on survival after intracerebral hemorrhage (ICH) are not well understood.ObjectiveAssess the association between baseline PHE, baseline C-reactiv
Externí odkaz:
https://doaj.org/article/96b76d613e3346df8c8f4fbd97528056
Autor:
José J Rodríguez, Victoria C Jones, Masashi Tabuchi, Stuart M Allan, Elysse M Knight, Frank M LaFerla, Salvatore Oddo, Alexei Verkhratsky
Publikováno v:
PLoS ONE, Vol 3, Iss 8, p e2935 (2008)
It has become generally accepted that new neurones are added and integrated mainly in two areas of the mammalian CNS, the subventricular zone and the subgranular zone (SGZ) of the dentate gyrus (DG) of the hippocampus, which is of central importance
Externí odkaz:
https://doaj.org/article/9633c7e1f4594c43911db1cbfc3dc288
Autor:
Sarah E. Withers, Charlie F. Rowlands, Victor S. Tapia, Frances Hedley, Ioana-Emilia Mosneag, Siobhan Crilly, Gillian I. Rice, Andrew P. Badrock, Andrew Hayes, Stuart M. Allan, Tracy A. Briggs, Paul R. Kasher
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
Aicardi-Goutières syndrome (AGS1-9) is a genetically determined encephalopathy that falls under the type I interferonopathy disease class, characterized by excessive type I interferon (IFN-I) activity, coupled with upregulation of IFN-stimulated gen
Externí odkaz:
https://doaj.org/article/21996985323c40e7b3c222331fcef159