Endothelial Cell Gene Expression of a Neutrophil Chemotactic Factor by TNF-$\alpha $, LPS, and IL-1$\beta $

Autor: Strieter, R. M., Kunkel, S. L., Showell, H. J., Remick, D. G., Phan, S. H., Ward, P. A., Marks, R. M.

Publikováno v: Science, 1989 Mar . 243(4897), 1467-1469.

Externí odkaz: https://www.jstor.org/stable/1703132

IL-4 prevents insulitis and insulin-dependent diabetes mellitus in nonobese diabetic mice by potentiation of regulatory T helper-2 cell function

Autor: Cameron, M. J., Arreaza, G. A., Zucker, P., Chensue, S. W., Strieter, R. M., Subrata Chakrabarti, Delovitch, T. L.

Publikováno v: Scopus-Elsevier

Beginning at the time of insulitis, nonobese diabetic (NOD) mice demonstrate a thymocyte and peripheral T cell proliferative hyporesponsiveness induced by TCR cross-linking, which is associated with reduced IL-2 and IL-4 secretion. We previously repo

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aa859144fa7cc9620c645ca51daaf80e
https://doi.org/10.4049/jimmunol.159.10.4686

Chemokine monocyte chemoattractant protein-1 is expressed by astrocytes after mechanical injury to the brain

Autor: Glabinski, A. R., Balasingam, V., Tani, M., Kunkel, S. L., Strieter, R. M., V. Wee Yong, Ransohoff, R. M.

Publikováno v: Scopus-Elsevier

By 24 h after mechanical trauma to the cerebral cortex, astroglial reaction begins and injury sites are infiltrated by activated mononuclear phagocytes derived from blood-borne monocytes and endogenous microglia. There is little information about cel

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70ad7c85adff29a59fc3f5f18d1c7cf5
https://doi.org/10.4049/jimmunol.156.11.4363

CXCL11 attenuates bleomycin-induced pulmonary fibrosis via inhibition of vascular remodeling

Autor: Burdick, M D, Murray, L A, Keane, M P, Xue, Y Y, Zisman, D A, Belperio, J A, Strieter, R M

Publikováno v: Burdick, M D; Murray, L A; Keane, M P; Xue, Y Y; Zisman, D A; Belperio, J A; et al.(2005). CXCL11 attenuates bleomycin-induced pulmonary fibrosis via inhibition of vascular remodeling. American Journal of Respiratory and Critical Care Medicine, 171(3), 261-268. UCLA: Retrieved from: http://www.escholarship.org/uc/item/60q3f2g1

Aberrant vascular remodeling is a central hallmark for the development and progression of idiopathic pulmonary fibrosis. The mechanisms underlying the pathophysiologic alterations, however, are poorly understood. A recent phase 11 trial of interferon

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______325::a87233734c0b430fd63df5391c691089
http://www.escholarship.org/uc/item/60q3f2g1

Effects of interferon-gamma 1b on biomarker expression in patients with idiopathic pulmonary fibrosis

Autor: Strieter, R M, Starko, K M, Enelow, R I, Noth, I, Valentine, V G

Publikováno v: Strieter, R M; Starko, K M; Enelow, R I; Noth, I; & Valentine, V G. (2004). Effects of interferon-gamma 1b on biomarker expression in patients with idiopathic pulmonary fibrosis. American Journal of Respiratory and Critical Care Medicine, 170(2), 133-140. UCLA: Retrieved from: http://www.escholarship.org/uc/item/6hx534td

In a recent study of IFN-gamma 1b in 330 patients with idiopathic pulmonary fibrosis (IPF), progression-free survival was unchanged; however, a trend toward lower mortality was seen in IFN-gamma 1b-treated patients compared with placebo-treated patie

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______325::a17b97ba1636fa0d7a4a28a8b8a9bd7f
http://www.escholarship.org/uc/item/6hx534td