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Withdrawal: Exercise increases mitochondrial PGC-1α content and promotes nuclear-mitochondrial cross-talk to coordinate mitochondrial biogenesis.

Autor: Safdar A, Little JP, Stokl AJ, Hettinga BP, Akhtar M, Tarnopolsky MA

Publikováno v: The Journal of biological chemistry [J Biol Chem] 2020 Dec 18; Vol. 295 (51), pp. 17888.

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Exercise increases mitochondrial PGC-1 α content and promotes nuclear-mitochondrial cross-talk to coordinate mitochondrial biogenesis.

Autor: Safdar A, Little JP, Stokl AJ, Hettinga BP, Akhtar M, Tarnopolsky MA

Publikováno v: The Journal of biological chemistry [J Biol Chem] 2018 Mar 30; Vol. 293 (13), pp. 4953.

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Exercise increases mitochondrial PGC-1alpha content and promotes nuclear-mitochondrial cross-talk to coordinate mitochondrial biogenesis.

Autor: Safdar A; Department of Kinesiology, McMaster University, Hamilton, Ontario L8N 3Z5, Canada., Little JP, Stokl AJ, Hettinga BP, Akhtar M, Tarnopolsky MA

Publikováno v: The Journal of biological chemistry [J Biol Chem] 2011 Mar 25; Vol. 286 (12), pp. 10605-17. Date of Electronic Publication: 2011 Jan 18.

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Clinical trial in adults of an antimicrobial varnish for reducing mutans streptococci.

Autor: Sandham HJ; Faculty of Dentistry, University of Toronto, Ontario, Canada., Brown J, Chan KH, Phillips HI, Burgess RC, Stokl AJ

Publikováno v: Journal of dental research [J Dent Res] 1991 Nov; Vol. 70 (11), pp. 1401-8.

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Full-length transcriptomic analysis in murine and human heart reveals diversity of PGC-1α promoters and isoforms regulated distinctly in myocardial ischemia and obesity.

Autor: Oehler, Daniel^1,2 (AUTHOR) Daniel.Oehler@med.uni-duesseldorf.de, Spychala, André³ (AUTHOR), Gödecke, Axel^2,3 (AUTHOR), Lang, Alexander^1,2 (AUTHOR), Gerdes, Norbert^1,2 (AUTHOR), Ruas, Jorge⁴ (AUTHOR), Kelm, Malte^1,2 (AUTHOR), Szendroedi, Julia^5,6 (AUTHOR), Westenfeld, Ralf^1,2 (AUTHOR) Ralf.Westenfeld@med.uni-duesseldorf.de

Publikováno v: BMC Biology. 7/30/2022, Vol. 20 Issue 1, p1-20. 20p.

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PGC-1α regulation by FBXW7 through a novel mechanism linking chaperone-mediated autophagy and the ubiquitin-proteasome system.

Autor: Eleuteri S; Department of Neurology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, USA., Wang B; Department of Neurology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, USA.; Department of Neurosurgery, Tangdu Hospital: Fourth Military Medical University, Xi'an, China., Cutillo G; Department of Neurology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, USA., Zhang Fang TS; Department of Neurology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, USA., Tao K; Department of Neurosurgery, Tangdu Hospital: Fourth Military Medical University, Xi'an, China., Qu Y; Department of Neurosurgery, Tangdu Hospital: Fourth Military Medical University, Xi'an, China., Yang Q; Department of Neurosurgery, Tangdu Hospital: Fourth Military Medical University, Xi'an, China., Wei W; Department of Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, USA., Simon DK; Department of Neurology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, USA.

Publikováno v: The FEBS journal [FEBS J] 2024 Oct 21. Date of Electronic Publication: 2024 Oct 21.

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Mitochondria and Parkinson's Disease: Clinical, Molecular, and Translational Aspects.

Autor: Borsche, Max^1,2 (AUTHOR), Pereira, Sandro L.³ (AUTHOR), Klein, Christine¹ (AUTHOR) christine.klein@neuro.uni-luebeck.de, Grünewald, Anne^1,3 (AUTHOR)

Publikováno v: Journal of Parkinson's Disease. 2021, Vol. 11 Issue 1, p45-60. 16p.

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