Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Stine N. Hansen"'
Autor:
Clara Mayer, Lluís Riera-Ponsati, Sakari Kauppinen, Henrik Klitgaard, Janine T. Erler, Stine N. Hansen
Publikováno v:
Frontiers in Pharmacology, Vol 15 (2024)
Neurodegenerative diseases constitute a global health issue and a major economic burden. They significantly impair both cognitive and motor functions, and their prevalence is expected to rise due to ageing societies and continuous population growth.
Externí odkaz:
https://doaj.org/article/ed186951ce8645ee905a86c980cc5ef6
Publikováno v:
RNA Biology, Vol 19, Iss 1, Pp 594-608 (2022)
RNA therapeutics comprise a diverse group of oligonucleotide-based drugs such as antisense oligonucleotides (ASOs), small interfering RNAs (siRNAs), and short hairpin RNAs (shRNAs) that can be designed to selectively interact with drug targets curren
Externí odkaz:
https://doaj.org/article/59b6bf0a675a472594fab016df0d96f8
Autor:
Stine N. Hansen, Jane M. Bjørn Jørgensen, Jens R. Nyengaard, Jens Lykkesfeldt, Pernille Tveden-Nyborg
Publikováno v:
Nutrients, Vol 10, Iss 6, p 749 (2018)
Approximately 15% of the Western world population, including pregnant women and their children, is characterized as vitamin C (vitC) deficient. In guinea pigs, early life vitC deficiency causes spatial memory deficits, decreased hippocampal volume an
Externí odkaz:
https://doaj.org/article/911bde882f4b474ead1abdf5e90fdf2d
Autor:
Carina von Schantz, Catherine Kielar, Stine N. Hansen, Charlie C. Pontikis, Noreen A. Alexander, Outi Kopra, Anu Jalanko, Jonathan D. Cooper
Publikováno v:
Neurobiology of Disease, Vol 34, Iss 2, Pp 308-319 (2009)
Finnish variant LINCL (vLINCLFin) is the result of mutations in the CLN5 gene. To gain insights into the pathological staging of this fatal pediatric disorder, we have undertaken a stereological analysis of the CNS of Cln5 deficient mice (Cln5−/−
Externí odkaz:
https://doaj.org/article/cf7cf862ee644b84883b4ebd8eb3edb1
Publikováno v:
Experimental and clinical psychopharmacology.
Glucagon-like peptide 1 (GLP-1) receptor agonists can decrease alcohol intake by central mechanisms that are still poorly understood. The lateral septum (LS) and the ventral/caudal part of the hippocampus are enriched in GLP-1 receptors, and activity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70baf56a763df83c35cde37a080ff9df
https://pubmed.ncbi.nlm.nih.gov/36480394
https://pubmed.ncbi.nlm.nih.gov/36480394
Autor:
Stine N Hansen, Desiree Gordian, Lotte Bjerre Knudsen, Sarah J Paulsen, Mette Q Ludwig, Martin G. Myers, Christopher J. Rhodes, Tune H. Pers, Wenwen Cheng, Anna Secher, Charles Pyke, Julie Lee, Kristoffer L. Egerod, Pernille Barkholt
Publikováno v:
Nature Metabolism. 3:530-545
The brainstem dorsal vagal complex (DVC) is known to regulate energy balance and is the target of appetite-suppressing hormones, such as glucagon-like peptide 1 (GLP-1). Here we provide a comprehensive genetic map of the DVC and identify neuronal pop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9ef0cddb8b41c7acd82d947677cda716
https://doi.org/10.1038/s42255-021-00363-1
https://doi.org/10.1038/s42255-021-00363-1
Autor:
Mette Q, Ludwig, Wenwen, Cheng, Desiree, Gordian, Julie, Lee, Sarah J, Paulsen, Stine N, Hansen, Kristoffer L, Egerod, Pernille, Barkholt, Christopher J, Rhodes, Anna, Secher, Lotte Bjerre, Knudsen, Charles, Pyke, Martin G, Myers, Tune H, Pers
Publikováno v:
Nature metabolism. 3(4)
The brainstem dorsal vagal complex (DVC) is known to regulate energy balance and is the target of appetite-suppressing hormones, such as glucagon-like peptide 1 (GLP-1). Here we provide a comprehensive genetic map of the DVC and identify neuronal pop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c2eda6be79972a4837e72ed68744e898
https://pubmed.ncbi.nlm.nih.gov/33767443
https://pubmed.ncbi.nlm.nih.gov/33767443
Autor:
Stine N. Hansen, Anu Jalanko, Catherine Kielar, Outi Kopra, Noreen A. Alexander, Jonathan D. Cooper, Charlie C. Pontikis, Carina von Schantz
Publikováno v:
Neurobiology of Disease, Vol 34, Iss 2, Pp 308-319 (2009)
Finnish variant LINCL (vLINCL Fin ) is the result of mutations in the CLN5 gene. To gain insights into the pathological staging of this fatal pediatric disorder, we have undertaken a stereological analysis of the CNS of Cln5 deficient mice ( Cln5 −
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::43ce5ecdce4ca3ce22b0279103ff31e0
http://www.sciencedirect.com/science/article/pii/S096999610900028X
http://www.sciencedirect.com/science/article/pii/S096999610900028X