Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Stijntje Hibender"'
Autor:
Stijntje Hibender, Siyu Li, Alex V Postma, Myrthe E Hoogeland, Denise Klaver, Richard B Pouw, Hans W Niessen, Antoine HG Driessen, David R Koolbergen, Carlie JM de Vries, Marieke JH Baars, Arjan C Houweling, Paul A Krijnen, Vivian de Waard
Publikováno v:
Vascular Biology, Vol 4, Iss 1, Pp 40-49 (2022)
Marfan syndrome (MFS) is a connective tissue disorder causing aortic aneurysm formation. Currently, only prophylactic aortic surgery and blood pressure-lowering drugs are available to reduce the risk of aortic rupture. Upon whole genome sequencing of
Externí odkaz:
https://doaj.org/article/d46834c056ed40f195ae750f5a23a4ec
Autor:
Romy Franken, Stijntje Hibender, Alexander W den Hartog, Teodora Radonic, Carlie J M de Vries, Aeilko H Zwinderman, Maarten Groenink, Barbara J M Mulder, Vivian de Waard
Publikováno v:
PLoS ONE, Vol 9, Iss 9, p e107221 (2014)
AIMS:Patients with Marfan syndrome have an increased risk of life-threatening aortic complications, mostly preceded by aortic dilatation. In the FBN1(C1039G/+) Marfan mouse model, losartan decreases aortic root dilatation. We recently confirmed this
Externí odkaz:
https://doaj.org/article/291beb41452c49dbb62569b3b81539e6
Autor:
Mariska Vos, Vivian de Waard, Ron Balm, Shaynah Wanga, Ingeborg van der Made, Stijntje Hibender, Carlie J.M. de Vries, Barbara J.M. Mulder
Publikováno v:
Cardiovascular pathology, 38, 1-6. Elsevier Inc.
Marfan syndrome (MFS) is a connective tissue disorder caused by mutations in the fibrillin-1 gene (FBN1), resulting in aortic aneurysm formation and dissections. Interestingly, variable aortopathy is observed even within MFS families with the same mu
Autor:
Cindy P. A. A. van Roomen, Luigi Amjg van Riel, Barbara J.M. Mulder, Shaynah Wanga, Ingeborg van der Made, Nicole van Vliet, Aeilko H. Zwinderman, Carlie J.M. de Vries, Romy Franken, Vivian de Waard, Maarten Groenink, Stijntje Hibender, Mariska Vos, Jeroen Essers, Yanto Ridwan
Publikováno v:
The Journal of Pathology. 243:294-306
Marfan syndrome (MFS) is a connective tissue disorder in which aortic rupture is the major cause of death. MFS patients with an aortic diameter below the advised limit for prophylactic surgery (
Autor:
Stijntje Hibender, Barbara J.M. Mulder, Carlie J.M. de Vries, Anique ter Braake, Aeilko H. Zwinderman, Maurice J. van den Hoff, Esther Lutgens, M. Groenink, Edith E. Schermer, Romy Franken, Y. M. Pinto, Cindy van Roomen, Vivian de Waard, Quinn Gunst, Ingeborg van der Made
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology, 36(8), 1618-1626. Lippincott Williams and Wilkins
Arteriosclerosis, Thrombosis, and Vascular Biology, 36, 1618-26
Arteriosclerosis, Thrombosis, and Vascular Biology, 36, 8, pp. 1618-26
Arteriosclerosis, Thrombosis, and Vascular Biology, 36, 1618-26
Arteriosclerosis, Thrombosis, and Vascular Biology, 36, 8, pp. 1618-26
Objective— Marfan syndrome (MFS) is a connective tissue disorder caused by mutations in the fibrillin-1 gene. Patients with MFS are at risk of aortic aneurysm formation and dissection. Usually, blood pressure–lowering drugs are used to reduce aor
Autor:
Stijntje, Hibender, Shaynah, Wanga, Ingeborg, van der Made, Mariska, Vos, Barbara Jm, Mulder, Ron, Balm, Carlie Jm, de Vries, Vivian, de Waard
Publikováno v:
Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology. 38
Marfan syndrome (MFS) is a connective tissue disorder caused by mutations in the fibrillin-1 gene (FBN1), resulting in aortic aneurysm formation and dissections. Interestingly, variable aortopathy is observed even within MFS families with the same mu
Disulfide bonds confer stability and activity to proteins. Bioinformatic approaches allow predictions of which organisms make protein disulfide bonds and in which subcellular compartments disulfide bond formation takes place. Such an analysis, along
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::35f76fca44c15f957d4fb3d623928c91
https://europepmc.org/articles/PMC5845738/
https://europepmc.org/articles/PMC5845738/
Autor:
Shaynah Wanga, Stijntje Hibender, Yanto Ridwan, Cindy van Roomen, Mariska Vos, Ingeborg van der Made, Nicole van Vliet, Romy Franken, Maarten Groenink, Aeilko H Zwinderman, Barbara J Mulder, Carlie J de Vries, Jeroen Essers, Vivian de Waard
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 37
Marfan syndrome (MFS) is a genetic connective tissue disorder, in which aortic rupture is the major cause of death. MFS patients with an aortic diameter below the advised limit for prophylactic surgery (C1039G/+ MFS aortic SMCs, ALP mRNA and activity
Autor:
Shaynah, Wanga, Stijntje, Hibender, Yanto, Ridwan, Cindy, van Roomen, Mariska, Vos, Ingeborg, van der Made, Nicole, van Vliet, Romy, Franken, Luigi Amjg, van Riel, Maarten, Groenink, Aeilko H, Zwinderman, Barbara Jm, Mulder, Carlie Jm, de Vries, Jeroen, Essers, Vivian, de Waard
Publikováno v:
The Journal of pathology. 243(3)
Marfan syndrome (MFS) is a connective tissue disorder in which aortic rupture is the major cause of death. MFS patients with an aortic diameter below the advised limit for prophylactic surgery (5 cm) may unexpectedly experience an aortic dissection o
Autor:
Mark Hoogenboezem, Arginell F. Girigorie, Jaap D. van Buul, Stijntje Hibender, Carlie J.M. de Vries, Vivian de Waard, Goran Marinković, Amber van Broekhoven, Anouk A.J. Hamers, Jan Stap, Natascha Bleeker
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology, 33(10), 2380-2388. Lippincott Williams and Wilkins
Objective— In aortic aneurysms the arterial vessel wall is dilated because of destruction of its integrity, which may lead to lethal vessel rupture. Chronic infiltration of inflammatory cells into the vessel wall is fundamental to aneurysm patholog