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Autor:
Amit Goldberg, Jeffrey H. Lipton, Allan D. Novetsky, Steven Lichter, Jen-Chin Wang, Tsong H. Chang
Publikováno v:
Blood. 108:3607-3607
Currently, the prevailing concept concerning the etiology of bone marrow fibrosis in patients with idiopathic myelofibrosis (IMF) is that it results from excessive production of fibrosing growth factors including transforming growth factor beta (TGF-