Zobrazeno 1 - 10
of 84
pro vyhledávání: '"Steinar Aase"'
Autor:
Steinar Aase, Jan Ivar Pedersen
Publikováno v:
Tidsskrift for Den norske legeforening.
Autor:
Jaran Apold, Ketil Heimdal, Juul T. Wijnen, Elin H. Kure, Inger Marie Bowitz-Lothe, Astrid Stormorken, Tove Norèn, Pål Møller, Steinar Aase
Publikováno v:
Journal of Clinical Oncology. 23:4705-4712
Purpose Hereditary nonpolyposis colorectal cancer (HNPCC) may be caused by mutations in mismatch repair (MMR) genes. The aim of this study was to validate immunohistochemistry and family history as prescreening tools to predict germline mutations in
Autor:
Jan Thomas Lien, Anne Marie Gabrielsen, Steinar Aase, Janna Berg, Martin Veel Svendsen, Torunn Helene Søland, Gerhardt Hauss
Publikováno v:
APMIS. 113:208-212
In order to elucidate the relative contributions made by cytology and histology in the diagnosis of lung cancer, we studied the cytology and histology reports of all patients who received a microscopic diagnosis of lung cancer in our hospital during
Autor:
Gabriela Möslein, Steinar Aase, Ketil Heimdal, Wolfram Müller, Pål Møller, Juul T. Wijnen, Astrid Stormorken, Annika Lindblom, Tove Norèn, Inger Marie Bowitz Lothe
Publikováno v:
Familial Cancer. 2:9-13
Hereditary nonpolyposis colorectal cancer (HNPCC) may be caused by germline truncating mutations in DNA mismatch repair (MMR) genes. Whether or not missense or inframe mutations are disease-associated has become a practical clinical problem, because
Autor:
Steinar Aase, Per Ove Hagestuen
Publikováno v:
Tidsskrift for Den norske legeforening. 132:152-154
BACKGROUND There has been a considerable reduction in the frequency of autopsies over the past few decades. We wanted to investigate whether doctors consider this a proper and natural development, or whether they consider that autopsies should be giv
Publikováno v:
Japanese Journal of Cancer Research : Gann
Duodenogastric reflux is a risk factor for gastric carcinogenesis, but the pathogenesis is not fully understood. We studied the risk of N-methyl-N-nitro-N-nitrosoguanidine (MNNG)-induced carcinogenesis in the antrum of rats with duodenogastric reflux
Publikováno v:
Digestive Diseases and Sciences. 47:2354-2369
The mucosal changes by which duodenogastric reflux may predispose to gastric cancer have not been fully clarified. In this study in rats, duodenal fluid was directed into the stomach through a gastroenterostomy (jejunal reflux, N = 29) or through the
Publikováno v:
The Histochemical Journal. 32:551-556
Neuroendocrine cells are often disclosed in human gastric adenocarcinomas and may be recognised by their immunoreactivity towards chromogranin A. However, in dedifferentiated neuroendocrine tumour cells, the chromogranin A content may be reduced maki
Publikováno v:
Alimentary Pharmacology & Therapeutics. 14:135-141
Background: Gastric ischaemia appears to be a common pathogenetic factor for stress ulcers. These ulcers occur predominantly in the oxyntic mucosa, suggesting that the acid secretory process or its stimulation is involved in the pathogenesis. Methods
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 277:G1064-G1073
Calcitonin gene-related peptide (CGRP) released from sensory neurons, which are closely apposed to mast cells and blood vessels, mediates gastric hyperemia in response to acid challenge of the damaged mucosa. Substance P (SP) is coreleased with CGRP