Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Stefan Flaschberger"'
Autor:
Nóra Garam, Zoltán Prohászka, Ágnes Szilágyi, Christof Aigner, Alice Schmidt, Martina Gaggl, Gere Sunder-Plassmann, Dóra Bajcsi, Jürgen Brunner, Alexandra Dumfarth, Daniel Cejka, Stefan Flaschberger, Hana Flögelova, Ágnes Haris, Ágnes Hartmann, Andreas Heilos, Thomas Mueller, Krisztina Rusai, Klaus Arbeiter, Johannes Hofer, Dániel Jakab, Mária Sinkó, Erika Szigeti, Csaba Bereczki, Viktor Janko, Kata Kelen, György S. Reusz, Attila J. Szabó, Nóra Klenk, Krisztina Kóbor, Nika Kojc, Maarten Knechtelsdorfer, Mario Laganovic, Adrian Catalin Lungu, Anamarija Meglic, Rina Rus, Tanja Kersnik-Levart, Ernesta Macioniene, Marius Miglinas, Anna Pawłowska, Tomasz Stompór, Ludmila Podracka, Michael Rudnicki, Gert Mayer, Romana Rysava, Jana Reiterova, Marijan Saraga, Tomáš Seeman, Jakub Zieg, Eva Sládková, Tamás Szabó, Andrei Capitanescu, Simona Stancu, Miroslav Tisljar, Kresimir Galesic, András Tislér, Inga Vainumäe, Martin Windpessl, Tomas Zaoral, Galia Zlatanova, Dorottya Csuka
Publikováno v:
Orphanet Journal of Rare Diseases, Vol 14, Iss 1, Pp 1-14 (2019)
Abstract Background Acquired or genetic abnormalities of the complement alternative pathway are the primary cause of C3glomerulopathy(C3G) but may occur in immune-complex-mediated membranoproliferative glomerulonephritis (IC-MPGN) as well. Less is kn
Externí odkaz:
https://doaj.org/article/69c3689758b34c40a3dddf8458dfe664
Autor:
Nóra Garam, Marcell Cserhalmi, Zoltán Prohászka, Ágnes Szilágyi, Nóra Veszeli, Edina Szabó, Barbara Uzonyi, Attila Iliás, Christof Aigner, Alice Schmidt, Martina Gaggl, Gere Sunder-Plassmann, Dóra Bajcsi, Jürgen Brunner, Alexandra Dumfarth, Daniel Cejka, Stefan Flaschberger, Hana Flögelova, Ágnes Haris, Ágnes Hartmann, Andreas Heilos, Thomas Mueller, Krisztina Rusai, Klaus Arbeiter, Johannes Hofer, Dániel Jakab, Mária Sinkó, Erika Szigeti, Csaba Bereczki, Viktor Janko, Kata Kelen, György S. Reusz, Attila J. Szabó, Nóra Klenk, Krisztina Kóbor, Nika Kojc, Maarten Knechtelsdorfer, Mario Laganovic, Adrian Catalin Lungu, Anamarija Meglic, Rina Rus, Tanja Kersnik Levart, Ernesta Macioniene, Marius Miglinas, Anna Pawłowska, Tomasz Stompór, Ludmila Podracka, Michael Rudnicki, Gert Mayer, Romana Rysava, Jana Reiterova, Marijan Saraga, Tomáš Seeman, Jakub Zieg, Eva Sládková, Natasa Stajic, Tamás Szabó, Andrei Capitanescu, Simona Stancu, Miroslav Tisljar, Kresimir Galesic, András Tislér, Inga Vainumäe, Martin Windpessl, Tomas Zaoral, Galia Zlatanova, Mihály Józsi, Dorottya Csuka
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
BackgroundFactor H-related protein 5 (FHR-5) is a member of the complement Factor H protein family. Due to the homology to Factor H, the main complement regulator of the alternative pathway, it may also be implicated in the pathomechanism of kidney d
Externí odkaz:
https://doaj.org/article/0c908e2545e94fcd87521ab9c91fb160
Autor:
Attila Szabo, Barbara Uzonyi, Ágnes Szilágyi, Krešimir Galešić, Dóra Bajcsi, Gere Sunder-Plassmann, Mária Sinkó, Ernesta Macioniene, Anna Pawłowska, Nóra Klenk, Johannes Hofer, Marcell Cserhalmi, Klaus Arbeiter, Tanja Kersnik-Levart, Marius Miglinas, Dorottya Csuka, Ágnes Hartmann, Galia Zlatanova, Kata Kelen, Tomáš Seeman, Andreas Heilos, Marijan Saraga, Eva Sládková, Krisztina Rusai, Gert Mayer, Martin Windpessl, Erika Szigeti, Jana Reiterova, Ludmila Podracka, Nóra Veszeli, Edina Szabó, Martina Gaggl, Mario Laganović, Tamás Szabó, Tomasz Stompór, Stefan Flaschberger, Daniel Cejka, Rina Rus, Nika Kojc, Maarten Knechtelsdorfer, Nóra Garam, Miroslav Tisljar, András Tislér, Michael A. Rudnicki, Christof Aigner, Ágnes Haris, Natasa Stajic, Anamarija Meglic, Hana Flögelová, Jürgen Brunner, Simona Stancu, György Reusz, Attila Iliás, Jakub Zieg, Adrian Catalin Lungu, Viktor Janko, Thomas Mueller, Dániel Jakab, Inga Vainumäe, Krisztina Kóbor, Tomas Zaoral, Mihály Józsi, Csaba Bereczki, Romana Rysava, Alice Schmidt, Andrei Capitanescu, Zoltán Prohászka, Alexandra Dumfarth
Background: Factor H-related-5 (FHR-5) is a member of the complement Factor H protein family. Due to the homology to Factor H, the main complement regulator of the alternative pathway, it may also be implicated in pathomechanism of kidney diseases wh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3aa783b1d7de2a6da574f817148d4831
https://doi.org/10.21203/rs.3.rs-32618/v1
https://doi.org/10.21203/rs.3.rs-32618/v1
Autor:
Dániel Jakab, Ágnes Szilágyi, Gert Mayer, Nóra Garam, Miroslav Tisljar, Viktor Janko, Attila Szabo, Inga Vainumäe, Christof Aigner, Ágnes Hartmann, Simona Stancu, Galia Zlatanova, Tamás Szabó, Marius Miglinas, András Tislér, Krisztina Kóbor, Tomáš Seeman, Mária Sinkó, Alice Schmidt, Stefan Flaschberger, Hana Flögelová, Nika Kojc, Klaus Arbeiter, Jana Reiterova, Tomas Zaoral, Michael A. Rudnicki, Gere Sunder-Plassmann, Romana Rysava, Jürgen Brunner, Maarten Knechtelsdorfer, Daniel Cejka, Ágnes Haris, György Reusz, Krešimir Galešić, Mario Laganović, Tomasz Stompór, Anna Pawłowska, Anamarija Meglic, Eva Sládková, Dorottya Csuka, Andrei Capitanescu, Tanja Kersnik-Levart, Zoltán Prohászka, Rina Rus, Adrian Catalin Lungu, Csaba Bereczki, Marijan Saraga, Thomas Mueller, Ernesta Macioniene, Andreas Heilos, Krisztina Rusai, Kata Kelen, Erika Szigeti, Alexandra Dumfarth, Dóra Bajcsi, Ludmila Podracka, Martina Gaggl, Jakub Zieg, Nóra Klenk, Johannes Hofer, Martin Windpessl
Publikováno v:
Clinical kidney journal, Oxford : Oxford University Press, 2020, vol. 13, no. 2, p. 225-234
Clinical Kidney Journal
Clinical Kidney Journal
Background A novel data-driven cluster analysis identified distinct pathogenic patterns in C3-glomerulopathies and immune complex-mediated membranoproliferative glomerulonephritis. Our aim was to replicate these observations in an independent cohort
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::91ee3af43c8f066fdd8352003d651df6
https://www.bib.irb.hr/1259464
https://www.bib.irb.hr/1259464
Autor:
Ágnes Szilágyi, Christof Aigner, Attila Szabo, Tanja Kersnik-Levart, Dorottya Csuka, Ágnes Haris, Alice Schmidt, Rina Rus, Stefan Flaschberger, Ernesta Macioniene, Hana Flögelová, Anamarija Meglic, Ludmila Podracka, Gere Sunder-Plassmann, Alexandra Dumfarth, Martina Gaggl, Andrei Capitanescu, Viktor Janko, Anna Pawłowska, Gert Mayer, Thomas Mueller, Ágnes Hartmann, András Tislér, Kata Kelen, Erika Szigeti, Galia Zlatanova, Martin Windpessl, Tamás Szabó, Klaus Arbeiter, Mario Laganović, György Reusz, Romana Rysava, Krisztina Kóbor, Jakub Zieg, Daniel Cejka, Michael A. Rudnicki, Andreas Heilos, Krisztina Rusai, Dániel Jakab, Tomas Zaoral, Nóra Klenk, Johannes Hofer, Nóra Garam, Krešimir Galešić, Jürgen Brunner, Miroslav Tisljar, Marius Miglinas, Mária Sinkó, Csaba Bereczki, Inga Vainumäe, Nika Kojc, Marijan Saraga, Tomáš Seeman, Simona Stancu, Eva Sládková, Jana Reiterova, Zoltán Prohászka, Maarten Knechtelsdorfer, Adrian Catalin Lungu, Tomasz Stompór, Dóra Bajcsi
Publikováno v:
Orphanet journal of rare diseases, London : BioMed Central Ltd., 2019, vol. 14, art. no. 247, p. [1-14]
Orphanet Journal of Rare Diseases, Vol 14, Iss 1, Pp 1-14 (2019)
Orphanet Journal of Rare Diseases
Orphanet Journal of Rare Diseases, Vol 14, Iss 1, Pp 1-14 (2019)
Orphanet Journal of Rare Diseases
Background Acquired or genetic abnormalities of the complement alternative pathway are the primary cause of C3glomerulopathy(C3G) but may occur in immune-complex-mediated membranoproliferative glomerulonephritis (IC-MPGN) as well. Less is known about
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b32a62ff9e5d5682081ce89ce931d33
https://repository.vu.lt/VU:ELABAPDB55905040&prefLang=en_US
https://repository.vu.lt/VU:ELABAPDB55905040&prefLang=en_US