Mycoplasma Contamination of Cultured Amniotic Fluid Cells: Potential Hazard to Prenatal Chromosomal Diagnosis

Autor: Schneider, E. L., Stanbridge, E. J., Epstein, C. J., Golbus, M., Abbo-Halbasch, G., Rodgers, G.

Publikováno v: Science, 1974 Apr . 184(4135), 477-480.

Externí odkaz: https://www.jstor.org/stable/1738525

Further Studies on the Radiation-Induced Expression of a Tumor-Specific Antigen in Human Cell Hybrids

Autor: Sun, C., Redpath, J. L., Colman, M., Stanbridge, E. J.

Publikováno v: Radiation Research, 1988 Apr 01. 114(1), 84-93.

Externí odkaz: https://www.jstor.org/stable/3577146

Satellite Colony Formation: A Potential Problem in the Quantitative Assay for Neoplastic Transformation of Human Cell Hybrids (HeLa × Skin Fibroblasts)

Autor: Redpath, J. L., Sun, C., Stanbridge, E. J.

Publikováno v: Radiation Research, 1989 Feb 01. 117(2), 356-358.

Externí odkaz: https://www.jstor.org/stable/3577337

Proteasomal inhibition attenuates transcriptional activity of hypoxia-inducible factor 1 (HIF-1) via specific effect on the HIF-1 alpha C-terminal activation domain

Autor: Kaluz, S, Kaluzova, M, Stanbridge, E J

Publikováno v: Kaluz, S; Kaluzova, M; & Stanbridge, E J. (2006). Proteasomal inhibition attenuates transcriptional activity of hypoxia-inducible factor 1 (HIF-1) via specific effect on the HIF-1 alpha C-terminal activation domain. Molecular and Cellular Biology, 26(15), 5895-5907. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/4q50j5th

The ubiquitin-proteasome pathway (UPP) is involved in regulation of multiple cellular processes. Hypoxia-inducible factor 1 alpha (HIF-1 alpha) is a prototypic target of the UPP and, as such, is stabilized under conditions of proteasomal inhibition.

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______325::99fb869448bdc6ac8a027d1a0688aa1a
http://www.escholarship.org/uc/item/4q50j5th

Studies on phenotypic complementation of ataxia-telangiectasia cells by chromosome transfer

Autor: Jongmans, W, Verhaegh, G W, Jaspers, N G, Oshimura, M, Stanbridge, E J, Lohman, P H, Zdzienicka, M Z

Cells derived from patients with the cancer-prone inherited disorder ataxia-telangiectasia (A-T) show an abnormal response to ionizing radiation-induced DNA damage, such as an increased cell killing and a diminished inhibition of DNA synthesis. The e

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::472ded1db787f3af03c30dcc247dcb6a
https://europepmc.org/articles/PMC1801140/