Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Stéphane Heitz"'
Autor:
Raphael Leschiera, Anne-Marie Haeberlé, Stéphane Heitz, Yannick Bailly, Guy Bombarde, Nancy J. Grant, Valérie Demais
Publikováno v:
Brain Pathology. 20:119-132
In Ngsk prion protein (PrP)-deficient mice (NP(0/0)), ectopic expression of PrP-like protein Doppel (Dpl) in central neurons induces significant Purkinje cell (PC) death resulting in late-onset ataxia. NP(0/0) PC death is partly prevented by either k
Autor:
Alice Pavlowsky, Jamel Chelly, Henriette Skala, Malik Khelfaoui, Yann Humeau, Bernard Poulain, Frédéric Gambino, Stéphane Heitz, Pierre Billuart, Nicolas Vitale, Marie Kneib
Publikováno v:
European Journal of Neuroscience
European Journal of Neuroscience, Wiley, 2009, 30 (8), pp.1476-1486. ⟨10.1111/j.1460-9568.2009.06975.x⟩
European Journal of Neuroscience, Wiley, 2009, 30 (8), pp.1476-1486. ⟨10.1111/j.1460-9568.2009.06975.x⟩
Abnormalities in the formation and function of cerebellar circuitry potentially contribute to cognitive deficits in humans. In the adult, the activity of the sole output neurons of the cerebellar cortex - the Purkinje cells (PCs) - is shaped by the b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::80846a8d1ea75a4b9c70062b5906452c
https://hal.archives-ouvertes.fr/hal-02392416
https://hal.archives-ouvertes.fr/hal-02392416
Autor:
Stéphane, Heitz, Nancy J, Grant, Raphael, Leschiera, Anne-Marie, Haeberlé, Valérie, Demais, Guy, Bombarde, Yannick, Bailly
Publikováno v:
Brain Pathol
In Ngsk prion protein (PrP)-deficient mice (NP(0/0)), ectopic expression of PrP-like protein Doppel (Dpl) in central neurons induces significant Purkinje cell (PC) death resulting in late-onset ataxia. NP(0/0) PC death is partly prevented by either k
Autor:
Stéphane Heitz, Guy Bombarde, Michael W. Vogel, J.-L. Rodeau, Y. Lutz, Yannick Bailly, Jean Mariani, F. Richard, J.-P. Fuchs, Hadi S. Zanjani, Izumi Sugihara, Vanessa Gautheron
Publikováno v:
Developmental Neurobiology
Developmental Neurobiology, Wiley, 2008, 68 (3), pp.332-48. ⟨10.1002/dneu.20555⟩
Developmental Neurobiology, Wiley, 2008, 68 (3), pp.332--348. ⟨10.1002/dneu.20555⟩
Developmental Neurobiology, 2008, 68 (3), pp.332-48. ⟨10.1002/dneu.20555⟩
Developmental Neurobiology, Wiley, 2008, 68 (3), pp.332-48. ⟨10.1002/dneu.20555⟩
Developmental Neurobiology, Wiley, 2008, 68 (3), pp.332--348. ⟨10.1002/dneu.20555⟩
Developmental Neurobiology, 2008, 68 (3), pp.332-48. ⟨10.1002/dneu.20555⟩
International audience; The pro-apoptotic factor BAX has recently been shown to contribute to Purkinje cell (PC) apoptosis induced by the neurotoxic prion-like protein Doppel (Dpl) in the prion-protein-deficient Ngsk Prnp(0/0) (NP(0/0)) mouse. In vie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f7b3d023d1b18dfe9b73b3d95a5e4c2
https://hal.archives-ouvertes.fr/hal-00303707
https://hal.archives-ouvertes.fr/hal-00303707
Autor:
Vanessa Gautheron, F. Richard, J.-P. Fuchs, Yannick Bailly, Guy Bombarde, Hadi Shojaeian Zanjani, J.-L. Rodeau, Stéphane Heitz, Michael W. Vogel, Jean Mariani, Y. Lutz
Publikováno v:
Developmental Neurobiology
Developmental Neurobiology, Wiley, 2007, 67 (5), pp.670--686. ⟨10.1002/dneu.20366⟩
Dev Neurobiol
Dev Neurobiol, 2007, 67 (5), pp.670-686. ⟨10.1002/dneu.20366⟩
Developmental Neurobiology, Wiley, 2007, 67 (5), pp.670--686. ⟨10.1002/dneu.20366⟩
Dev Neurobiol
Dev Neurobiol, 2007, 67 (5), pp.670-686. ⟨10.1002/dneu.20366⟩
Research efforts to deduce the function of the prion protein (PrP(c)) in knock-out mouse mutants have revealed that large deletions in the PrP(c) genome result in the ectopic neuronal expression of the prion-like protein Doppel (Dpl). In our analysis
Autor:
Jean Mariani, Yannick Bailly, Fekrije Selimi, Stéphane Heitz, Alexis Lalouette, Ann M. Lohof, Christopher I. Jarvis
Publikováno v:
Neuron
Neuron, 2003, 37, pp.813-819. ⟨10.1016/S0896-6273(03)00093-X⟩
Neuron, Elsevier, 2003, 37, pp.813-819. ⟨10.1016/S0896-6273(03)00093-X⟩
Neuron, 2003, 37, pp.813-819. ⟨10.1016/S0896-6273(03)00093-X⟩
Neuron, Elsevier, 2003, 37, pp.813-819. ⟨10.1016/S0896-6273(03)00093-X⟩
The Lurcher mutation transforms the GRID2 receptor into a constitutively opened channel. In Lurcher heterozygous mice, cerebellar Purkinje cells are permanently depolarized, a characteristic that has been thought to be the primary cause of their deat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f3b268163a5e7a0ad32e7e10f57cb564
https://hal.science/hal-00068752
https://hal.science/hal-00068752