Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Srinivasan, Kasturirangan"'
Autor:
Vikram Brahmanandam, Anish Shah, Lianzhi Gu, Mihai Raicu, Melissa Robinson Wood, Samuel C. Dudley, Alan Schwartz, Li Zhou, Srinivasan Kasturirangan, Smita I. Negi, Ankit A. Desai, Antone Tatooles, Ge Gao
Publikováno v:
Journal of the American College of Cardiology. 63(21):2261-2269
Objectives The aim of this study was to determine the association of SCN5A cardiac sodium (Na+) channel mRNA splice variants in white blood cells (WBCs) with risk of arrhythmias in heart failure (HF). Background HF is associated with upregulation of
Autor:
Euy Myoung Jeong, Sassan Ghassemzadeh, Michael A. Sobieski, Amanda M. Herman, G. Bhat, Shu-Ching Huang, Antone Tatooles, Samuel C. Dudley, Edward J. Benz, An Xie, Ge Gao, Timothy J. Kamp, Anyu Zhou, Srinivasan Kasturirangan, Mihai Raicu, Jianhua Zhang
Publikováno v:
Circulation. 124(10)
Background— Human heart failure is associated with decreased cardiac voltage-gated Na + channel current (encoded by SCN5A), and the changes have been implicated in the increased risk of sudden death in heart failure. Nevertheless, the mechanism of
Autor:
Mihai Raicu, Vikram Brahmanandam, Anish Shah, Smita I. Negi, Lianzhi Gu, Srinivasan Kasturirangan, Ge Gao, Samuel C. Dudley, Alan Schwartz
Publikováno v:
Journal of the American College of Cardiology. 59:E720
Over half of heart failure (HF) patients with an implantable cardioverter-defibrillator (ICD) placed for the prevention of sudden cardiac death (SCD) never require an ICD shock based on current risk stratification guidelines. We previously found that
Autor:
Ge, Gao, Vikram, Brahmanandam, Mihai, Raicu, Lianzhi, Gu, Li, Zhou, Srinivasan, Kasturirangan, Anish, Shah, Smita I, Negi, Melissa R, Wood, Ankit A, Desai, Antone, Tatooles, Alan, Schwartz, Samuel C, Dudley
Publikováno v:
Journal of the American College of Cardiology. (21):2261-2269
ObjectivesThe aim of this study was to determine the association of SCN5A cardiac sodium (Na+) channel mRNA splice variants in white blood cells (WBCs) with risk of arrhythmias in heart failure (HF).BackgroundHF is associated with upregulation of two