Inconsequential role for chemerin-like receptor 1 in the manifestation of ozone-induced lung pathophysiology in male mice.

Autor: Johnston RA; Health Effects Laboratory Division, National Institute for Occupational Safety and Health, Centers for Disease Control and Prevention, United States Department of Health and Human Services, Morgantown, West Virginia, USA.; Section of Pulmonary, Critical Care, and Sleep Medicine, Department of Medicine, School of Medicine, West Virginia University, Morgantown, West Virginia, USA.; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.; Department of Integrative Biology and Pharmacology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA., Pilkington AW 4th; Health Effects Laboratory Division, National Institute for Occupational Safety and Health, Centers for Disease Control and Prevention, United States Department of Health and Human Services, Morgantown, West Virginia, USA., Atkins CL; Division of Pulmonary Medicine, Department of Pediatrics, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA., Boots TE; Health Effects Laboratory Division, National Institute for Occupational Safety and Health, Centers for Disease Control and Prevention, United States Department of Health and Human Services, Morgantown, West Virginia, USA., Brown PL; Health Effects Laboratory Division, National Institute for Occupational Safety and Health, Centers for Disease Control and Prevention, United States Department of Health and Human Services, Morgantown, West Virginia, USA., Jackson WT; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA., Spencer CY; Section of Pediatric Pulmonology, Department of Pediatrics, Baylor College of Medicine, Houston, Texas, USA., Siddiqui SR; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA., Haque IU; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.; Division of Critical Care, Department of Pediatrics, Sidra Medicine, Doha, Qatar.

Publikováno v: Physiological reports [Physiol Rep] 2024 Apr; Vol. 12 (8), pp. e16008.

Airway Autoimmune Inflammatory Response (AAIR) Syndrome: An Asthma-Autoimmune Overlap Disorder?

Autor: Spencer CY; Department of Pediatrics (Kravis Children's Hospital), Icahn School of Medicine at Mount Sinai, New York, New York., Millman J; Department of Pediatrics (Kravis Children's Hospital), Icahn School of Medicine at Mount Sinai, New York, New York., Veiga K; Department of Pediatrics (Kravis Children's Hospital), Icahn School of Medicine at Mount Sinai, New York, New York., Vicencio AG; Department of Pediatrics (Kravis Children's Hospital), Icahn School of Medicine at Mount Sinai, New York, New York alfin.vicencio@mssm.edu.

Publikováno v: Pediatrics [Pediatrics] 2018 Mar; Vol. 141 (3). Date of Electronic Publication: 2018 Feb 15.

Chemokine (C-C Motif) Receptor-Like 2 is not essential for lung injury, lung inflammation, or airway hyperresponsiveness induced by acute exposure to ozone.

Autor: Malik F; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas., Cromar KR; Marron Institute of Urban Management New York University, New York, New York., Atkins CL; Division of Pulmonary Medicine, Department of Pediatrics, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas., Price RE; Comparative Pathology Laboratory, Center for Comparative Medicine, Baylor College of Medicine, Houston, Texas., Jackson WT; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas., Siddiqui SR; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas., Spencer CY; Section of Pediatric Pulmonology, Department of Pediatrics, Baylor College of Medicine, Houston, Texas., Mitchell NC; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas., Haque IU; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas., Johnston RA; Division of Critical Care Medicine, Department of Pediatrics, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas rfj1@cdc.gov.; Department of Integrative Biology and Pharmacology, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas.

Publikováno v: Physiological reports [Physiol Rep] 2017 Dec; Vol. 5 (24).

Resistin deficiency in mice has no effect on pulmonary responses induced by acute ozone exposure.

Autor: Razvi SS; Division of Critical Care Medicine, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas;, Richards JB; Division of Pulmonary, Critical Care, and Sleep Medicine, Department of Medicine, Medical University of South Carolina, Charleston, South Carolina;, Malik F; Division of Critical Care Medicine, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas;, Cromar KR; Department of Environmental Medicine, New York University School of Medicine, Tuxedo, New York;, Price RE; Comparative Pathology Laboratory, Center for Comparative Medicine, Baylor College of Medicine, Houston, Texas;, Bell CS; Division of Nephrology, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas;, Weng T; Department of Biochemistry and Molecular Biology, The University of Texas Medical School at Houston, Houston, Texas;, Atkins CL; Division of Pulmonary Medicine, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas;, Spencer CY; Pediatric Pulmonary Section, Department of Pediatrics, Baylor College of Medicine, Houston, Texas;, Cockerill KJ; Pediatric Research Center, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas;, Alexander AL; Pediatric Research Center, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas;, Blackburn MR; Department of Biochemistry and Molecular Biology, The University of Texas Medical School at Houston, Houston, Texas;, Alcorn JL; Department of Biochemistry and Molecular Biology, The University of Texas Medical School at Houston, Houston, Texas; Pediatric Research Center, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas; Division of Neonatal-Perinatal Medicine, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas; and., Haque IU; Division of Critical Care Medicine, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas;, Johnston RA; Division of Critical Care Medicine, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas; Pediatric Research Center, Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas; Department of Integrative Biology and Pharmacology, The University of Texas Medical School at Houston, Houston, Texas Richard.A.Johnston@uth.tmc.edu.

Publikováno v: American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2015 Nov 15; Vol. 309 (10), pp. L1174-85. Date of Electronic Publication: 2015 Sep 18.