Zobrazeno 1 - 10
of 23
pro vyhledávání: '"Sophie Parmentier-Batteur"'
Autor:
Angela Marie Jablonski, Lee Warren, Marija Usenovic, Heather Zhou, Jonathan Sugam, Sophie Parmentier-Batteur, Bhavya Voleti
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-18 (2021)
Abstract ApoEε4 is a major genetic risk factor for Alzheimer’s disease (AD), a disease hallmarked by extracellular amyloid-beta (Aβ) plaques and intracellular neurofibrillary tangles (NFTs). The presence of the ApoEε4 allele is associated with i
Externí odkaz:
https://doaj.org/article/4fe83d6941c44b5bbe01ae4d30b636f0
Autor:
Yuan Tian, Yi Wang, Angela M. Jablonski, Yinghui Hu, Jonathan A. Sugam, Markus Koglin, Shawn J. Stachel, Heather Zhou, Jason M. Uslaner, Sophie Parmentier-Batteur
Publikováno v:
Neurobiology of Disease, Vol 161, Iss , Pp 105548- (2021)
TDP-43 pathology is a hallmark of Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal lobar degeneration (FTLD). Namely, both diseases feature aggregated and phosphorylated TDP-43 containing inclusions in the cytoplasm and a loss of nuclear TDP-43
Externí odkaz:
https://doaj.org/article/10127822ae8a4b869c780c0f430e8e11
Autor:
Jonathan A. Sugam, Jason M. Uslaner, Sophie Parmentier-Batteur, Markus Koglin, Angela Marie Jablonski, Yi Wang, Heather Zhou, Yuan Tian, Shawn J. Stachel, Yinghui Hu
Publikováno v:
Neurobiology of Disease, Vol 161, Iss, Pp 105548-(2021)
TDP-43 pathology is a hallmark of Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal lobar degeneration (FTLD). Namely, both diseases feature aggregated and phosphorylated TDP-43 containing inclusions in the cytoplasm and a loss of nuclear TDP-43
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::26483812e9293df7d140293b1f92c574
https://pubmed.ncbi.nlm.nih.gov/34752923
https://pubmed.ncbi.nlm.nih.gov/34752923
Autor:
J.B. Schachter, Marija Usenovic, Cheryl A. Gretzula, M.K. Schultz, C. Ware, Sophie Parmentier-Batteur, Hatim A. Zariwala, R. Gentzel
Publikováno v:
Neurobiology of Disease, Vol 118, Iss, Pp 161-176 (2018)
In Alzheimer's Disease (AD), tau pathology has a spatiotemporally distinct pattern of progressive spread along anatomically connected neural pathways. Extracellular tau in the brain interstitial space increases in response to neuronal activity sugges
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e44991f7efe700779745b710c81a980e
https://doi.org/10.1016/j.nbd.2018.07.003
https://doi.org/10.1016/j.nbd.2018.07.003
Publikováno v:
Alzheimer's & Dementia. 14
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d5f4f87013434af552a12a9237f467bf
https://doi.org/10.1016/j.jalz.2018.06.1550
https://doi.org/10.1016/j.jalz.2018.06.1550
Autor:
Sophie Parmentier-Batteur, John J. Renger, Joel B. Schachter, Nathan G. Hatcher, Marija Usenovic, Robert E. Drolet, Renee C. Gaspar, Shahriar Niroomand, Lihang Yao
Publikováno v:
The Journal of Neuroscience. 35:14234-14250
Neuronal inclusions of hyperphosphorylated and aggregated tau protein are a pathological hallmark of several neurodegenerative tauopathies, including Alzheimer's disease (AD). The hypothesis of tau transmission in AD has emerged from histopathologica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7f9f2d5c1c89c380ff84a4551838bf1
https://doi.org/10.1523/jneurosci.1523-15.2015
https://doi.org/10.1523/jneurosci.1523-15.2015
[P3–083]: PHENOTYPIC SCREENING FOR NOVEL INHIBITORS OF TAU AGGREGATION IN HUMAN IPSC‐DERIVED NEURONS
Autor:
Bhavya Voleti, Jing Li, Marija Usenovic, Sophie Parmentier-Batteur, Heather Zhou, Bin Luo, Lee Warren, Abbas Walji, Joel B. Schachter
Publikováno v:
Alzheimer's & Dementia. 13
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::34fa6ffa781c1e2c5d2f680e3d3b97c6
https://doi.org/10.1016/j.jalz.2017.06.1293
https://doi.org/10.1016/j.jalz.2017.06.1293
Publikováno v:
Alzheimer's & Dementia. 13
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::11ee959827f20038c538aeb54af8f675
https://doi.org/10.1016/j.jalz.2017.06.1977
https://doi.org/10.1016/j.jalz.2017.06.1977
Autor:
Kelly-Ann S. Schlegel, Peter S. Kutchukian, Lee Warren, Sophie Parmentier-Batteur, Jason Cassaday, Victor N. Uebele, Brian C. Magliaro, Brian Squadroni, Danielle M. Hurzy, Eric N. Johnson, J. Chris Culberson, Gregory O'Donnell, Adam Amoss, Fiona Thomson, Michael F.A. Finley, Paul Zuck, Andrew J. Cooke, Danette Pascarella, Jeffrey D. Hermes
N-methyl-d-aspartate receptors (NMDARs) mediate glutamatergic signaling that is critical to cognitive processes in the central nervous system, and NMDAR hypofunction is thought to contribute to cognitive impairment observed in both schizophrenia and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::83ee89032dec0bd4c1bf64d0fea584c0
https://eprints.gla.ac.uk/146530/1/146530.pdf
https://eprints.gla.ac.uk/146530/1/146530.pdf
Autor:
Sarah L. Huszar, Thomas W. Rosahl, Sophie Parmentier-Batteur, H. Fauré, Thomas J. Tucker, Craig A. Stump, Emannuel Le Poul, Richard Hargreaves, Brian C. Magliaro, Xu-Fang Zhang, Neville J. Anthony, Thomas Forest, Theresa M. Williams, Jason M. Uslaner, Nathalie Lambeng, Julie A. O'Brien, Robert P. Gomez, Robert M. Tynebor, Peter H. Hutson, Sonia Poli, Jean-Philippe Rocher, Britta A. Mattson, Karsten Menzel
Publikováno v:
Neuropharmacology. 82:161-173
Previous work has suggested that activation of mGlu5 receptor augments NMDA receptor function and thereby may constitute a rational approach addressing glutamate hypofunction in schizophrenia and a target for novel antipsychotic drug development. Her
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::44024f123a60505445bf06fd0e251b96
https://doi.org/10.1016/j.neuropharm.2012.12.003
https://doi.org/10.1016/j.neuropharm.2012.12.003