Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Somang Kang"'
Autor:
Young-Suk Choi, Somang Kang, Sang-Yoon Ko, Saeram Lee, Jae Young Kim, Hansol Lee, Jae Eun Song, Dong-Hyun Kim, Eosu Kim, Chul Hoon Kim, Lisa Saksida, Ho-Taek Song, Jong Eun Lee
Publikováno v:
Molecular Brain, Vol 11, Iss 1, Pp 1-12 (2018)
Abstract Higher dietary intakes of saturated fatty acid increase the risk of developing Alzheimer’s disease and dementia, and even in people without diabetes higher glucose levels may be a risk factor for dementia. The mechanisms causing neuronal d
Externí odkaz:
https://doaj.org/article/a2814ebb79024ec1986848c294d26fae
Publikováno v:
Neuropharmacology. 113:467-479
The risk of Alzheimer's disease (AD) is higher in patients with type 2 diabetes mellitus (T2DM). Previous studies in high-fat diet-induced AD animal models have shown that brain insulin resistance in these animals leads to the accumulation of amyloid
Autor:
Juhyun Song, Eosu Kim, Chul Hoon Kim, Jong Eun Lee, Somang Kang, Yumi Oh, Byeori Lee, Ho Taek Song
Publikováno v:
Experimental Neurobiology
Neuronal senescence caused by diabetic neuropathy is considered a common complication of diabetes mellitus. Neuronal senescence leads to the secretion of pro-inflammatory cytokines, the production of reactive oxygen species, and the alteration of cel
Autor:
Ho Taek Song, Eosu Kim, Lisa M. Saksida, Chul Hoon Kim, Jong Eun Lee, Somang Kang, Jae Eun Song, Jae-Young Kim, Young Suk Choi, Sang Yoon Ko, Donghyun Kim, Hansol Lee, Saeram Lee
Publikováno v:
Molecular Brain
Molecular Brain, Vol 11, Iss 1, Pp 1-12 (2018)
Molecular Brain, Vol 11, Iss 1, Pp 1-12 (2018)
Higher dietary intakes of saturated fatty acid increase the risk of developing Alzheimer’s disease and dementia, and even in people without diabetes higher glucose levels may be a risk factor for dementia. The mechanisms causing neuronal dysfunctio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::385da20fe7c9d4a9d146e85f4e923634
https://www.repository.cam.ac.uk/handle/1810/287195
https://www.repository.cam.ac.uk/handle/1810/287195
Autor:
Choi, Young-Suk, Somang Kang, Sang-Yoon Ko, Saeram Lee, Kim, Jae, Hansol Lee, Song, Jae, Kim, Dong-Hyun, Eosu Kim, Kim, Chul, Saksida, Lisa, Ho-Taek Song, Lee, Jong
Figure S1. Hyperpolarized [1-13C]lactate/[1-13C]pyruvate ratio in medial temporal lobe showed a negative correlation with time to spent in the target quadrant (n = 9, Pearson’s r = − 0.692, P
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::240cf7677fb95b569c1ccbfa2a0d81bc
Publikováno v:
Yonsei Medical Journal
Alzheimer's disease (AD) is a degenerative brain disease and the most common cause of dementia. AD is characterized by the extracellular amyloid beta (Aβ) plaques and intraneuronal deposits of neurofibrillary tangles (NFTs). Recently, as aging has b
Publikováno v:
Experimental Neurobiology
Differentiation of neural progenitor cells (NPCs) is important for protecting neural cells and brain tissue during inflammation. Interleukin-1 beta (IL-1β) is the most common pro- inflammatory cytokine in brain inflammation, and increased IL-1β lev
Publikováno v:
Neuroscience. 301
In the central nervous system (CNS), insulin resistance (I/R) can cause defective neurite outgrowth and neuronal cell death, which can eventually lead to cognitive deficits. Recent research has focused on the relationship between I/R and the cognitiv
Publikováno v:
CELL DEATH & DISEASE(6)
Cell Death & Disease
Cell Death & Disease
In the central nervous system (CNS), hyperglycemia leads to neuronal damage and cognitive decline. Recent research has focused on revealing alterations in the brain in hyperglycemia and finding therapeutic solutions for alleviating the hyperglycemia-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e5a8200a8bf5a350719e1d52d503ffc5
http://open-repository.kisti.re.kr/cube/handle/open_repository/474323.do
http://open-repository.kisti.re.kr/cube/handle/open_repository/474323.do
Publikováno v:
Yonsei Medical Journal; May2017, Vol. 58 Issue 3, p479-488, 10p