Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Sofia Makeenok"'
Autor:
Konstantin Gusev, Alexey Shalygin, Dmitrii Kolesnikov, Leonid Shuyskiy, Sofia Makeenok, Lyubov Glushankova, Konstantin Sivak, Kirill Yakovlev, Yana Orshanskaya, Guanghui Wang, Andrey Bakhtyukov, Kira Derkach, Alexander Shpakov, Elena Kaznacheyeva
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 8, p 7259 (2023)
Type 2 diabetes mellitus (DM2) is a widespread metabolic disorder that results in podocyte damage and diabetic nephropathy. Previous studies demonstrated that TRPC6 channels play a pivotal role in podocyte function and their dysregulation is associat
Externí odkaz:
https://doaj.org/article/9b7698ee08bd46609be2f91c51036368
Autor:
Konstantin Gusev, Aleksandr Khudiakov, Anastasia Zaytseva, Kseniya Perepelina, Sofia Makeenok, Elena Kaznacheyeva, Anna Kostareva
Publikováno v:
Cellular Physiology and Biochemistry, Vol 54, Iss 4, Pp 696-706 (2020)
Externí odkaz:
https://doaj.org/article/c2afc2d4c79b470083544a451f4a8d58
Autor:
Konstantin Gusev, Alexey Shalygin, Dmitrii Kolesnikov, Leonid Shuyskiy, Sofia Makeenok, Lyubov Glushankova, Konstantin Sivak, Kirill Yakovlev, Yana Orshanskaya, Guanghui Wang, Andrey Bakhtyukov, Kira Derkach, Alexander Shpakov, Elena Kaznacheyeva
Type 2 diabetes mellitus (DM2) is a widespread metabolic disorder that results in podocyte damage and diabetic nephropathy. Previous studies demonstrated that TRPC6 channels play a pivotal role in podocyte function and their dysregulation is associat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9fdb0765b07b972448abfb9cdfc95259
https://doi.org/10.21203/rs.3.rs-2511159/v1
https://doi.org/10.21203/rs.3.rs-2511159/v1
Autor:
Aleksandr Khudiakov, Kseniya Perepelina, Anastasia K Zaytseva, Anna Kostareva, Elena Kaznacheyeva, Sofia Makeenok, Konstantin Gusev
Publikováno v:
Cellular Physiology and Biochemistry, Vol 54, Iss 4, Pp 696-706 (2020)
Background/aims Mutations of desmosomal genes are known to cause arrhythmogenic cardiomyopathy characterized by arrhythmias and sudden cardiac death. Previously, we described a novel genetic variant H1684R in desmoplakin gene (DSP), associated with a