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Cyprinid herpesvirus 3 evolves in vitro through an assemblage of haplotypes that alternatively become dominant or under-represented

Autor: Klafack, S., Fiston-Lavier, A. S., Bergmann, S. M., Hammoumi, S., Schroder, L., Fuchs, W., Lusiastuti, A., Lee, P. Y., Heredia, S. V., Gosselin-Grenet, A. S., Avarre, Jean-Christophe, Amoyal, C., Amraoui, H., Bigourdan, A., Boujandir, I., Bueno, O., Cotteux, B., de San Nicolas, N., Delattre, R., Diouf, M. B., Dussaut, R., Gawron, K., Imbert, J. B., Mouilhi, H., Nifaut, E., Pages, A., Platon, L., Sanchez, S., Simon, L., Sobry, H., Souche, M., Taillefer, B., Guemgne, E. N., Sacchi, O., Diouf, A., Ouzerdine, A. L., Durand, F., Brooke, J., Cremaschi, J., Massaviol, M., Hamouma, M., Salma, M., Soulie, M., Delorme, Q., Virgoulay, T., Klein, Valentin, Antoine, Y., Berne, A., Gaal, C., Master Student Consortium

Viruses are able to evolve in vitro by mutations after serial passages in cell cultures, which can lead to either a loss, or an increase, of virulence. Cyprinid herpesvirus 3 (CyHV-3), a 295-kb double-stranded DNA virus, is the etiological agent of t

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