Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Slávka Carnická"'
Autor:
Tomas Rajtik, Slavka Carnicka, Adrian Szobi, Zoltan Giricz, Jin O-Uchi, Veronika Hassova, Pavel Svec, Peter Ferdinandy, Tanya Ravingerova, Adriana Adameova
Publikováno v:
Data in Brief, Vol 7, Iss , Pp 730-734 (2016)
Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data art
Externí odkaz:
https://doaj.org/article/7a49268a8081425485b908240be473ac
Publikováno v:
AIP Advances, Vol 7, Iss 5, Pp 056718-056718-6 (2017)
Magnetic Resonance Imaging (MRI) is increasingly used to map the magnetic susceptibility of tissue to identify cerebral microbleeds associated with traumatic brain injury and pathological iron deposits associated with neurodegenerative diseases such
Externí odkaz:
https://doaj.org/article/dddcf48c296b4d98b985ebb41ec9076d
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 87:1055-1062
Opening of mitochondrial KATPchannels (mitoKATP) has been reported to underlie protection against ischaemia–reperfusion injury induced by ischaemic preconditioning (I-PC); however, the molecular mechanisms of its antiarrhythmic effect have not been
Autor:
Tána, Ravingerová, Adriana, Adameová, Tara, Kelly, Efthymia, Antonopoulou, Dezider, Pancza, Mária, Ondrejcáková, Vinoth Kumar Megraj, Khandelwal, Slávka, Carnická, Antigone, Lazou
Publikováno v:
Canadian journal of physiology and pharmacology. 87(12)
Peroxisome proliferator-activated receptors (PPAR), which are key transcriptional regulators of lipid metabolism and energy production, have been suggested to play an important role in myocardial ischaemia-reperfusion (I/R) injury. Their role in card
Publikováno v:
Canadian journal of physiology and pharmacology. 87(12)
Opening of mitochondrial KATP channels (mitoKATP) has been reported to underlie protection against ischaemia-reperfusion injury induced by ischaemic preconditioning (I-PC); however, the molecular mechanisms of its antiarrhythmic effect have not been