Zobrazeno 1 - 10 of 37 pro vyhledávání: '"Sjenet E. van Emst-de Vries"'
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Akademický článek
Prompt inhibition of fMLP-induced Ca2+ mobilization by parenteral lipid emulsions in human neutrophils
Autor: Geert Wanten, Angelique Rops, Sjenet E. van Emst-de Vries, Ton Naber, Peter H.G.M. Willems
Publikováno v: Journal of Lipid Research, Vol 43, Iss 4, Pp 550-556 (2002)
It remains unclear whether modulation of immune system functions by lipids contributes to the increased infection rate observed in patients treated with parenteral nutrition. We therefore evaluated the effects of lipid emulsions derived from fish oil
Externí odkaz: https://doaj.org/article/6a26286d675f41b5a0dcda26f4d836f1
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3
The decylTPP mitochondria-targeting moiety lowers electron transport chain supercomplex levels in primary human skin fibroblasts
Autor: Elianne P. Bulthuis, Claudia Einer, Felix Distelmaier, Laszlo Groh, Sjenet E. van Emst - de Vries, Els van de Westerlo, Melissa van de Wal, Jori Wagenaars, Richard J. Rodenburg, Jan A.M. Smeitink, Niels P. Riksen, Peter H.G.M. Willems, Merel J.W. Adjobo-Hermans, Hans Zischka, Werner J.H. Koopman
Publikováno v: Free Radical Biology and Medicine, 188, pp. 434-446
Free Radical Biology and Medicine 188 (2022)
Free Radical Biol. Med. 188, 434-446 (2022)
Free Radical Biology and Medicine, 188, 434-446
Attachment of cargo molecules to lipophilic triphenylphosphonium (TPP+) cations is a widely applied strategy for mitochondrial targeting. We previously demonstrated that the vitamin E-derived antioxidant Trolox increases the levels of active mitochon
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::18dae1c924e2eac5b5e0ad826c47c289
https://hdl.handle.net/https://repository.ubn.ru.nl/handle/2066/253380
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4
NDUFS4 deletion triggers loss of NDUFA12 in Ndufs4
Autor: Merel J W, Adjobo-Hermans, Ria, de Haas, Peter H G M, Willems, Aleksandra, Wojtala, Sjenet E, van Emst-de Vries, Jori A, Wagenaars, Mariel, van den Brand, Richard J, Rodenburg, Jan A M, Smeitink, Leo G, Nijtmans, Leonid A, Sazanov, Mariusz R, Wieckowski, Werner J H, Koopman
Publikováno v: Biochimica et biophysica acta. Bioenergetics. 1861(8)
Mutations in NDUFS4, which encodes an accessory subunit of mitochondrial oxidative phosphorylation (OXPHOS) complex I (CI), induce Leigh syndrome (LS). LS is a poorly understood pediatric disorder featuring brain-specific anomalies and early death. T
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::17f057b315e2d62d5f878aaa0c00d4c5
https://pubmed.ncbi.nlm.nih.gov/32335026
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5
Variants in NGLY1 lead to intellectual disability, myoclonus epilepsy, sensorimotor axonal polyneuropathy and mitochondrial dysfunction
Autor: Charlotte A. Haaxma, Daan M. Panneman, Nicole I. Wolf, Els M.A. van de Westerlo, Sjenet E. van Emst-de Vries, Werner J.H. Koopman, Maaike de Vries, Saskia B. Wortmann, Dirk Lefeber, Frans van den Brandt, Yvonne Hendriks, Richard J. Rodenburg, Jan A.M. Smeitink, Peter M. van Hasselt, Benno Küsters, Liesbeth T. Wintjes
Publikováno v: Clinical Genetics, 97, 4, pp. 556-566
Clinical Genetics, 97, 556-566
Panneman, D M, Wortmann, S B, Haaxma, C A, van Hasselt, P M, Wolf, N I, Hendriks, Y, Küsters, B, van Emst-de Vries, S, van de Westerlo, E, Koopman, W J H, Wintjes, L, van den Brandt, F, de Vries, M, Lefeber, D J, Smeitink, J A M & Rodenburg, R J 2020, ' Variants in NGLY1 lead to intellectual disability, myoclonus epilepsy, sensorimotor axonal polyneuropathy and mitochondrial dysfunction ', Clinical Genetics, vol. 97, no. 4, pp. 556-566 . https://doi.org/10.1111/cge.13706
Clinical Genetics, 97(4), 556-566. Wiley-Blackwell
Clinical Genetics
Clin. Genet. 97, 556-566 (2020)
Contains fulltext : 218925.pdf (Publisher’s version ) (Open Access) NGLY1 encodes the enzyme N-glycanase that is involved in the degradation of glycoproteins as part of the endoplasmatic reticulum-associated degradation pathway. Variants in this ge
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8963f6324659dbd47f99639baf9be848
https://doi.org/10.1111/cge.13706
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6
NDUFS4 deletion triggers loss of NDUFA12 in Ndufs4(-/-) mice and Leigh syndrome patients: A stabilizing role for NDUFAF2
Autor: Leo G.J. Nijtmans, Mariël A.M. van den Brand, Jori A. L. Wagenaars, Leonid A. Sazanov, Werner J.H. Koopman, Jan A.M. Smeitink, Merel J. W. Adjobo-Hermans, Sjenet E. van Emst-de Vries, Mariusz R. Wieckowski, Richard J. Rodenburg, Peter H.G.M. Willems, Aleksandra Wojtala, Ria de Haas
Publikováno v: Biochimica et Biophysica Acta. Bioenergetics, 1861
Biochimica et Biophysica Acta. Bioenergetics, 1861, 8
Mutations in NDUFS4, which encodes an accessory subunit of mitochondrial oxidative phosphorylation (OXPHOS) complex I (CI), induce Leigh syndrome (LS). LS is a poorly understood pediatric disorder featuring brain-specific anomalies and early death. T
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bbeb3235af78648702b370eecd47a8b4
https://hdl.handle.net/2066/220251
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9
Biodegradable synthetic organelles demonstrate ROS shielding in human-complex-I-deficient fibroblasts
Autor: Loai K. E. A. Abdelmohsen, David S. Williams, Werner J.H. Koopman, Sjenet E. van Emst-de Vries, Roland Brock, Daniela A. Wilson, Jan C. M. van Hest, Lisanne M.P.E. van Oppen, Jan A.M. Smeitink, Pascal L.W. Welzen, Peter H.G.M. Willems
Publikováno v: ACS Central Science, 4(7), 917-928. American Chemical Society
Acs Central Science, 4, 917-928
Acs Central Science, 4, 7, pp. 917-928
ACS Central Science, Vol 4, Iss 7, Pp 917-928 (2018)
ACS Central Science
Biodegradable, semipermeable nanoreactors that are capable of undergoing cellular integration and, subsequently, function as synthetic organelles represent an exciting therapeutic technology. Polymersomal nanoreactors have been investigated as a suit
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::823dfe51081abd0b6d9afc5c32e0560b
https://research.tue.nl/nl/publications/73ee6268-13cf-4e40-84af-e2c00fa4bd86
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10
Mitochondrial complex I inhibition triggers a mitophagy-dependent ROS increase leading to necroptosis and ferroptosis in melanoma cells
Autor: Johannes Cw Hermeling, Hasse M Bossenbroek, Mélanie Héroult, Laura Schöckel, Werner J.H. Koopman, Lisanne M.P.E. van Oppen, Farhan Basit, Sander Grefte, Sjenet E. van Emst-de Vries, Charlotte Christine Kopitz, Peter H.G.M. Willems
Publikováno v: Cell Death & Disease, 8(3)
Cell Death and Disease, 8, e2716-e2716
Cell Death & Disease
Cell Death and Disease, 8, 3, pp. e2716-e2716
Cell Death & Disease 8 (2017) 3
Inhibition of complex I (CI) of the mitochondrial respiratory chain by BAY 87-2243 (‘BAY’) triggers death of BRAFV600E melanoma cell lines and inhibits in vivo tumor growth. Here we studied the mechanism by which this inhibition induces melanoma
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::544da234dd63742f1a2b0b78010d5719
https://research.wur.nl/en/publications/mitochondrial-complex-i-inhibition-triggers-a-mitophagy-dependent
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