Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Sivasithamparam Niranjali Devaraj"'
Publikováno v:
Food Science and Human Wellness, Vol 7, Iss 4, Pp 273-281 (2018)
The purpose of this study was to investigate the possible prophylactic effects of Alpinia officinarum hance on experimentally induced acute and chronic colitis models, in-vivo and in-vitro. Acute and chronic colitis were induced in Male Wistar rats b
Externí odkaz:
https://doaj.org/article/43b60138a8434884b282670fe160dc67
Publikováno v:
In Journal of Functional Foods February 2018 41:240-249
Akademický článek
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Autor:
Kasinathan, Nirmal Kumar, Subramaniya, Bharathi Raja, Pandian, Ilakkiya, Sivasithamparam, Niranjali Devaraj
Publikováno v:
In Biomedicine & Preventive Nutrition April-June 2014 4(2):307-317
Publikováno v:
In Biomedicine & Preventive Nutrition July-September 2013 3(3):299-305
Publikováno v:
In Biomedicine & Preventive Nutrition January-March 2013 3(1):19-25
Autor:
Ramadass, Nandhakumar, Kombiyil, Salini, Kasinathan, Nirmal Kumar, Sivasithamparam, Niranjali Devaraj
Publikováno v:
In Biomedicine & Preventive Nutrition January-March 2012 2(1):9-15
Publikováno v:
Journal of Cellular Physiology. 234:7040-7050
The activation of two transcription factors, NFκB and NICD (notch intracellular domain), plays a crucial role in different stages of atherosclerotic disease progression, from early endothelial activation by modified lipids like oxidized low-density
Publikováno v:
Life Sciences. 196:28-37
Atherogenic Diet (AD) was given to rats to understand the key role of inflammatory mediators in atherosclerotic lesion formation, as a serendipitous study, the diet induced inflammatory mediators in liver and brain, whereas pancreas, kidney and splee
Autor:
Sivasithamparam Niranjali Devaraj, Sakeena Sadullah Mohammed Sadullah, Ashokkumar Rathinavel, Jamuna Sankar
Publikováno v:
Fundamental & Clinical Pharmacology. 32:51-59
Extracellular matrix (ECM) remodeling is a major pathophysiological process during post-myocardial infarction (MI). The activation, differentiation, and proliferation of cardiac fibroblasts to myofibroblasts regulate the expression of ECM proteins. T