Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Sivan Elhanati"'
Autor:
Sivan Elhanati, Rotem Ben-Hamo, Yariv Kanfi, Alexander Varvak, Renana Glazz, Batia Lerrer, Sol Efroni, Haim Y. Cohen
Publikováno v:
Cell Reports, Vol 14, Iss 2, Pp 234-242 (2016)
Mice overexpressing the longevity protein SIRT6 or deficient for the liver’s most prevalent microRNA miR-122 display a similar set of phenotypes, including improved lipid profile and protection against damage linked to obesity. Here, we show that m
Externí odkaz:
https://doaj.org/article/a00fb58347d543fe94282f269d498f61
Autor:
Sivan Elhanati, Yariv Kanfi, Alexander Varvak, Asael Roichman, Ilana Carmel-Gross, Shaul Barth, Gilad Gibor, Haim Y. Cohen
Publikováno v:
Cell Reports, Vol 4, Iss 5, Pp 905-912 (2013)
The NAD+-dependent protein deacetylase SIRT6 regulates genome stability, cancer, and lifespan. Mice overexpressing SIRT6 (MOSES) have lower low-density lipoprotein cholesterol levels and are protected against the physiological damage of obesity. Here
Externí odkaz:
https://doaj.org/article/40ec652d95a4433f8056d09c69c5ae2b
Autor:
Haim Y. Cohen, A. Di Francesco, Orly Yaron, Alexey E. Lyashkov, Yael Shahar, I. Lebenthal-Loinger, Batya Lerrer, Asael Roichman, Miguel A. Aon, Matan Y. Avivi, Sivan Elhanati, Pablo J. Fernandez-Marcos, Yariv Kanfi, Ceereena Ubaida-Mohien, A. Shuchami, Ifat Abramovich, Manuel Serrano, Z. Petrover, R. de Cabo, Eyal Gottlieb, M. Shurgi, Y. Wiesner, A. Rubinstein
Publikováno v:
Dipòsit Digital de la UB
Universidad de Barcelona
Nature Communications, Vol 12, Iss 1, Pp 1-18 (2021)
Nature Communications
Universidad de Barcelona
Nature Communications, Vol 12, Iss 1, Pp 1-18 (2021)
Nature Communications
Aging leads to a gradual decline in physical activity and disrupted energy homeostasis. The NAD+-dependent SIRT6 deacylase regulates aging and metabolism through mechanisms that largely remain unknown. Here, we show that SIRT6 overexpression leads to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6def2446946f4ce3fd3c05338acece95
https://doi.org/10.1038/s41467-021-23545-7
https://doi.org/10.1038/s41467-021-23545-7
Autor:
Batia Lerrer, Rotem Ben-Hamo, Renana Glazz, Yariv Kanfi, Sivan Elhanati, Alexander Varvak, Sol Efroni, Haim Y. Cohen
Publikováno v:
Cell Reports, Vol 14, Iss 2, Pp 234-242 (2016)
SummaryMice overexpressing the longevity protein SIRT6 or deficient for the liver’s most prevalent microRNA miR-122 display a similar set of phenotypes, including improved lipid profile and protection against damage linked to obesity. Here, we show
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a96d72dc850055c686f5a602d1aaac4a
https://doi.org/10.1016/j.celrep.2015.12.023
https://doi.org/10.1016/j.celrep.2015.12.023