Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Sindy Kueh"'
Autor:
Asma Redwan, Leonit Kiriaev, Sindy Kueh, John W. Morley, Peter Houweling, Ben D. Perry, Stewart I. Head
Publikováno v:
Frontiers in Physiology, Vol 14 (2023)
Introduction: It has been proposed that an increased susceptivity to oxidative stress caused by the absence of the protein dystrophin from the inner surface of the sarcolemma is a trigger of skeletal muscle necrosis in the destructive dystrophin defi
Externí odkaz:
https://doaj.org/article/a181e69843b34752b923fd38b92ebb05
Autor:
Leonit Kiriaev, Sindy Kueh, John W. Morley, Kathryn N. North, Peter J. Houweling, Stewart I. Head
Publikováno v:
Frontiers in Physiology, Vol 12 (2021)
Duchenne muscular dystrophy is caused by the absence of the protein dystrophin from skeletal muscle and is characterized by progressive cycles of necrosis/regeneration. Using the dystrophin deficient mdx mouse model, we studied the morphological and
Externí odkaz:
https://doaj.org/article/f0bb11040720471a978812368c2c20d7
Autor:
Stephen Chan, Sindy Kueh, John W. Morley, Stewart I. Head, Leonit Kiriaev, Peter J. Houweling, Kathryn N. North
Publikováno v:
American Journal of Physiology-Cell Physiology. 321:C704-C720
Duchenne muscular dystrophy (DMD) is the second most common fatal genetic disease in humans and is characterized by the absence of a functional copy of the protein dystrophin from skeletal muscle. In dystrophin-negative humans and rodents, regenerate
There is a lack of consensus in the literature regarding the effects of dystrophin deficiency on the Ca2+-handling properties of the SR in mdx mice, an animal model of Duchenne muscular dystrophy. One possible reason for this is that only a few studi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7ed7c139e06addc70caa02f55af69f22
https://doi.org/10.1101/2021.11.29.470495
https://doi.org/10.1101/2021.11.29.470495
Autor:
Stewart I. Head, Sindy Kueh, Leonit Kiriaev, John W. Morley, Peter J. Houweling, Kathryn N. North
Duchenne muscular dystrophy is caused by the absence of the protein dystrophin from skeletal muscle and is characterized by progressive cycles of necrosis/regeneration. Using the dystrophin deficient mdx mouse model we studied the morphological and c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9d38cee12091297421bf8e27cde50f42
https://doi.org/10.1101/2021.09.07.459226
https://doi.org/10.1101/2021.09.07.459226
Autor:
John W. Morley, Peter J. Houweling, Stewart I. Head, Kathryn N. North, Leonit Kiriaev, Sindy Kueh
Publikováno v:
American Journal of Physiology-Cell Physiology. 314:C662-C674
A striking pathological feature of dystrophinopathies is the presence of morphologically abnormal branched skeletal muscle fibers. The deterioration of muscle contractile function in Duchenne muscular dystrophy is accompanied by both an increase in n
Autor:
Peter J. Houweling, Sindy Kueh, Leonit Kiriaev, Stewart I. Head, John W. Morley, Kathryn N. North
Publikováno v:
Biophysical Journal. 118:121a
Autor:
Jonathan Tapson, Yossi Buskila, André van Schaik, Paul P. Breen, Sindy Kueh, John W. Morley, Orsolya Kékesi, Alba Bellot-Saez, Morven A. Cameron
Publikováno v:
Journal of visualized experiments : JoVE. (120)
Acute neuronal tissue preparations, brain slices and retinal wholemount, can usually only be maintained for 6 - 8 h following dissection. This limits the experimental time, and increases the number of animals that are utilized per study. This limitat
Autor:
Mohammad S. Zaman, Cindy Kersaitis, Sindy Kueh, Gabriele Bobek, Ming J. Wu, Yossi Buskila, Adam J. Johnson, Trevor D. Bailey
Publikováno v:
Metallomics : integrated biometal science. 8(1)
Protein kinase CK2 is a pleiotropic tetrameric enzyme, regulating numerous biological processes from cell proliferation to stress response. This study demonstrates for the first time that CK2 is involved in the regulation of metal uptake and toxicity
Publikováno v:
Neurobiology of Disease, Vol 43, Iss 3, Pp 558-564 (2011)
Duchenne muscular dystrophy (DMD) is caused by the absence of a functional transcript of the protein dystrophin. DMD is associated with a range of cognitive deficits that are thought to result from a lack of the protein dystrophin in brain structures