Výsledky vyhledávání - "Simona DeFranco"

Deficiency of the Fas apoptosis pathway without Fas gene mutations is a familial trait predisposing to development of autoimmune diseases and cancer

Autor: Caterina Gambaruto, Irma Dianzani, Sara Bonissoni, Umberto Dianzani, Guido Valesini, Franco Merletti, Fabrizio Conti, Ugo Ramenghi, Simona DeFranco, Flavia Bottarel, D DiFranco, Giuseppe Migliaretti, Roberta Priori

Publikováno v: Blood. 95:3176-3182

Fas/Apo-1 (CD95) triggers programmed cell death (PCD) and is involved in immune response control and cell-mediated cytotoxicity. In the autoimmune/lymphoproliferative syndrome (ALPS), inherited loss-of-function mutations of the Fas gene cause nonmali

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::5adcc54455e18de1e4d897a4855602e4
https://doi.org/10.1182/blood.v95.10.3176

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The Cell Death-Inducing Ability of Glycoprotein 120 from Different HIV Strains Correlates with Their Ability to Induce CD4 Lateral Association with CD95 on CD4+ T Cells

Autor: Fabio Malavasi, Manuela Bragardo, Ugo Ramenghi, Sara Bonissoni, Simona DeFranco, Umberto Dianzani, Donatella Buonfiglio, María José Feito, Flavia Bottarel, Thea Bensi

Publikováno v: AIDS Research and Human Retroviruses. 15:1255-1263

CD4 cross-linking by HIV gp120 triggers CD4+ T cell death. Several authors have suggested that this effect is mediated by CD95, but this possibility is debated by other authors. In a previous work, we found by co-capping that gp120(451) and gp120MN,

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d3bd7d88c73b28d945f900c8b0ef9899
https://doi.org/10.1089/088922299310151

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Defective function of Fas in patients with type 1 diabetes associated with other autoimmune diseases

Autor: Umberto Dianzani, Alfredo Brusco, Sara Bonissoni, Claudio Pignata, Andrea Corrias, Gianni Bona, Giuseppe Loffredo, Franco Cerutti, Francesco Cadario, Simona DeFranco, Flavia Bottarel, Ivana Rabbone, Ugo Ramenghi

Publikováno v: Scopus-Elsevier

Fas (CD95) triggers programmed cell death and is involved in cell-mediated cytotoxicity and in shutting off the immune response. Inherited loss-of-function mutations hitting the Fas system cause the autoimmune/lymphoproliferative syndrome (ALPS). We

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bd4d7ee3a4ecc497564b375dd4362e04
http://hdl.handle.net/11588/155918

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