Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Simon P. M. Lems"'
Autor:
Friso L H Muntinghe, Wayel H Abdulahad, Minke G Huitema, Jeffrey Damman, Marc A Seelen, Simon P M Lems, Bouke G Hepkema, Gerjan Navis, Johanna Westra
Publikováno v:
PLoS ONE, Vol 7, Iss 2, p e31257 (2012)
BACKGROUND: In patients with end stage renal disease (ESRD) we observed protection from inflammation-associated mortality in CCR5Δ32 carriers, leading to CCR5 deficiency, suggesting impact of CCR5Δ32 on inflammatory processes. Animal studies have s
Externí odkaz:
https://doaj.org/article/e1d288459b5747bb8376eeac635df6ec
Autor:
Aiko P. J. de Vries, Rutger M. van Ree, Laura V. de Vries, Stephan J. L. Bakker, Willem J. van Son, Reinold O. B. Gans, Simon P. M. Lems, Dorien M. Zelle, Jan P. Schouten, Leendert H. Oterdoom
Publikováno v:
ResearcherID
Medical Science Monitor : International Medical Journal of Experimental and Clinical Research
Medical Science Monitor, 17(11), CR609-CR617. INT SCIENTIFIC INFORMATION, INC
Medical Science Monitor : International Medical Journal of Experimental and Clinical Research
Medical Science Monitor, 17(11), CR609-CR617. INT SCIENTIFIC INFORMATION, INC
Background: Cytomegalovirus(CMV) is a risk factor for rejection and mortality soon after renal transplantation. Little is known about its consequences longer after transplantation. We prospectively investigated whether latent CMV infection is a risk
Autor:
Jan P. Schouten, Theo Borghuis, Winston W. Bakker, Gerjan Navis, M. A. Seelen, Rijk O. B. Gans, Stephan J. L. Bakker, Jan A. Krikken, Astrid Klooster, Simon P. M. Lems, Rutger M. van Ree
Publikováno v:
Transplant International. 23:805-812
Chronic low-grade inflammation is involved in late renal transplant dysfunction. Recent studies suggest a role for hemopexin, an acute phase protein, in kidney damage. We investigated whether hemopexin activity (Hx) predicts graft failure in renal tr
Autor:
Coen A. Stegeman, Jan Willem Cohen-Tervaert, Patricia M. Stassen, Bouke G. Hepkema, Simon P. M. Lems, Cees G. M. Kallenberg
Publikováno v:
Rheumatology, 48(6), 622-625. Oxford University Press
Objectives. As the HLA system is involved in recognition of self and non-self, an association with the development of ANCA-associated vasculitis (AAV) seems probable. In this study, the relation between HLA antigens and AAV and its severity were inve
Autor:
Stephan J. L. Bakker, Simon P. M. Lems, Jaap J. Homan van der Heide, Paul E. de Jong, Esther Meijer, Willem J. van Son, Joachim Struck, Ron T. Gansevoort
Publikováno v:
Transplantation, 88(4), 561-567. LIPPINCOTT WILLIAMS & WILKINS
Transplantation, 88(4), 561-567. Lippincott Williams and Wilkins
Transplantation, 88(4), 561-567. Lippincott Williams and Wilkins
Background. Chronically elevated vasopressin (VP) plasma levels have been shown to induce accelerated renal function decline in rats with chronic renal failure. Whether endogenous VP is a renal risk factor in humans has not been investigated yet. We
Autor:
Maarten H. L. Christiaans, Johan W. de Fijter, Jaap J. Homan van der Heide, Ella M. van den Berg-Loonen, Frederike J. Bemelman, R. J. Hene, Henderikus G. Otten, Andries J. Hoitsma, Willem Weimar, Wil A. Allebes, Jeroen Aalten, Johannes P. van Hooff, Frans H.J. Claas, Bouke G. Hepkema, N M Lardy, Simon P. M. Lems
Publikováno v:
Nephrology Dialysis Transplantation, 24(8), 2559-2566. Oxford University Press
Nephrology Dialysis Transplantation, 24(8), 2559. Oxford University Press
Nephrology, Dialysis, Transplantation, 24, 2559-66
Nephrology, dialysis, transplantation, 24(8), 2559-2566. Oxford University Press
Nephrology, Dialysis, Transplantation, 24, 8, pp. 2559-66
Nephrology Dialysis Transplantation, 24(8), 2559. Oxford University Press
Nephrology, Dialysis, Transplantation, 24, 2559-66
Nephrology, dialysis, transplantation, 24(8), 2559-2566. Oxford University Press
Nephrology, Dialysis, Transplantation, 24, 8, pp. 2559-66
Contains fulltext : 81930.pdf (Publisher’s version ) (Closed access) BACKGROUND: Female renal transplant candidates are prone to be sensitized by prior pregnancies, and undetected historical sensitization might decrease transplantation outcome. Hyp
Autor:
Nic J. G. M. Veeger, Erik A M Verschuuren, Jaap J. Homan van der Heide, Hanneke C. Kluin-Nelemans, Gustaaf W. van Imhoff, Nicolaas A. Bakker, Bouke G. Hepkema, Philip M. Kluin, Willem J. van Son, Simon P. M. Lems
Publikováno v:
Transplantation, 80(5), 595-599. LIPPINCOTT WILLIAMS & WILKINS
Transplantation, 80(5), 595-599. Lippincott Williams and Wilkins
Transplantation, 80(5), 595-599. Lippincott Williams and Wilkins
Although several risk factors for posttransplant lymphoproliferative disease (PTLD) after solid organ transplantation have been identified, the immunosuppressive regimen probably as most important one, their exact pathogenic role and relevance is sti
Autor:
Simon P. M. Lems, Aad P. van den Berg, Robert J. Porte, Elisabeth M. TenVergert, Koert P. de Jong, Bouke G. Hepkema, Charles M. A. Bijleveld, Annette S. H. Gouw, Maarten J. H. Slooff, Paul M. J. G. Peeters, Egbert Sieders
Publikováno v:
Liver Transplantation, 11(12), 1541-1549. Wiley
The aim of this study was to analyze the effect of human leukocyte antigen (HLA) class I and HLA-DR mismatching, sharing cross-reactive antigen groups (CREGs), and sharing HLA-DR antigens on the outcome after pediatric liver transplantation. Outcome
Identification of three new DRB1 alleles, DRB1*0107, *0425 and *13012 and confirmation of DRB4*01033
Publikováno v:
Tissue Antigens. 61:398-402
The characterization of three novel DRB1 alleles is described, DRB1*0107, DRB1*0425 and DRB1*13012 as well as confirmation of DRB4*01033. Two alleles, DRB1*0107 and *0425, showed amino acid differences with previously identified HLA molecules. In DRB
Autor:
Christien Voorter, E. M. T. Mulkers, Simon P. M. Lems, E.M. Van Den Berg‐Loonen, Bouke G. Hepkema
Publikováno v:
Tissue Antigens. 58:42-46
In our recent study using high-resolution HLA-B locus typing by sequence-based typing (SBT) we identified 9 new alleles in a total of 355 unrelated individuals (4). Three of them concerned an allele belonging to the B22 group. One of them, B*5607, sh