Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Simon Crook"'
Autor:
Simon Crook
Publikováno v:
Religions, Vol 9, Iss 11, p 375 (2018)
This paper proceeds from the concurrent interpretation of two distinct, apparently unrelated disciplinary contexts, at the crossroads of the positivism of archaeology and the imaginary world of literature. The character of the reciprocal relationship
Externí odkaz:
https://doaj.org/article/c7103e176c514bdfbf087bf7b78394a3
Autor:
Tarek M A Elsaba, Luisa Martinez-Pomares, Adrian R Robins, Simon Crook, Rashmi Seth, Darryl Jackson, Amy McCart, Andrew R Silver, Ian P M Tomlinson, Mohammad Ilyas
Publikováno v:
PLoS ONE, Vol 5, Iss 5, p e10714 (2010)
CD133 is a membrane molecule that has been, controversially, reported as a CSC marker in colorectal cancer (CRC). In this study, we sought to clarify the expression and role of CD133 in CRC. Initially the size of the CD133-expressing (CD133+) populat
Externí odkaz:
https://doaj.org/article/e5e0e815a30e4c1e8372e3bd0693a63b
Autor:
Alexander T. Brasier, Simon Crook, John Parnell, Paul F. Dennis, Timothy Culwick, John Still, David Muirhead, David Wacey, Adrian J. Boyce, Stephen A. Bowden, Martin D. Brasier
Publikováno v:
Precambrian Research. 328:309-320
Putative stromatolites and associated carbonate minerals in 1.1 Ga Stoer Group lacustrine sedimentary rocks were analysed to deduce their likely origins. Potential stromatolite examples included finely laminated and sometimes wrinkled carbonate-silic
Publikováno v:
International Journal of Science Education. 37:272-293
Our study capitalized on a unique natural experiment rather than a researcher-designed, randomized experiment whereby, thanks to the Australian Government's Digital Education Revolution, half of grade 9 students in 2008 received laptops and half did
Publikováno v:
Gut. 58:1234-1241
KRAS and BRAF mutations occur in colorectal cancers (CRCs) and are considered mutually exclusive methods of activating the RAS/RAF/MEK/ERK pathway. This pathway is a therapeutic target and KRAS mutation may predict tumour responsiveness. The purpose
Publikováno v:
Journal of Clinical Pathology. 62:598-603
Background: Both the putative modifier gene ATP5a1 and the tumour suppressor gene TP53 are involved in the regulation of apoptosis and may be involved in the development of colorectal cancers. Aims: To investigate the relationship between these genes
Autor:
Ahmed Benhasouna, Mohammad Ilyas, Abdulkader M. Albasri, Rashmi Seth, Abdolrahman S. Nateri, Roger W. Chapman, Simon Crook, Darryl Jackson
Publikováno v:
The Journal of Pathology. 218:57-65
The Tensin gene family encodes proteins thought to modulate integrin function. C-terminal Tensin-like (CTEN) is a member of the Tensin gene family which lacks the N-terminus actin-binding domain. Cten is reported to have both oncogenic and tumour-sup
Publikováno v:
Australasian Journal of Educational Technology. 29
As schools start investigating and investing in the idea of 1:1 iPads and tablets, are there any lessons that can be learnt from recent 1:1 laptop deployments? In Australia, since 2008, 1:1 laptops have been introduced into every secondary school. Th
Autor:
Amy McCart, Rashmi Seth, Andrew Silver, Ian Tomlinson, Mohammad Ilyas, Darryl Jackson, Adrian Robins, Simon Crook, Tarek M. Elsaba, Luisa Martinez-Pomares
Publikováno v:
PLoS ONE, Vol 5, Iss 5, p e10714 (2010)
PLoS ONE
PLoS ONE
CD133 is a membrane molecule that has been, controversially, reported as a CSC marker in colorectal cancer (CRC). In this study, we sought to clarify the expression and role of CD133 in CRC. Initially the size of the CD133-expressing (CD133+) populat
Autor:
Abdulkader, Albasri, Rashmi, Seth, Darryl, Jackson, Ahmed, Benhasouna, Simon, Crook, Abdolrahman S, Nateri, Roger, Chapman, Mohammad, Ilyas
Publikováno v:
The Journal of pathology. 218(1)
The Tensin gene family encodes proteins thought to modulate integrin function. C-terminal Tensin-like (CTEN) is a member of the Tensin gene family which lacks the N-terminus actin-binding domain. Cten is reported to have both oncogenic and tumour-sup