Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Silvia Kelsen"'
Publikováno v:
Frontiers in Behavioral Neuroscience, Vol 18 (2024)
BackgroundDrug seeking behavior occurs in response to environmental contexts and drug-associated cues. The presence of these pervasive stimuli impedes abstinence success. β-adrenergic receptors (β-ARs) have a long-standing historical implication in
Externí odkaz:
https://doaj.org/article/5fb519f7f02c4bbf928eb24e33f0ae86
Publikováno v:
American Journal of Physiology-Renal Physiology. 303:F576-F583
Renal artery stenosis (RAS), the main cause of chronic renovascular disease (RVD), is associated with significant oxidative stress. Chronic RVD induces renal injury partly by promoting renal microvascular (MV) damage and blunting MV repair in the ste
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3890204ab356399578c357329211cedf
https://doi.org/10.1152/ajprenal.00154.2012
https://doi.org/10.1152/ajprenal.00154.2012
Publikováno v:
Hypertension. 52:660-665
The main goal of this study was to determine whether kidney-specific induction of heme oxygenase-1 (HO-1) can prevent the development of angiotensin (Ang) II–dependent hypertension. To test this hypothesis, intrarenal medullary interstitial cathete
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::82ede73e182e40a4bf1d36c4c989c2bc
https://doi.org/10.1161/hypertensionaha.108.114884
https://doi.org/10.1161/hypertensionaha.108.114884
Autor:
Gerald R. McLemore, Silvia Kelsen, David E. Stec, Michael J. Ryan, John M. Rimoldi, Trinity Vera, Rama S.V. Gadepalli
Publikováno v:
American Journal of Hypertension. 21:189-193
BACKGROUND Induction of heme oxygenase-1 (HO-1) attenuates the development of angiotensin II (Ang II)-dependent hypertension in mice. However, the mechanism by which HO-1 lowers blood pressure in this model is not clear. This study was designed to de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2e5758f034753bd2513869509e74031
https://doi.org/10.1038/ajh.2007.29
https://doi.org/10.1038/ajh.2007.29
Autor:
Silvia Kelsen, Alejandro R. Chade
Publikováno v:
American journal of physiology. Renal physiology. 302(10)
Renal microvascular (MV) damage and loss contribute to the progression of renal injury in renovascular disease (RVD). Whether a targeted intervention in renal microcirculation could reverse renal damage is unknown. We hypothesized that intrarenal vas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::369db1dda37faf6d48480afe8a16d484
https://pubmed.ncbi.nlm.nih.gov/22357917
https://pubmed.ncbi.nlm.nih.gov/22357917
Endothelin (ET)-1, a potent renal vasoconstrictor with mitogenic properties, is upregulated by ischemia and has been shown to induce renal injury via the ET-A receptor. The potential role of ET-A blockade in chronic renovascular disease (RVD) has not
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8ef8242eb772b89381a170eb6b4d9833
https://europepmc.org/articles/PMC3129888/
https://europepmc.org/articles/PMC3129888/
Autor:
Alejandro R. Chade, Silvia Kelsen
Publikováno v:
Circulation. Cardiovascular interventions. 3(4)
Background— Percutaneous transluminal renal angioplasty (PTRA) is the most frequent therapeutic approach to resolving renal artery stenosis (RAS). However, renal function recovers in only 30% of the cases. The causes of these poor outcomes are stil
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::601ec1c97bc3562dcb7dc5671d337710
https://pubmed.ncbi.nlm.nih.gov/20587789
https://pubmed.ncbi.nlm.nih.gov/20587789
Publikováno v:
The FASEB Journal. 24
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::90f530084990b92e1035c2cc0f33f325
https://doi.org/10.1096/fasebj.24.1_supplement.774.4
https://doi.org/10.1096/fasebj.24.1_supplement.774.4
Publikováno v:
The FASEB Journal. 24
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e507a7291405cb1ec87b7b787fdcf207
https://doi.org/10.1096/fasebj.24.1_supplement.774.5
https://doi.org/10.1096/fasebj.24.1_supplement.774.5
Background. Renal artery stenosis (RAS) causes renal injury partly via microvascular (MV) endothelial dysfunction and damage. Vascular endothelial growth factor (VEGF) is crucial for preservation of microvasculature and promotes vascular proliferatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d8b7efded00ca14556fccea675a81d09
https://europepmc.org/articles/PMC2902859/
https://europepmc.org/articles/PMC2902859/