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pro vyhledávání: '"Sillence, Daniel J"'
Autor:
Lachmann, Robin H *, te Vruchte, Danielle, Lloyd-Evans, Emyr, Reinkensmeier, Gabriele, Sillence, Daniel J, Fernandez-Guillen, Luisa, Dwek, Raymond A, Butters, Terry D, Cox, Timothy M, Platt, Frances M
Publikováno v:
In Neurobiology of Disease 2004 16(3):654-658
Autor:
Neville, David C.A, Coquard, Virginie, Priestman, David A, te Vruchte, Danielle J.M, Sillence, Daniel J, Dwek, Raymond A, Platt, Frances M, Butters, Terry D
Publikováno v:
In Analytical Biochemistry 2004 331(2):275-282
Autor:
Sillence, Daniel J. *, †, Raggers, René J. *, Neville, David C.A. †, Harvey, David J. †, van Meer, Gerrit *, 1
Publikováno v:
In Journal of Lipid Research August 2000 41(8):1252-1260
Autor:
Wheeler, S., Sillence, Daniel J.
Gaucher disease is caused by mutations in the Gba1 gene encoding an acid β-glucocerebrosidase (GBA1), the lysosomal hydrolase which breaks down glucosylceramide (GlcCer). In Gaucher type 1 disease the accumulation of this simple glycolipid is mainly
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::6780f9b22b951de517de27f6a32853ae
http://hdl.handle.net/2086/11005
http://hdl.handle.net/2086/11005
Autor:
Sillence, Daniel J.
BACKGROUND: Recent evidence suggests that the activation of a non-specific lipid scramblase during apoptosis induces the flipping of sphingomyelin from the cell surface to the cytoplasmic leaftet of the plasma membrane. Inner leaflet sphingomyelin is
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::8b78272c3ccff0b9b11ba4b57051faed
http://hdl.handle.net/2086/2874
http://hdl.handle.net/2086/2874
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