Zobrazeno 1 - 10
of 37
pro vyhledávání: '"Si-yu Zeng"'
Publikováno v:
Guoji Yanke Zazhi, Vol 23, Iss 10, Pp 1662-1666 (2023)
Maintaining the homeostasis of intracellular components degradation and recycling, autophagy is a critical control mechanism of cellular quality. It promotes the degradation of cell components to provide nutrients and energy for cellular metabolism i
Externí odkaz:
https://doaj.org/article/73eea55f12894ba3bd03dd404d34f461
Publikováno v:
Guoji Yanke Zazhi, Vol 21, Iss 4, Pp 718-721 (2021)
AIM: To investigate effects of different corneal protective agents on ocular surface in vitrectomy in patients with diabetes.METHODS: Totally 90 patients(90 eyes)with diabetes who received vitrectomy were randomly divided into HPMC group(30 eyes), SH
Externí odkaz:
https://doaj.org/article/2dcbf6c13e814b55a72eda0604e1eff7
Publikováno v:
Guoji Yanke Zazhi, Vol 20, Iss 3, Pp 576-579 (2020)
AIM: To compare the effect of platelet-rich fibrin(PRF)grafts and conjunctival-limbal autografts in pterygium excision.METHODS: Totally 62 cases(62 eyes)of primary pterygium were randomly divided into group 1(32 eyes)and group 2(30 eyes). After ptery
Externí odkaz:
https://doaj.org/article/d89c633edbf34ce4bfed0ee4c2873086
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 109, Iss , Pp 1907-1914 (2019)
NADPH oxidases (Noxs) 1/4 dual inhibitor GKT137831 prevents hypertensive cardiac remodelling in angiotensin II-infused transgenic mice with cardiomyocyte-specific human Nox4 (c-hNo x 4 Tg); however, further research is still required to determine the
Externí odkaz:
https://doaj.org/article/dd609a6b35544ac59682b637da7fee37
Publikováno v:
Mediators of Inflammation, Vol 2020 (2020)
Oxidative stress, inflammation, and hypertension constitute a self-perpetuating vicious circle to exacerbate hypertension and subsequent hypertensive cardiac hypertrophy. NADPH oxidase (Nox) 1/4 inhibitor GKT137831 alleviates hypertensive cardiac hyp
Externí odkaz:
https://doaj.org/article/397e6e1f43d049eba6f34dd70517e003
Publikováno v:
PPAR Research, Vol 2018 (2018)
The peroxisome proliferator-activated receptor-α (PPAR-α) agonist fenofibrate ameliorates cardiac hypertrophy; however, its mechanism of action has not been completely determined. Our previous study indicated that a disintegrin and metalloproteinas
Externí odkaz:
https://doaj.org/article/48def44a3c46418d8e0152925191b7bb
Publikováno v:
Mediators of Inflammation, Vol 2018 (2018)
Calcitonin gene-related peptide (CGRP) has a potent protective action on the cardiovascular system; however, little is known about the role of CGRP in angiotensin II- (Ang II-) induced inflammation of vascular smooth muscle cells (VSMCs). This study
Externí odkaz:
https://doaj.org/article/81b9c53d04154306b4f62dab5901b7e3
Publikováno v:
Journal of Cellular and Molecular Medicine
Rutaecarpine attenuates hypertensive cardiac hypertrophy in the rats with abdominal artery constriction (AAC); however, its mechanism of action remains largely unknown. Our previous study indicated that NADPH oxidase 4 (Nox4) promotes angiotensin II
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13686063e91317ac8979caa90820ecd4
https://doi.org/10.1111/jcmm.14308
https://doi.org/10.1111/jcmm.14308
Publikováno v:
Mediators of Inflammation, Vol 2020 (2020)
Mediators of Inflammation
Mediators of Inflammation
Oxidative stress, inflammation, and hypertension constitute a self-perpetuating vicious circle to exacerbate hypertension and subsequent hypertensive cardiac hypertrophy. NADPH oxidase (Nox) 1/4 inhibitor GKT137831 alleviates hypertensive cardiac hyp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa975777eab3fab185f60b2b9e58de75
Publikováno v:
Bioengineered. 8:124-128
Plant hormones are a class of organic substances which are synthesized during the plant metabolism. They have obvious physiological effect on plant growth at very low concentrations. Generally, plant hormones are mainly divided into 5 categories: aux
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::14c4c4f27a709ea542900f1aa1cbf0fe
https://doi.org/10.1080/21655979.2016.1212138
https://doi.org/10.1080/21655979.2016.1212138