Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Shweta Hakre"'
Publikováno v:
HemaSphere, Vol 7, p e03128c0 (2023)
Externí odkaz:
https://doaj.org/article/a7d6f40ce0f544fb9007d8fb1c32b5e2
Publikováno v:
PLoS Biology, Vol 13, Iss 11, p e1002302 (2015)
[This corrects the article DOI: 10.1371/journal.pbio.1001206.].
Externí odkaz:
https://doaj.org/article/1ccccc5e2cfe41f491b9b3fae9fafffd
Publikováno v:
PLoS Biology, Vol 9, Iss 11, p e1001206 (2011)
Persistence of a reservoir of latently infected memory T cells provides a barrier to HIV eradication in treated patients. Several reports have implicated the involvement of SWI/SNF chromatin remodeling complexes in restricting early steps in HIV infe
Externí odkaz:
https://doaj.org/article/d9aea4dfd971430eb0fbdbd05dfd2c4d
Publikováno v:
Journal of the National Comprehensive Cancer Network. 21:HSR23-119
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::61584a1fc882e35e633cf2a851962221
https://doi.org/10.6004/jnccn.2022.7221
https://doi.org/10.6004/jnccn.2022.7221
Autor:
Lindsey Elizabeth Roeker, Maral DerSarkissian, Kellie Ryan, Yan Chen, Mei Sheng Duh, Svea K. Wahlstrom, Shweta Hakre, Louise Yu, Helen Guo, Anthony Mato
Publikováno v:
Blood Advances.
Novel agents, including Bruton tyrosine kinase inhibitors (BTKis), have become the standard of care for patients with chronic lymphocytic leukemia (CLL). We conducted a real-world retrospective analysis of CLL patients treated with acalabrutinib vs i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::496409d06453b1264f0f320ac7092cf9
https://doi.org/10.1182/bloodadvances.2023009739
https://doi.org/10.1182/bloodadvances.2023009739
Autor:
Lindsey E. Roeker, Maral DerSarkissian, Kellie J. Ryan, Yan Chen, Svea K. Wahlstrom, Shweta Hakre, Louise Yu, Helen Guo, Anthony R. Mato
Publikováno v:
Blood. 140:4156-4158
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::735fc4b7ed41f37e74f9d89cbb1e99df
https://doi.org/10.1182/blood-2022-167213
https://doi.org/10.1182/blood-2022-167213
Publikováno v:
Antiviral Chemistry and Chemotherapy. 23:145-149
Persistence of HIV-1 in latently infected CD4+ T-cells prevents eradication in HIV-infected treated patients. Latency is characterized by a reversible silencing of transcription of integrated HIV-1. Several molecular mechanisms have been described wh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c898017f570b6d49e413da0040ad331e
https://doi.org/10.3851/imp2551
https://doi.org/10.3851/imp2551
Autor:
Robert F. Siliciano, Erik Verschueren, Ryan J. Conrad, Jonathan K.L. Chan, Nevan J. Krogan, Emilie Besnard, Warner C. Greene, Jonathan S. Weissman, Michael C. Bassik, Martin Kampmann, J. Peter Svensson, Shweta Hakre, Melanie Ott, Andrea Gramatica, Hyung W. Lim, Nina N. Hosmane, Emilie Battivelli, Eric Verdin, Alyssa R. Martin
Publikováno v:
Cell host & microbe, vol 20, iss 6
A population of CD4T lymphocytes harboring latent HIV genomes can persist in patients on antiretroviral therapy, posing a barrier to HIV eradication. To examine cellular complexes controlling HIV latency, we conducted a genome-wide screen with a pool
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::43f5a766ce7745fc5572ac44cd9d8f78
https://europepmc.org/articles/PMC5354304/
https://europepmc.org/articles/PMC5354304/
Publikováno v:
Trends in Microbiology. 21:277-285
Latent HIV persists in CD4(+) T cells in infected patients under antiretroviral therapy (ART). Latency is associated with transcriptional silencing of the integrated provirus and driven, at least in part, by histone deacetylases (HDACs), a family of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d16bf96291f3e32dd7faf901d91ed13d
https://doi.org/10.1016/j.tim.2013.02.005
https://doi.org/10.1016/j.tim.2013.02.005
Autor:
Shweta Hakre, Ananda L. Roy, Amy S. Lee, Min Hong, Jen-Ming Huang, Meng-yin Lin, Peter Baumeister
Publikováno v:
Journal of Biological Chemistry. 280:16821-16828
TFII-I is a signal-induced multi-functional transcription factor that has recently been implicated as a regulatory component of the endoplasmic reticulum (ER) stress response. TFII-I acts through ER stress-induced binding to the ER stress element, wh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8db0b44d4ca620f8dd5d08cf3032ef80
https://doi.org/10.1074/jbc.m413753200
https://doi.org/10.1074/jbc.m413753200