Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Shun Mitsui"'
Autor:
Masahisa Nozaki, Asako Otomo, Shun Mitsui, Suzuka Ono, Ryohei Shirakawa, YongPing Chen, Yutaro Hama, Kai Sato, XuePing Chen, Toshiyasu Suzuki, Hui-Fang Shang, Shinji Hadano
Publikováno v:
eNeurologicalSci, Vol 22, Iss , Pp 100301- (2021)
Amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD) are genetically, pathologically and clinically-related progressive neurodegenerative diseases. Thus far, several SQSTM1 variations have been identified in patients with ALS and FTD
Externí odkaz:
https://doaj.org/article/da9e314e3b97477aaef197375f9e6865
Autor:
Shun Mitsui, Asako Otomo, Masahisa Nozaki, Suzuka Ono, Kai Sato, Ryohei Shirakawa, Hiroaki Adachi, Masashi Aoki, Gen Sobue, Hui-Fang Shang, Shinji Hadano
Publikováno v:
Molecular Brain, Vol 11, Iss 1, Pp 1-16 (2018)
Abstract Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disease characterized by a selective loss of upper and lower motor neurons. Recent studies have shown that mutations in SQSTM1 are linked to ALS. SQSTM1 encodes SQSTM1/p6
Externí odkaz:
https://doaj.org/article/8580ca713204450288e370952c8766f7
Publikováno v:
Crystals, Vol 7, Iss 2, p 52 (2017)
The photoinduced surface relief formation via mass transfer upon irradiation with patterned light has long been a subject of extensive investigation. In azobenzene-containing liquid crystalline materials, UV light irradiation that generates the cis i
Externí odkaz:
https://doaj.org/article/46e4d32efc544711aa954671d65c9817
Autor:
Suzuka Ono, Kai Sato, Asako Otomo, Kento Shimakura, Hiroshi Kimura, Shinji Hadano, Shun Mitsui
Publikováno v:
Neuroscience Research. 174:46-52
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease characterized by selective loss of motor neurons. We have previously shown that autophagosome-like vesicular structures are progressively accumulated in the spinal axons of an ALS mou
Publikováno v:
Biochemical and Biophysical Research Communications. 569:106-111
ALS2, a product of the causative gene for familial amyotrophic lateral sclerosis (ALS) type 2, plays a pivotal role in the regulation of endosome dynamics by activating small GTPase Rab5 via its intrinsic guanine nucleotide-exchange factor activity.
Autor:
Kai Sato, Kyoko Suzuki-Utsunomiya, Shun Mitsui, Suzuka Ono, Kento Shimakura, Asako Otomo, Shinji Hadano
Publikováno v:
Biochemical and biophysical research communications. 638
ALS2/alsin, the causative gene product for a number of juvenile recessive motor neuron diseases, acts as a guanine nucleotide exchange factor (GEF) for Rab5, regulating early endosome trafficking and maturation. It has been demonstrated that ALS2 for
Autor:
Shigeo Wada, Shun Mitsui
Publikováno v:
IEEJ Transactions on Electronics, Information and Systems. 141:592-601
Autor:
Shun Mitsui, Asako Otomo, Kai Sato, Masahito Ishiyama, Kento Shimakura, Chisa Okada-Yamaguchi, Eiji Warabi, Toru Yanagawa, Masashi Aoki, Hui-Fang Shang, Shinji Hadano
Publikováno v:
Neurochemistry international. 158
Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disease characterized by selective loss of motor neurons in the brain and spinal cord. Recent studies have shown that mutations in SQSTM1 are linked to ALS. It has also been demon
Autor:
Asako, Otomo, Suzuka, Ono, Kai, Sato, Shun, Mitsui, Kento, Shimakura, Hiroshi, Kimura, Shinji, Hadano
Publikováno v:
Neuroscience research. 174
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease characterized by selective loss of motor neurons. We have previously shown that autophagosome-like vesicular structures are progressively accumulated in the spinal axons of an ALS mou
Autor:
Yongping Chen, Suzuka Ono, Toshiyasu Suzuki, Yutaro Hama, Xueping Chen, Asako Otomo, Masahisa Nozaki, Ryohei Shirakawa, Shinji Hadano, Kai Sato, Huifang Shang, Shun Mitsui
Publikováno v:
eNeurologicalSci, Vol 22, Iss, Pp 100301-(2021)
Amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD) are genetically, pathologically and clinically-related progressive neurodegenerative diseases. Thus far, several SQSTM1 variations have been identified in patients with ALS and FTD