Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Shuju Feng"'
Publikováno v:
International Breastfeeding Journal, Vol 18, Iss 1, Pp 1-8 (2023)
Abstract Background The breastfeeding rates of late preterm infants are lower than both term and extremely preterm infants. To explore the interventions of increasing full breast milk feeding rate of hospitalized late preterm infants on the 7th day a
Externí odkaz:
https://doaj.org/article/6aecc14bc8fc4ef7a7db09a3a2cb2696
Autor:
Shuju Feng, Xiaowen Liang, Miguel A Cruz, Hangoc Vu, Zhou Zhou, Naresh Pemmaraju, Jing-Fei Dong, Michael H Kroll, Vahid Afshar-Kharghan
Publikováno v:
PLoS ONE, Vol 8, Iss 8, p e73715 (2013)
Complement factor H (fH) is a plasma protein that regulates activation of the alternative pathway, and mutations in fH are associated with a rare form of thrombotic microangiopathy (TMA), known as atypical hemolytic uremic syndrome (aHUS). A more com
Externí odkaz:
https://doaj.org/article/45d0d74a491a40a38598b930247fe0f1
Publikováno v:
Pain Management Nursing. 22:668-673
Background Venipuncture is a common procedure in the neonatal intensive care unit (NICU) and causes significant pain for neonates. Aim To evaluate the effect of maternal voice on pain caused by venipuncture (including peripheral venipuncture and femo
Autor:
Lisa S. Chen, Varsha Gandhi, Wei Qiao, William G. Wierda, Qi Wu, Prithviraj Bose, Xuelin Huang, Nichole Cruz, Yongying Jiang, Nitin Jain, Philip A. Thompson, Michael J. Keating, Michael H. Kroll, Shuju Feng
Publikováno v:
Blood. 132:2249-2259
Ibrutinib is highly efficacious and used at 420 mg/d for treatment of chronic lymphocytic leukemia (CLL). We previously demonstrated a decline in Bruton’s tyrosine kinase (BTK) protein levels in CLL cells after 1 cycle of ibrutinib, suggesting ibru
Publikováno v:
Platelets. 27:716-718
Paraneoplastic thrombocytosis has been reported in different types of solid tumors, including ovarian epithelial cancer, and found to be associated with a worse outcome. Although the effect of cancer on increasing platelet counts is well documented,
Publikováno v:
Blood. 102:2122-2129
We examined the hypothesis that filamin A binding to the cytoplasmic tail of platelet glycoprotein Ibα (GpIbα) is regulated by pathologic shear stress and modulates von Willebrand factor (VWF)–induced platelet activation. To begin, we examined fi
Autor:
Julio C. Reséndiz, Michael C. Berndt, Michael H. Kroll, Ketia A. Francis, Guilan Ji, Shuju Feng
Publikováno v:
Molecular Pharmacology. 63:639-645
Pathologically elevated shear stress triggers aspirin-insensitive platelet thrombosis. Signaling mechanisms involved in shear-induced platelet thrombosis are not well understood. To investigate these, we examined the hypothesis that functionally impo
Publikováno v:
Blood. 125(6)
Several complement proteins interact with hemostatic factors. We discovered that von Willebrand factor (VWF) acts as a cofactor for factor I–mediated cleavage of complement C3b, thereby shutting down complement activation. The complement regulatory
Publikováno v:
Thrombosis Research. 102:133-142
The extracellular domain of glycoprotein (Gp) Ibalpha serves as the von Willebrand factor (vWf) receptor that triggers shear stress-dependent platelet aggregation. Its intracellular domain associates with actin-binding protein-280 (filamin 1a) that b
Publikováno v:
Blood. 95:551-557
Shear stress causes the platelet glycoprotein (Gp) Ib/IX/V to bind to von Willebrand factor, resulting in platelet adhesion. GpIb/IX/V also functions to stimulate transmembranous signaling, leading to platelet activation and the expression of a ligan