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pro vyhledávání: '"Shreya M. Kanth"'
Autor:
Han Wen, Julio A. Huapaya, Shreya M. Kanth, Junfeng Sun, Brianna P. Matthew, Simone C. Lee, Michael Do, Marcus Y. Chen, Ashkan A. Malayeri, Anthony F. Suffredini
Publikováno v:
Journal of Imaging, Vol 9, Iss 8, p 150 (2023)
(1) Background: A reduction in the diffusion capacity of the lung for carbon monoxide is a prevalent longer-term consequence of COVID-19 infection. In patients who have zero or minimal residual radiological abnormalities in the lungs, it has been deb
Externí odkaz:
https://doaj.org/article/dc0c4f6cb00649ee9c1eeeaf2ee35219
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
Semaphorins are a group of proteins that have been studied extensively for their critical function in neuronal development. They have been shown to regulate airway development, tumorigenesis, autoimmune diseases, and the adaptive immune response. Not
Externí odkaz:
https://doaj.org/article/544f3abf93bb4c6e824cc4366acdcfaa
Autor:
Shreya M. Kanth, Parizad Torabi-Parizi
Publikováno v:
Crit Care Med
There is an unmet need to improve the description of the state of T-cell exhaustion in patients with sepsis, its reproducibility and correlation with the outcomes before including immunotherapy (like recombinant interleukin-7 or immune checkpoint inh
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
Frontiers in Immunology
Frontiers in Immunology
Semaphorins are a group of proteins that have been studied extensively for their critical function in neuronal development. They have been shown to regulate airway development, tumorigenesis, autoimmune diseases, and the adaptive immune response. Not
Autor:
Shreya M. Kanth, Cumhur Y Demirkale, Anthony F. Suffredini, Salina Gairhe, Parizad Torabi-Parizi, R.J. Noveck
Publikováno v:
C59. DISSECTING ACUTE LUNG INJURY AND ARDS.
Autor:
Akihiko Hoji, Shreya M. Kanth, James D. Londino, Rama K. Mallampalli, Nathaniel M. Weathington, Sally E. Wenzel, Qiaoke Gong, John B. Trudeau, Mauricio Rojas
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology. 57:346-354
IL-25 and IL-4 signaling in the setting of infection or allergic responses can drive Type 2 inflammation. IL-25 requires the IL-17 receptor B (IL-17Rb) to mediate signaling through nuclear factor κ B (NF-κB) transcriptional activation. Despite the