Zobrazeno 1 - 10
of 31
pro vyhledávání: '"Shou-Ching Shih"'
Autor:
Harold F. Dvorak, Lawrence F. Brown, Janice A. Nagy, Lay-Hong Ang, Guanmei Liu, Dan Li, Andrew Zukauskas, Shou-Ching Shih
Transmembrane-4-L-six-family-1 (TM4SF1) was originally described as a cancer cell protein. Here, we show that it is highly expressed in the vascular endothelium of human cancers and in a banded pattern in the filopodia of cultured endothelial cells (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::49cf912f8286be5ff0ded7cde4e625e9
https://doi.org/10.1158/0008-5472.c.6499659.v1
https://doi.org/10.1158/0008-5472.c.6499659.v1
Autor:
Harold F. Dvorak, Lawrence F. Brown, Janice A. Nagy, Lay-Hong Ang, Guanmei Liu, Dan Li, Andrew Zukauskas, Shou-Ching Shih
Supplementary Figure 1 from The L6 Protein TM4SF1 Is Critical for Endothelial Cell Function and Tumor Angiogenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::052a6cf16a442c6e8a76bb8f3bee0c3e
https://doi.org/10.1158/0008-5472.22380750.v1
https://doi.org/10.1158/0008-5472.22380750.v1
Autor:
Peter J. Zwiers, Grietje Molema, Shou-Ching Shih, Manoj Bhasin, Amber N. Stratman, Janice A. Nagy, George E. Davis, Peter Oettgen, William C. Aird, Alexandra Le Bras, Anastasia Sacharidou, Lei Yuan, Katherine Spokes
Publikováno v:
Blood, 118(4), 1145-1153. AMER SOC HEMATOLOGY
ERG is a member of the ETS transcription factor family that is highly enriched in endothelial cells (ECs). To further define the role of ERG in regulating EC function, we evaluated the effect of ERG knock-down on EC lumen formation in 3D collagen mat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::21f1280183631b267c3960f2307de391
https://hdl.handle.net/11370/407ad839-b16c-4d73-994b-16f4c2d8f214
https://hdl.handle.net/11370/407ad839-b16c-4d73-994b-16f4c2d8f214
Autor:
Janice A. Nagy, Pamela Johnson, Christina K. Chan, Thomas N. Wight, Lawrence F. Brown, Shou-Ching Shih, Yineng Fu, Harold F. Dvorak
Publikováno v:
Journal of Histochemistry & Cytochemistry. 59:463-473
Malignant tumors and chronic inflammatory diseases induce angiogenesis by overexpressing vascular endothelial growth factor A (VEGF-A/VPF). VEGF-A-induced pathological angiogenesis can be mimicked in immunoincompetent mice with an adenoviral vector e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::55f3e94bc2a14e94348a965a41702e04
https://doi.org/10.1369/0022155411401748
https://doi.org/10.1369/0022155411401748
Autor:
Adam S. Curatolo, Andrew C. Dudley, Taturo Udagawa, Shou-Ching Shih, Marsha A. Moses, Michael Klagsbrun, Juan M. Melero-Martin
Publikováno v:
Blood. 116:3367-3371
The hypothesis that bone marrow–derived, circulating endothelial cells incorporate into tumor blood vessels is unresolved. We have measured the numbers of bone marrow–derived versus resident endothelial cells in spontaneous prostate cancers durin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5d9b84924641e1ea7430cc19a511db6d
https://doi.org/10.1182/blood-2010-02-271122
https://doi.org/10.1182/blood-2010-02-271122
Publikováno v:
Journal of Investigative Dermatology. 130(5):1307-1317
UV light-based damage to skin cells can cause photoaging and skin cancer. A major cause of UV light-induced damage to skin is increased free radicals, such as superoxides. Increased superoxides can cause oxidative and nitrative damage to cell compone
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04a1622a3d12a9ac3fcbf213acbf728f
Publikováno v:
Seminars in Thrombosis and Hemostasis. 36:321-331
The blood vessels supplying tumors are strikingly heterogeneous and differ from their normal counterparts with respect to organization, structure, and function. Six distinctly different tumor vessel types have been identified, and much has been learn
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ec95296ee33f7ac9275dfd882083243b
https://doi.org/10.1055/s-0030-1253454
https://doi.org/10.1055/s-0030-1253454
Autor:
Kohei Matsuda, Chikara Muraki, Kunihiko Tsuchiya, Takuro Kurosu, Tomoshige Akino, Yasuhiro Hida, Shou-Ching Shih, Yasunori Totsuka, Kyoko Hida, Michael Klagsbrun, Noritaka Ohga, Masanobu Shindoh
Publikováno v:
Biochemical and Biophysical Research Communications. 394:947-954
Tumor angiogenesis is necessary for solid tumor progression and metastasis. Increasing evidence indicates that tumor endothelial cells (TECs) are more relevant to the study of tumor angiogenesis than normal endothelial cells (NECs) because their morp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ee86a79edb2741b5e82795c04d8d81f7
https://doi.org/10.1016/j.bbrc.2010.03.089
https://doi.org/10.1016/j.bbrc.2010.03.089
Autor:
Shou-Ching Shih, Ju Liu, Yoshiaki Okada, Erzsébet Ravasz Regan, Katherine Spokes, Takashi Minami, Dan Li, Enjing Jin, William C. Aird, Lauren Janes, David L. Beeler, Kiichiro Yano, Vesna Nikolova-Krstevski, Peter Oettgen, Lei Yuan, Jun-ichi Suehiro
Publikováno v:
Blood. 114:5557-5566
Vascular endothelial growth factor receptor 1 (VEGFR1) is a marker for endothelial-specific gene expression. We previously reported that the human VEGFR1 promoter (between −748 and +284) contains information for expression in the intact endothelium
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73688cffa4fa86ae60855e78b273f93e
https://doi.org/10.1182/blood-2009-05-220434
https://doi.org/10.1182/blood-2009-05-220434
Autor:
Robert J. Nadeau, Md. Ruhul Abid, Shou-Ching Shih, Dan Li, William C. Aird, Takashi Minami, Katherine Spokes
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 28:2042-2048
Objective— Recently, we reported that the forkhead transcription factor, FKHR/FOXO1, is required for vascular endothelial growth factor (VEGF)–mediated upregulation of a number of genes in endothelial cells. Here, we tested the hypothesis that he
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c46032e0c3472c4b0ee96aa257f7a07c
https://doi.org/10.1161/atvbaha.108.175109
https://doi.org/10.1161/atvbaha.108.175109