Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Shoshana Merchav"'
Autor:
Shoshana Merchav, Natalie Abramov, Doron Shinar, Mitchell Shirvan, Miron Weinreb, Aharon Schwartz, Irene Ginis
Publikováno v:
BioResearch Open Access, Vol 1, Iss 2, Pp 69-78 (2012)
BioResearch Open Access
BioResearch Open Access
Tissue-engineered bone grafts seeded with mesenchymal stem cells (MSCs) have been sought as a replacement for bone grafts currently used for bone repair. For production of osteogenic constructs, MSCs are isolated from bone marrow (BM) or other tissue
Aplastic anemia associated with in vitro inhibition of erythropoiesis by bone marrow-adherent cells*
Publikováno v:
European Journal of Haematology. 41:429-433
A patient with aplastic anemia that evolved following pure red cell aplasia is described. Cultures of the patient's marrow cells revealed greatly reduced numbers of primitive (BFU-E) and relatively mature (CFU-E) erythroid progenitors, but normal num
Autor:
Michael Aviram, Tony Hayek, Mira Rosenblat, Raymond Coleman, Rachel Levy, Jacob Vaya, Shoshana Merchav, Paula Belinky
Publikováno v:
Journal of Biological Chemistry. 274:13790-13799
Macrophage-mediated oxidation of low density lipoprotein (LDL) is considered to be of major importance in early atherogenesis; therefore, intervention means to inhibit this process are being extensively studied. In the present study, we questioned th
Autor:
Shoshana Merchav, Akihiko Yoshimura, Yael Cohen, Akira Matsumoto, Yael Sandowski, Arieh Gertler, Daniel Helman
Publikováno v:
FEBS Letters. 441:287-291
The ability of five members of the cytokine-inducible SH2 protein family (CIS1–4) and JAK2 binding (JAB) protein to affect prolactin receptor (PRLR)-mediated activity was tested in human 293 embryonic kidney fibroblasts transiently transfected with
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 74:447-452
The erythroid-potentiating effects of biosynthetic human insulin-like growth factor-I (IGF-I) and IGF-II were evaluated and compared in serum-free cultures of human marrow erythroid progenitors. IGF-I and IGF-II enhanced the in vitro growth of relati
Publikováno v:
British Journal of Haematology. 74:424-431
This study investigates the capacity of interleukin-1 alpha (IL-1 alpha), interleukin-1 beta (IL-1 beta) and tumour necrosis factor-alpha (TNF-alpha) to induce interleukin-6 (IL-6) production in freshly isolated myeloma cells (MC) and bone marrow-der
Autor:
G. Shkolnik, Judith Chezar, Batya Kristal, Joseph Manaster, Revital Shurtz-Swirski, Shifra Sela, R. Sharon, Shoshana Merchav, Galina Shapiro, Shaul M. Shasha
Publikováno v:
Nephron. 88(3)
A previous study from our laboratory has shown that erythropoietin (EPO), beside its traditional role in erythropoiesis, acts as an alleviator of oxidative stress and inflammation in chronic hemodialysis (HD) patients, conferred in part by activated
The prolactin (PRL) receptor (PRLR) utilizes the JAK2/STAT-5 pathway and induces expression of cytokine-inducible SH2 (CIS)/JAK2 binding (JAB) signaling inhibitors. We and others recently showed that CIS-3 and JAB abolish PRLR-mediated JAK2 activatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::969601b19a7bd88163d84948bc21b39c
https://biblio.vub.ac.be/vubir/prolactininduced-expressionof-cytokineinducible-sh2-signaling-inhibitors-in-human-hematopoietic-progenitors(837b7d76-1750-4ad1-993f-89cbddcb5a81).html
https://biblio.vub.ac.be/vubir/prolactininduced-expressionof-cytokineinducible-sh2-signaling-inhibitors-in-human-hematopoietic-progenitors(837b7d76-1750-4ad1-993f-89cbddcb5a81).html
Publikováno v:
Pflugers Archiv : European journal of physiology. 431(4)
Primitive human hemopoietic progenitor cells identified by surface membrane markers CD33−CD34+ are capable of expansion into lineage-restricted precursors following in vitro stimulation by hemopoietic regulators such as stem cell factor (SCF) and i
Publikováno v:
Journal of cellular physiology. 157(1)
The effect of biosynthetic human insulin-like growth factor I (IGF-I) and IGF-II on the in vitro growth of human marrow myeloid progenitors in the presence of recombinant human granulocyte colony stimulating factor (rhG-CSF), granulocyte-macrophage C