Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Shoichiro, Yara"'
Autor:
Tadakuni Monma, Junichi Iwamoto, Akira Honda, Hajime Ueda, Fumio Kakizaki, Shoichiro Yara, Teruo Miyazaki, Tadashi Ikegami
Publikováno v:
Metabolites, Vol 12, Iss 4, p 331 (2022)
Since intestinal secondary bile acids (BAs) prevent Clostridium difficile infection (CDI), the serum BA profile may be a convenient biomarker for CDI susceptibility in human subjects. To verify this hypothesis, we investigated blood samples from 71 p
Externí odkaz:
https://doaj.org/article/a37c09d0aca749bb8b91976288c1bd63
Autor:
Tadakuni, Monma, Junichi, Iwamoto, Hajime, Ueda, Makoto, Tamamushi, Fumio, Kakizaki, Naoki, Konishi, Shoichiro, Yara, Teruo, Miyazaki, Takeshi, Hirayama, Tadashi, Ikegami, Akira, Honda
Publikováno v:
World Journal of Clinical Cases. 10:12484-12493
Dysbiosis in the intestinal microflora can affect the gut production of microbial metabolites, and toxic substances can disrupt the barrier function of the intestinal wall, leading to the development of various diseases. Decreased levels of Clostridi
Autor:
Shunichiro Nakagawa, Junichi Iwamoto, Yuki Moriyama, Fumio Kakizaki, Makoto Tamamushi, Hajime Ueda, Tadakuni Monma, Naoki Konishi, Shoichiro Yara, Takeshi Hirayama, Akira Honda, Tadashi Ikegami
Publikováno v:
Progress of Digestive Endoscopy. 100:45-49
Autor:
Shoichiro Yara, Akira Honda, Tadashi Ikegami, Takeshi Hirayama, Hajime Ueda, Teruo Miyazaki, Yukio Morishita, Junichi Iwamoto, Tadakuni Monma
Publikováno v:
Hepatology Communications, Vol 5, Iss 12, Pp 2052-2067 (2021)
Hepatology Communications
Hepatology Communications
Western-style high-fat/high-sucrose diet (HFHSD) changes gut microbiota and bile acid (BA) profiles. Because gut microbiota and BAs could influence each other, the mechanism of changes in both by HFHSD is complicated and remains unclear. We first aim
Autor:
Makoto Tamamushi, Tadakuni Monma, Tadashi Ikegami, Takeshi Hirayama, Masashi Murakami, Junichi Iwamoto, Hajime Ueda, Akira Honda, Naoki Konishi, Yuji Mizokami, Shoichiro Yara
Publikováno v:
Journal of Clinical Biochemistry and Nutrition. 66(2):158-162
Non-steroidal anti-inflammatory drugs (NSAIDs) or low-dose aspirin (LDA) are the most common causes of drug-induced gastroduodenal ulcer and We investigated preventive treatment with use of concomitant anti-ulcer drugs and the clinical features of ga
Autor:
Masashi Murakami, Tadashi Ikegami, Akira Honda, Makoto Nakamuta, Teruo Miyazaki, Takeshi Hirayama, Shoichiro Yara, Motoyuki Kohjima, Junichi Iwamoto
Publikováno v:
GastroHep. 1:302-310
Autor:
Tadakuni Monma, Tadashi Ikegami, Shoichiro Yara, Takeshi Hirayama, Junichi Iwamoto, Akira Honda, Hajime Ueda, Naoki Konishi, Meikei Hayashi, Masashi Murakami, Makoto Tamamushi
Publikováno v:
Progress of Digestive Endoscopy. 94:78-80
Autor:
Tadashi Ikegami, Shoichiro Yara, Takeshi Hirayama, Yasushi Matsuzaki, Naoki Konishi, Akira Honda, Junichi Iwamoto, Hajime Ueda, Tadakuni Monma, Masashi Murakami
Publikováno v:
Internal Medicine
A 66-year-old Japanese male with a history of Behcet disease exhibited oral and genital ulcers, and a round deep ileocecal ulcer. He was treated with a combination of mesalazine and 20 mg/day of prednisolone (PSL), but was only partially responsive t
Autor:
Masashi Murakami, Tadakuni Monma, Tadashi Ikegami, Shoichiro Yara, Naoki Konishi, Takeshi Hirayama, Makoto Tamamushi, Hajime Ueda, Junichi Iwamoto
Publikováno v:
Progress of Digestive Endoscopy. 96:181-182
Autor:
Junichi Iwamoto, Tadakuni Monma, Hajime Ueda, Makoto Tamamushi, Naoki Konishi, Shoichiro Yara, Masashi Murakami, Takeshi Hirayama, Akira Honda, Tadashi Ikegami, Yuji Mizokami
Background and Aim: Non-steroidal anti-inflammatory drugs (NSAIDs) or low-dose aspirin (LDA) are the most common causes of drug-induced gastroduodenal ulcer. We investigated preventive treatment with use of concomitant anti-ulcer drugs and the clinic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4af0611deaca89181ce9450bf9545cbc
https://doi.org/10.21203/rs.2.11424/v1
https://doi.org/10.21203/rs.2.11424/v1