Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Shogo Tsuyuki"'
Autor:
Anthony J. Coyle, Shogo Tsuyuki
Publikováno v:
Mediators of Inflammation, Vol 4, Iss 4, Pp 239-247 (1995)
The cytokines released from Th2 and Th2-like cells are likely to be central to the pathophysiolgy of asthma and allergy, contributing to aberrant IgE production, eosinophilia and, perhaps, mucosal susceptibility to viral infection. IL-4 has emerged a
Externí odkaz:
https://doaj.org/article/9a622b8a66054208877b7c5d2038895e
Autor:
Den-ichi Momose, Masaaki Sato, Toshiro Kobayashi, Yoshiki Tokutake, Shogo Tsuyuki, Kazuhiko Shinagawa, Yasuo Toyoshina Takehana
Publikováno v:
Biological and Pharmaceutical Bulletin. 21:350-355
The pharmacological profile of a newly synthesized histamine H, receptor antagonist, KAA-276 (1-[1-(4-fluorophenylmethyl)-1H-benzimidazol-2-yl]-5-[2-[4-(2-carboxyethyl)-phenyl]ethyl]-1,5-diazacyclooctane sulfate), was characterized. In a H, receptor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e6bc9cb4836523dc7243c5c7672a9587
https://doi.org/10.1248/bpb.21.350
https://doi.org/10.1248/bpb.21.350
Publikováno v:
The Journal of Experimental Medicine
Elevated levels of immunoglobulin (Ig) E are associated with bronchial asthma, a disease characterized by eosinophilic inflammation of the airways. Activation of antigen-specific T helper (Th) 2 cells in the lung with the subsequent release of interl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46acf8105d261a022dd09b50371790f0
https://doi.org/10.1084/jem.183.4.1303
https://doi.org/10.1084/jem.183.4.1303
Autor:
Gary P. Anderson, Claude Bertrand, Frangois Erard, Shogo Tsuyuki, Junko Tsuyuki, Anthony J. Coyle, Martin Wesp, Alexandre Trifilieff
Publikováno v:
Journal of Clinical Investigation. 96:2924-2931
While considerable progress has been made in understanding the events by which eosinophils accumulate in various pathophysiological conditions, the mechanisms controlling the resolution of eosinophilic inflammation are poorly understood. In the prese
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8bed7b8f964e22e0d13bc75d3ddfee6d
https://doi.org/10.1172/jci118364
https://doi.org/10.1172/jci118364
Autor:
A J Coyle, Shogo Tsuyuki
Publikováno v:
Mediators of Inflammation
Mediators of Inflammation, Vol 4, Iss 4, Pp 239-247 (1995)
Mediators of Inflammation, Vol 4, Iss 4, Pp 239-247 (1995)
The cytokines released from Th2 and Th2-like cells are likely to be central to the pathophysiolgy of asthma and allergy, contributing to aberrant IgE production, eosinophilia and, perhaps, mucosal susceptibility to viral infection. IL-4 has emerged a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::953ffedd30117ad39d60bc78f0e2f060
http://europepmc.org/articles/PMC2365650
http://europepmc.org/articles/PMC2365650
Publikováno v:
European journal of immunology. 28(9)
Eosinophilic inflammation of the airways is believed to play a central role in the pathogenesis of bronchial asthma. Inoculation of mice with the nematode Nippostrongylus brasiliensis induces pulmonary inflammation, characterized by a marked infiltra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9805ae255bbba3086108bac563e20848
https://pubmed.ncbi.nlm.nih.gov/9754552
https://pubmed.ncbi.nlm.nih.gov/9754552
Publikováno v:
American journal of respiratory and critical care medicine. 155(6)
In this study, we have examined the effect of endothelin (ET) receptor antagonists on lung granulocyte inflammation after antigen challenge in sensitized mice. The antagonists used were BQ-123, an ETA antagonist, BQ-788, an ETB antagonist, and SB2096
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::491098fbfe93224959e35707b05ea28a
https://pubmed.ncbi.nlm.nih.gov/9196091
https://pubmed.ncbi.nlm.nih.gov/9196091
Publikováno v:
The Journal of Experimental Medicine
The recruitment of eosinophils into the airways after allergen exposure is dependent on interleukin (IL) 5 secreted from antigen-specific CD4+ T cells of the T helper cell (Th) 2 subset. However, while it is established that costimulation through CD2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ed6955f53421b8c1fd650021450f3b01
https://pubmed.ncbi.nlm.nih.gov/9151904
https://pubmed.ncbi.nlm.nih.gov/9151904
Autor:
Claude Bertrand, Shogo Tsuyuki, Graham Le Gros, Simone Walti, Hanspeter Pircher, Francois Erard, A J Coyle
Publikováno v:
Annals of the New York Academy of Sciences. 796
Viral infections of the lung have been postulated to be a major factor in the etiology of bronchial asthma, a disease characterized by eosinophilic inflammation of the airways. In addition, upper respiratory tract infection in asthmatic individuals r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b55dc67f285e33175c29f37ec021b68a
https://pubmed.ncbi.nlm.nih.gov/8906216
https://pubmed.ncbi.nlm.nih.gov/8906216
Autor:
G. Le Gros, Ch.H. Heusser, Gary P. Anderson, Manfred Kopf, Claude Bertrand, A J Coyle, Shogo Tsuyuki
Publikováno v:
Scopus-Elsevier
Aerosol antigen challenge of ovalbumin-sensitized mice induced an eosinophilic airway inflammation that was dependent on interleukin (IL)-5 and CD4+, but not CD8+, T lymphocytes. The involvement of the Th2 phenotype of CD4+ T cells was supported by d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::00bc077dabee32820801185d7f4fda0c
https://pubmed.ncbi.nlm.nih.gov/7598937
https://pubmed.ncbi.nlm.nih.gov/7598937