Zobrazeno 1 - 10
of 37
pro vyhledávání: '"Shiva Kazempour"'
Autor:
Sogand Sajedi, Ghazal Ebrahimi, Raheleh Roudi, Isha Mehta, Amirreza Heshmat, Hanie Samimi, Shiva Kazempour, Aamir Zainulabadeen, Thomas Roderick Docking, Sukeshi Patel Arora, Francisco Cigarroa, Sudha Seshadri, Aly Karsan, Habil Zare
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-10 (2023)
Abstract Analyzing different omics data types independently is often too restrictive to allow for detection of subtle, but consistent, variations that are coherently supported based upon different assays. Integrating multi-omics data in one model can
Externí odkaz:
https://doaj.org/article/46004c6c5edd4eae922198954ba955dd
Autor:
Jamie M. Walker, Shiva Kazempour Dehkordi, Anna Fracassi, Alison Vanschoiack, Anna Pavenko, Giulio Taglialatela, Randall Woltjer, Timothy E. Richardson, Habil Zare, Miranda E. Orr
Publikováno v:
Acta Neuropathologica Communications, Vol 10, Iss 1, Pp 1-14 (2022)
Abstract Clinical symptoms correlate with underlying neurodegenerative changes in the vast majority of people. However, an intriguing group of individuals demonstrate neuropathologic changes consistent with Alzheimer disease (AD) yet remain cognitive
Externí odkaz:
https://doaj.org/article/046be168d3ca411d8f9e2f40f79a4fb0
Akademický článek
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Autor:
Jamie M, Walker, Shiva Kazempour, Dehkordi, Jeff, Schaffert, William, Goette, Charles L, White Iii, Timothy E, Richardson, Habil, Zare
Publikováno v:
Journal of Alzheimer's Disease. 91:683-695
Background: The strongest risk factor for the development of Alzheimer’s disease (AD) is age. The progression of Braak stage and Thal phase with age has been demonstrated. However, prior studies did not include cognitive status. Objective: We set o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46a16216a22cf5a8e7742d7db514c487
https://doi.org/10.3233/jad-220898
https://doi.org/10.3233/jad-220898
Autor:
Miranda Orr, Mitzi Gonzales, Valentina Garbarino, Tiffany Kautz, Juan Palavicini, Marisa Lopez-Cruzan, Shiva Kazempour Dehkordi, Julia Mathews, Habil Zare, Peng Xu, Bin Zhang, Crystal Franklin, Mohamad Habes, Suzanne Craft, Ronald Petersen, Tamara Tchkonia, James Kirkland, Arash Salardini, Sudha Seshadri, Nicolas Musi
Cellular senescence has been identified as a pathological mechanism linked to tau and amyloid beta (Aβ) accumulation in mouse models of Alzheimer's disease (AD). Clearance of senescent cells using the senolytic compounds dasatinib (D) and quercetin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ae6d960916525c27a42ab87bad78926e
https://doi.org/10.21203/rs.3.rs-2809973/v1
https://doi.org/10.21203/rs.3.rs-2809973/v1
Akademický článek
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Autor:
Shiva Kazempour Dehkordi, Jamie Walker, Eric Sah, Emma Bennett, Farzaneh Atrian, Bess Frost, Benjamin Woost, Rachel E. Bennett, Timothy C. Orr, Yingyue Zhou, Prabhakar S. Andhey, Marco Colonna, Peter H. Sudmant, Peng Xu, Minghui Wang, Bin Zhang, Habil Zare, Miranda E. Orr
Publikováno v:
Nat Aging
Senescent cells contribute to pathology and dysfunction in animal models(1). Their sparse distribution and heterogenous phenotype have presented challenges for detecting them in human tissues. We developed a senescence eigengene approach to identify
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0e28783a3b8c04b0aae5113f56d1f17b
https://doi.org/10.1038/s43587-021-00142-3
https://doi.org/10.1038/s43587-021-00142-3
Autor:
Miranda E Orr, Timothy C Orr, Shiva Kazempour Dehkordi, Habil Zare, Jamie M. Walker, C Dirk Keene
Publikováno v:
Alzheimer's & Dementia. 18
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bec19266670a3e4a227cc04635c7bd68
https://doi.org/10.1002/alz.069118
https://doi.org/10.1002/alz.069118
Autor:
Jamie M. Walker, Shiva Kazempour Dehkordi, Anna Fracassi, Alison Vanschoiack, Anna Pavenko, Giulio Taglialatela, Randall Woltjer, Timothy E. Richardson, Habil Zare, Miranda E. Orr
Publikováno v:
Acta neuropathologica communications. 10(1)
Clinical symptoms correlate with underlying neurodegenerative changes in the vast majority of people. However, an intriguing group of individuals demonstrate neuropathologic changes consistent with Alzheimer disease (AD) yet remain cognitively normal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::593e7fea7b4d028aadb402845839adfb
https://pubmed.ncbi.nlm.nih.gov/35164877
https://pubmed.ncbi.nlm.nih.gov/35164877
Akademický článek
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K zobrazení výsledku je třeba se přihlásit.
K zobrazení výsledku je třeba se přihlásit.