Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Shirin V. Sundar"'
Autor:
Brenda S. Magenheimer, Joseph S. Tash, Xiaogang Li, Darren P. Wallace, Sudhakar Jakkaraj, Shirin V. Sundar, Gail A. Reif, Gunda I. Georg, James P. Calvet, Xia Zhou, Alan S.L. Yu
Publikováno v:
American Journal of Physiology-Renal Physiology. 323:F492-F506
Autosomal dominant polycystic kidney disease (ADPKD) is a debilitating renal neoplastic disorder with limited treatment options. It is characterized by the formation of large fluid-filled cysts that develop from kidney tubules through abnormal cell p
Publikováno v:
Hospital Practice. 46:197-202
To estimate the cost difference associated with tolvaptan treatment vs. fluid restriction (FR) among hospitalized patients with heart failure (HF) and hyponatremia (HN) based on a real-world registry of HN patients.An Excel-based economic model was d
Autor:
Sandra L. Chase, Alpesh Amin, Jesse D. Ortendahl, Shirin V. Sundar, Robert Stellhorn, Siddhesh A Kamat, Amanda L Harmon
Publikováno v:
American journal of health-system pharmacy : AJHP : official journal of the American Society of Health-System Pharmacists. 76(6)
Purpose Costs associated with unplanned readmissions among patients with heart failure with and without hyponatremia were studied. Methods This study estimated the costs of patients hospitalized for heart failure (HF) discharged with or without corre
Autor:
Amanda L Harmon, Alpesh Amin, Sandra L. Chase, Siddhesh A Kamat, Jesse D. Ortendahl, Shirin V. Sundar, Robert Stellhorn
Publikováno v:
Current medical research and opinion. 34(3)
Assess characteristics of patients with heart failure (HF) and hyponatremia (HN) using tolvaptan, a selective vasopressin V2-receptor antagonist, for sodium correction, and estimate the budget impact of tolvaptan use in a hospital.The Premier hospita
Autor:
Judith Hellman, Arun Prakash, Samira Khakpour, Kevin Wilhelmsen, Jinhua Wang, Katherine Farrar, Shirin V. Sundar, Alphonso Tran, Fengyun Xu, Nathanael S. Gray
Inflammatory critical illness is a syndrome that is characterized by acute inflammation and organ injury, and it is triggered by infections and noninfectious tissue injury, both of which activate innate immune receptors and pathways. Although reports
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::09730bea0f0124384c5a859063d2ffa7
https://europepmc.org/articles/PMC5734625/
https://europepmc.org/articles/PMC5734625/
Autor:
Shirin V. Sundar, Ranjana K. Seth, Gurmel S. Sidhu, Pankaj Seth, Dinesh K. Kulshreshtha, S.C. Sharma, Radha K. Maheshwari
Publikováno v:
Shock. 19:169-175
Resuscitation from hemorrhagic shock initiates profound changes in the liver that are likely to contribute to end organ damage and resultant dysfunction after shock. Extensive research in this area has indicated the potential of free radical scavengi
Publikováno v:
Renaissance of Sickle Cell Disease Research in the Genome Era
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::911635b48b7c271168fcd502f076d062
https://doi.org/10.1142/9781860947964_0015
https://doi.org/10.1142/9781860947964_0015
Publikováno v:
Biochemical and biophysical research communications. 332(3)
Comprehensive knowledge of the gene expression changes induced by hemorrhage in vital organs will greatly improve prognosis and therapy. Therefore, we used a mouse model of non-resuscitated hemorrhagic shock to study the pattern of stress-induced gen
Autor:
Jaya P. Gaddipati, Jillian Calemine, Radha K. Maheshwari, Shirin V. Sundar, Pankaj Seth, Gurmel S. Sidhu
Publikováno v:
Shock (Augusta, Ga.). 19(2)
Inflammatory cytokines interleukin 1 (IL-1), IL-2, IL-6, and tumor necrosis factor-alpha (TNF-alpha) have been recognized as important mediators of pathophysiological and immunological events associated with shock. These inflammatory events after hem
Publikováno v:
Journal of interferoncytokine research : the official journal of the International Society for Interferon and Cytokine Research. 19(11)
We have shown previously that interferon-beta (IFN-beta) induces the alkalinization of trans-Golgi network (TGN) and inhibits the transport of G protein of vesicular stomatitis virus (VSV) in L(B) cells and gD protein of herpes simplex virus (HSV-1)