Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Shipan Dai"'
Publikováno v:
PLoS Pathogens, Vol 12, Iss 3, p e1005504 (2016)
Externí odkaz:
https://doaj.org/article/bdfd8db4efe94205a14f6da00ea3db85
Publikováno v:
PLoS Pathogens, Vol 9, Iss 1, p e1003114 (2013)
Complement receptor 3 (CR3, CD11b/CD18) is a major macrophage phagocytic receptor. The biochemical pathways through which CR3 regulates immunologic responses have not been fully characterized. Francisella tularensis is a remarkably infectious, facult
Externí odkaz:
https://doaj.org/article/5670708cfebb4193953b981b03821933
Publikováno v:
PLoS Computational Biology, Vol 8, Iss 11, p e1002757 (2012)
Complement Receptor 3 (CR3) and Toll-like Receptor 2 (TLR2) are pattern recognition receptors expressed on the surface of human macrophages. Although these receptors are essential components for recognition by the innate immune system, pathogen coord
Externí odkaz:
https://doaj.org/article/66547be67da0495b9c21c01f3ec8d6cb
Publikováno v:
Traffic. Oct2007, Vol. 8 Issue 10, p1365-1374. 10p. 4 Color Photographs, 1 Diagram, 1 Graph.
Francisella tularensis is a gram-negative coccobacillus that is found in diverse environments, including animal, protozoan, and insect hosts. Several animal models have been used for the study of F. tularensis infection, primarily rodents (including
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e965302b02407366a180b5f8eb380f3e
https://doi.org/10.1128/9781555818524.ch21
https://doi.org/10.1128/9781555818524.ch21
Publikováno v:
PLoS Pathogens, Vol 12, Iss 3, p e1005504 (2016)
PLoS Pathogens
PLoS Pathogens
Complement receptor 3 (CR3, CD11b/CD18) is a major macrophage phagocytic receptor. The biochemical pathways through which CR3 regulates immunologic responses have not been fully characterized. Francisella tularensis is a remarkably infectious, facult
Publikováno v:
Infection and Immunity. 80:1088-1097
Francisella tularensis is a remarkably infectious facultative intracellular pathogen that causes the zoonotic disease tularemia. Essential to the pathogenesis of F. tularensis is its ability to escape the destructive phagosomal environment and inhibi
Autor:
Matthew D. Neal, Bin Li, Thomas Prindle, Ward M. Richardson, Shipan Dai, Richard A. Shapiro, David J. Hackam, Selma Cetin, James H.-C. Wang, Maria F. Branca, Chhinder P. Sodhi, Congrong Ma
Publikováno v:
Journal of Biological Chemistry. 285:4995-5002
Toll-like receptor-4 (TLR4) is the receptor for bacterial lipopolysaccharide, yet it may also respond to a variety of endogenous molecules. Necrotizing enterocolitis (NEC) is the leading cause of death from gastrointestinal disease in newborn infants
Autor:
Chhinder P. Sodhi, Ward M. Richardson, Jeff W. Kohler, Jun Li, Rahul J. Anand, Shipan Dai, David J. Hackam, Rosemary A. Hoffman, Steven C. Gribar, Xiao Hua Shi, Maria F. Branca
Publikováno v:
The Journal of Immunology. 181:8534-8543
The pathways that lead to the internalization of pathogens via phagocytosis remain incompletely understood. We now demonstrate a previously unrecognized role for the gap junction protein connexin43 (Cx43) in the regulation of phagocytosis by macropha
Autor:
Christopher Rippel, Faisal G. Qureshi, Jun Li, Jeffrey W. Kohler, Rahul J. Anand, Steven C. Gribar, Chhinder P. Sodhi, Donna B. Stolz, Cynthia L. Leaphart, David J. Hackam, Shipan Dai
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 294:G109-G119
Enterocytes exist in close association with tissue macrophages, whose activation during inflammatory processes leads to the release of nitric oxide (NO). Repair from mucosal injury requires the migration of enterocytes into the mucosal defect, a proc