Zobrazeno 1 - 10
of 64
pro vyhledávání: '"Shinobu Matsui"'
Publikováno v:
International Journal of Angiology. 4:188-192
The purpose of this study was to evaluate the changes of plasma adenosine concentration by dynamic exercise in patients with chronic heart failure. We studied six patients with chronic heart failure (heart failure group) and six normal subjects (norm
Autor:
Jong-Dae Lee, Takayoshi Asaji, Kunihisa Miwa, Hisae Hayasaki, Izuru Nakae, Hiroyasu Uzui, Hideki Hayashi, Shinobu Matsui, Tatsuya Sawamura, Hiroaki Uenishi, Hiroshi Tada, Masatoshi Fujita, Tetsuya Matsumoto
Publikováno v:
International Journal of Cardiology. 176:1230-1232
Autor:
Yasuhito Ishigaki, Tsukasa Matsunaga, Shinobu Matsui, Tsutomu Takegami, Akihiro Nagao, Xia Zhao, Hideaki Nakagawa
Publikováno v:
Biochemical and Biophysical Research Communications. 370:301-305
To improve the efficiency of stable knockdown with short hairpin RNA (shRNA), we inserted multiple shRNA expression sequences into a single plasmid vector. In this study, the DNA repair factor XPA was selected as a target gene since it is not essenti
Autor:
Mituru Hayase, Michael Fu, Nobuo Yamaguchi, Noboru Takekoshi, Shinobu Matsui, Shogo Katsuda, Kohei Teraoka, Hidekazu Murano, Takayuki Kurihara
Publikováno v:
Autoimmunity. 39:121-128
Growing evidence suggests that autoimmune mechanism plays an important role in the pathogenesis of cardiomyopathy. The purpose of this study was to investigate whether passive transfer of IgG and/or lymphocytes from rabbits with autoimmune cardiomyop
Autor:
Shinobu Matsui, Jie Lin, Shinji Okubo, Noboru Takekoshi, Sumiyo Akazawa-Kudoh, Tsugiyasu Kanda, Koji Kajinami, Kenji Takeda, Seiyu Kanemitsu, Hiroichi Tsugawa
Publikováno v:
Journal of Molecular and Cellular Cardiology. 36:821-830
Transient glucose deprivation (TGD) has been shown to induce a resistance to a subsequent ischemia and reperfusion injury in the heart. Induction of cyclooxygenase-2 (COX-2) and heme oxygenase-1 (HO-1) is known to mediate the powerful defensive adapt
Autor:
Shinobu Matsui, Michael Fu, Mituru Hayase, Kohei Teraoka, Shogo Katsuda, Takayuki Kurihara, Noboru Takekoshi, Nobuo Yamaguchi
Publikováno v:
Journal of Cardiovascular Pharmacology. 42:S99-S103
Growing evidence suggests that the autoimmune mechanism plays an important role in the pathogenesis of dilated cardiomyopathy. The purpose of this study was to evaluate the effect on the cardiac structure and function by the transfer of immunoglobuli
Autor:
Kouji Kajinami, Ryoko Sato, Noboru Takekoshi, Sugako Kanayama, Shinobu Matsui, Seiyu Kanemitsu, Shinji Okubo, Naohiro Yamashita
Publikováno v:
The American Journal of Cardiology. 92:1113-1116
To find the clinical variables associated with atorvastatin's effects on bone metabolism markers, 35 patients with heterozygous familial hypercholesterolemia were treated with atorvastatin for 24 weeks, and the levels of bone formation markers (bone-
Publikováno v:
Cell Structure and Function. 28:97-104
Exponentially growing Meth-A cells expressing H-2K(d).D (d) antigen were found to induce alopecia when injected intraperitoneally into normal C57BL/6 mice, which express the H-2K(b).D (b) antigen. However, the capacity to induce alopecia disappeared
Autor:
Mitsuru Hayase, Hua-Mei Fu, Michael Lx Fu, Shogo Katsuda, Marie Persson, Shinobu Matsui, Kohei Teraoka, Takayuki Kurihara
Publikováno v:
Herz. 25:267-270
Idiopathic dilated cardiomyopathy is a severe disease of unknown etiology. Accumulating evidence suggests that agonist-like autoantibodies against the beta 1 adrenoceptor in the circulation of dilated cardiomyopathy may play an important role. The ai
Autor:
Takayuki Kurihara, T Katsuda, H Wakabayashi, Shinobu Matsui, Noboru Takekoshi, Nobuo Yamaguchi, Kohei Teraoka, Michael Fu, Mituru Hayase
Publikováno v:
Journal of Cardiovascular Pharmacology. 36:S43-S48
We have shown that a peptide corresponding to the sequence of the second extracellular loop of the human beta1-adrenoceptor (beta1-peptide) was able to induce an autoimmune cardiomyopathy in rabbits. In this study, we examined the effect of angiotens