Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Sheng-Qing Gao"'
Autor:
Shu-Hao Miao, Sheng-Qing Gao, Hui-Xin Li, Yun-Song Zhuang, Xue Wang, Tao Li, Chao-Chao Gao, Yan-Ling Han, Jia-Yin Qiu, Meng-Liang Zhou
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 16 (2023)
IntroductionEndothelial nitric oxide synthase (eNOS) uncoupling plays a significant role in acute vasoconstriction during early brain injury (EBI) after subarachnoid hemorrhage (SAH). Astrocytes in the neurovascular unit extend their foot processes a
Externí odkaz:
https://doaj.org/article/c1af99fff7bb472d8217e4ee85f3dd40
Autor:
Xue Wang, Hong-Ji Deng, Sheng-Qing Gao, Tao Li, Chao-Chao Gao, Yan-Ling Han, Yun-Song Zhuang, Jia-Yin Qiu, Shu-Hao Miao, Meng-Liang Zhou
Publikováno v:
Frontiers in Pharmacology, Vol 13 (2023)
Background: Erythrocytes and their breakdown products in the subarachnoid space (SAS) are the main contributors to the pathogenesis of subarachnoid hemorrhage (SAH). Dobutamine is a potent β1-adrenoreceptor agonist that can increase cardiac output,
Externí odkaz:
https://doaj.org/article/72607ad6425242279f46b0281d9c52d7
Autor:
Hong-Ji Deng, QuZhen Deji, WangDui Zhaba, Jia-Qiang Liu, Sheng-Qing Gao, Yan-Ling Han, Meng-Liang Zhou, Chun-Xi Wang
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
Nuclear factor (NF)-κB–ty -50mediated neuroinflammation plays a crucial role in early brain injury (EBI) after subarachnoid hemorrhage (SAH). As an important negative feedback regulator of NF-κB, A20 is essential for inflammatory homeostasis. Her
Externí odkaz:
https://doaj.org/article/c65f1d57e6794de5b50d22bc17eebe5e
Autor:
Qu-Zhen Deji, Xue Wang, Wang-Dui Zhaba, Hong-Ji Deng, Yan-Ling Han, Sheng-Qing Gao, Xi-Lin Liu, Meng-Liang Zhou
Publikováno v:
Neuroreport. 33(16)
Subarachnoid hemorrhage (SAH) is associated with sustained vasoconstriction in retinal vessels and vasoconstriction leads to retinal ischemia and hypoxia. Our previous finding also revealed the changes in hypoxia-related elements in the retina after
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e65d77c6ac0dcb76dbffec4f5a57508e
https://pubmed.ncbi.nlm.nih.gov/36165027
https://pubmed.ncbi.nlm.nih.gov/36165027
Autor:
Yun-Song Zhuang, Xue Wang, Sheng-Qing Gao, Shu-Hao Miao, Tao Li, Chao-Chao Gao, Yan-Ling Han, Jia-Yin Qiu, Meng-Liang Zhou, Han-Dong Wang
Publikováno v:
Brain Research. 1808:148324
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5a99d22dc4bcbd360f293394571d6b36
https://doi.org/10.1016/j.brainres.2023.148324
https://doi.org/10.1016/j.brainres.2023.148324
Autor:
Meng-Liang Zhou, Chun-Xi Wang, Wang-Dui Zhaba, Yan-Ling Han, Hong-Ji Deng, Sheng-Qing Gao, Qu-Zhen Deji, Jia-Qiang Liu
Publikováno v:
Frontiers in Immunology
Frontiers in Immunology, Vol 12 (2021)
Frontiers in Immunology, Vol 12 (2021)
Nuclear factor (NF)-κB–ty -50mediated neuroinflammation plays a crucial role in early brain injury (EBI) after subarachnoid hemorrhage (SAH). As an important negative feedback regulator of NF-κB, A20 is essential for inflammatory homeostasis. Her
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fc66551a155a24c5c848f32c197d279d
https://doi.org/10.3389/fimmu.2021.623256
https://doi.org/10.3389/fimmu.2021.623256
Autor:
Meng-Liang Zhou, Xi-Lin Liu, Tao Li, Hong-Ji Deng, Qu-Zhen Deji, Sheng-Qing Gao, Yan-Ling Han, Wang-Dui Zhaba, Chao-Chao Gao
Publikováno v:
Neuroreport. 32(6)
Traumatic brain injury (TBI) is recognized as the most influential risk factor for neurodegenerative diseases later in life, including Alzheimer's disease. The aberrant genesis of amyloid-β peptides, which is triggered by TBI, is associated with the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c86a91556259f28c587c74b1fa9fc2bf
https://pubmed.ncbi.nlm.nih.gov/33788818
https://pubmed.ncbi.nlm.nih.gov/33788818
Autor:
Sheng-Qing Gao, Lan-rong Zheng, Meng-Liang Zhou, Bin Yuan, Jia-Qiang Liu, Xi-Lin Liu, Yan-ling Han, Xiao-ming Zhou, Min Qi
Secondary brain injury following subarachnoid hemorrhage (SAH) is the critical contributor to the mortality of SAH patients. The underlying mechanisms are poorly understood. In this study, we utilized a mice model of SAH to investigate whether FoxO4
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7acda53c95d8f1e1b81a3c8776098527
Autor:
Meng-Liang Zhou, Qu-Zhen Deji, Xi-Lin Liu, Yan-Ling Han, Hong-Ji Deng, Wang-Dui Zhaba, Sheng-Qing Gao
Publikováno v:
Neuroscience Letters. 742:135554
Background and Purpose The patients who survive subarachnoid hemorrhage (SAH) often have long-term neurological complications. There are no reports about the pathological change of retina after SAH. Methods An experimental model of SAH was establishe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c02cfd647aae6562ac5c3a964de4969d
https://doi.org/10.1016/j.neulet.2020.135554
https://doi.org/10.1016/j.neulet.2020.135554
Autor:
Xi-Lin Liu, Meng-Liang Zhou, Sheng-Qing Gao, Qu-Zhen Deji, Wang-Dui Zhaba, Jia-Qiang Liu, Yan-Ling Han, Hong-Ji Deng
Publikováno v:
Life Sciences. 257:118050
Background and purpose Early brain injury is an essential pathological process after subarachnoid hemorrhage (SAH), with many cell death modalities. Ferroptosis is a newly discovered regulated cell death caused by the iron-dependent accumulation of l
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6153f9d5bec9065b950c186cde8e24c1
https://doi.org/10.1016/j.lfs.2020.118050
https://doi.org/10.1016/j.lfs.2020.118050