Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Shelly Kakar"'
Publikováno v:
Digestive Diseases and Sciences. 64:1356-1363
Cirrhosis secondary to nonalcoholic steatohepatitis (NASH) is projected to become the leading indication for liver transplantation (LT) in the USA in the next decade. The long-term implications of post-LT NASH, specifically on the development of allo
Autor:
Adeola O. Adebayo Michael, Sucha Singh, Ravi Vats, Kari Nejak-Bowen, Jacquelyn O. Russell, Minakshi Poddar, Simon C. Watkins, Pamela K. Cornuet, Shelly Kakar, Laura Molina, Prithu Sundd, Tirthadipa Pradhan-Sundd, Satdarshan P.S. Monga
Publikováno v:
Gastroenterology. 155(4)
Background & Aims Liver fibrosis, hepatocellular necrosis, inflammation, and proliferation of liver progenitor cells are features of chronic liver injury. Mouse models have been used to study the end-stage pathophysiology of chronic liver injury. How
Publikováno v:
Gastroenterology. 154(1)
Publikováno v:
Gastroenterology. 153(6)
Autor:
Douglas Borchman, Paul J. Rychwalski, Shelly Kakar, Gary N. Foulks, Marta C. Yappert, Eric Schwietz, Nathan Podoll
Publikováno v:
Ophthalmic Research. 44:34-42
Both lipids and mucins contribute to the stability of the tear film and lipids may inhibit tears from evaporating. Younger people have lower lipid viscosity, higher lipid volume, and a lower rate of tear evaporation. Since age-related changes in huma
Autor:
Bing Li, Chelsea King, Stephanie Wagner, Chuanlin Ding, Daniel W. Cramer, Richard Hansen, Jun Yan, Shelly Kakar
Publikováno v:
Clinical Immunology. 124:170-181
The therapeutic benefits of fungal β-glucans have been demonstrated as immuno-stimulating agents. In this study, we aimed to explore the mechanisms used by yeast β-glucan-rich particles to activate murine resident macrophages for cytokine secretion
Publikováno v:
American Journal of Gastroenterology. 111:S530-S531
Publikováno v:
American Journal of Gastroenterology. 111:S1014-S1015
Publikováno v:
Gastroenterology. 152:S1129
Publikováno v:
Advances in experimental medicine and biology. 614
Endogenously generated reactive oxygen species and genotoxic carcinogens can covalently modify bases in cellular DNA. If not recognized and removed prior to S-phase of the cell cycle, such modifications can block DNA replication fork progression. If