Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Shaye Stamatis"'
Autor:
James L. Zehnder, Jason Chia-Hsien Cheng, Leonssia Vlaycheva-Beisheim, Carol D. Jones, Jaya Rajamani, Shaye Stamatis, Mike Reichelt, Stefan L. Oliver, Marvin Sommer, Ann M. Arvin
Publikováno v:
Journal of Virology. 85:4095-4110
Varicella-zoster virus (VZV) is the alphaherpesvirus that causes chicken pox (varicella) and shingles (zoster). The two VZV glycoproteins gE and gI form a heterodimer that mediates efficient cell-to-cell spread. Deletion of gI yields a small-plaque-p
Autor:
Ann M. Arvin, George Kemble, Nancy Bouvier, Shaye Stamatis, Alenka M. Zeman, David B. Lewis, Tyson H. Holmes, Cornelia L. Dekker, Wenwei Tu, Harry B. Greenberg, Xiao-Song He
Publikováno v:
The Pediatric infectious disease journal. 26(2)
There have been no prior reports of the frequency of circulating influenza-specific, interferon gamma-producing memory CD4+ and CD8+ T-cells in healthy children who have received multiple influenza immunizations.We evaluated 21 previously immunized c
Publikováno v:
Journal of virology. 80(6)
The varicella-zoster virus (VZV) ORF62/63 intergenic region was cloned between the Renilla and firefly luciferase genes, which acted as reporters of ORF62 and ORF63 transcription, and recombinant viruses were generated that carried these reporter cas
Autor:
Garry P. Nolan, Chia-Chi Ku, Jean-François Fortin, Shaye Stamatis, Marvin Sommer, Leigh Zerboni, Ann M. Arvin, Anne Schaap
The pathogenesis of varicella-zoster virus (VZV) involves a cell-associated viremia during which infectious virus is carried from sites of respiratory mucosal inoculation to the skin. We now demonstrate that VZV infection of T cells is associated wit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ad6cc3abf3626d8600a037baeefd243
https://europepmc.org/articles/PMC1235817/
https://europepmc.org/articles/PMC1235817/
Autor:
Ann M. Arvin, Jason J. Cheng, Jennifer F. Moffat, Leigh Zerboni, Chengjun Mo, Marvin Sommer, Shaye Stamatis
Publikováno v:
Journal of virology. 78(22)
Varicella-zoster virus (VZV) glycoprotein E (gE) is essential for VZV replication. To further analyze the functions of gE in VZV replication, a full deletion and point mutations were made in the 62-amino-acid (aa) C-terminal domain. Targeted mutation