Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Shauna C. Kerr"'
Autor:
Abigail L. Campbell, Nicola C. Smith, James H. Reilly, Shauna C. Kerr, William J. Leach, Umberto G. Fazzi, Brian P. Rooney, George A. C. Murrell, Neal L. Millar
Publikováno v:
Mediators of Inflammation, Vol 2014 (2014)
The pathogenetic mechanisms underlying tendinopathy remain unclear, with much debate as to whether inflammation or degradation has the prominent role. Increasing evidence points toward an early inflammatory infiltrate and associated inflammatory cyto
Externí odkaz:
https://doaj.org/article/16958f57cdd344ad99491c0a60d4c790
Autor:
Sofia Fragkioudaki, Shauna C Kerr, James H. Reilly, George E Fragoulis, Iain B. McInnes, Haralampos M. Moutsopoulos
Publikováno v:
Journal of Autoimmunity. 73:85-91
Sicca symptoms occur in around 30% of rheumatoid arthritis (RA) patients. Herein, we examined the characteristics of RA patients bearing sicca symptomatology (RA-sicca) with a special focus on the immunohistopathological features of their labial mino
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a6008b5a0fc5395093d0d98f8d86e378
https://doi.org/10.1016/j.jaut.2016.06.008
https://doi.org/10.1016/j.jaut.2016.06.008
Autor:
Michael P. Kelly, J. Luke Campton, Shauna C Kerr, James H. Reilly, Cameron J. Elias-Jones, Luke Farrow, Neal L. Millar, R. M. Dominic Meek
Publikováno v:
The American Journal of Sports Medicine. 43:1875-1881
Background: Femoroacetabular impingement (FAI) is a significant cause of osteoarthritis (OA) in young active patients, but the pathophysiology remains unclear. Increasingly, mechanistic studies point toward an inflammatory component in OA. Purpose: T
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13448596c8e5da6826c81e43195642c6
https://doi.org/10.1177/0363546515588176
https://doi.org/10.1177/0363546515588176
Autor:
Umberto G. Fazzi, Susan M. Kitson, Lindsay A. N. Crowe, Moeed Akbar, George A.C. Murrell, Shauna C Kerr, Emma Garcia-Melchor, Jonathon Z B Cher, Michael McLean, Neal L. Millar, Stephen C Hannah
Publikováno v:
The American journal of sports medicine. 46(3)
Background: The pathophysiological mechanisms behind proliferation of fibroblasts and deposition of dense collagen matrix in idiopathic frozen shoulder remain unclear. Alarmins (also known as danger signals) are endogenous molecules that are released
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::08cfda8bc0b7398c42b480535886ddfa
https://pubmed.ncbi.nlm.nih.gov/29190116
https://pubmed.ncbi.nlm.nih.gov/29190116
Autor:
Shauna C Kerr, D McCarey, James H. Reilly, Iain B. McInnes, Mariola Kurowska-Stolarska, Aziza Elmesmari, Gianfranco Ferraccioli, Stefano Alivernini
Publikováno v:
Rheumatology. 56
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7255f5d27590c1acdf39935fb31ce1cd
https://doi.org/10.1093/rheumatology/kex061.036
https://doi.org/10.1093/rheumatology/kex061.036
Autor:
Manhal Khuder Makdasy, Sabina Patel, Shauna C Kerr, J. Alastair Gracie, L. Ballantine, James H. Reilly, Iain B. McInnes, Pamela B Wright, Mariola Kurowska-Stolarska, Darren L. Asquith, Jagtar Singh Nijjar
Publikováno v:
Annals of the Rheumatic Diseases. 72:2024-2031
ObjectivesMacrophages are central to the inflammatory processes driving rheumatoid arthritis (RA) synovitis. The molecular pathways that are induced in synovial macrophages and thereby promote RA disease pathology remain poorly understood.MethodsWe u
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ebe768f4d7936b22165d5dd5cfae3e8e
https://doi.org/10.1136/annrheumdis-2012-202872
https://doi.org/10.1136/annrheumdis-2012-202872
Autor:
Brian P. Rooney, Abigail L. Campbell, Kevin J. Little, Shauna C Kerr, James H. Reilly, Iain B. McInnes, Neal L. Millar, George A.C. Murrell, W. J. Leach
Publikováno v:
Annals of the Rheumatic Diseases. 71:302-310
To seek evidence for the role of hypoxia in early human tendinopathy, and thereafter to explore mechanisms whereby tissue hypoxia may regulate apoptosis, inflammatory mediator expression and matrix regulation in human tenocytes.Fifteen torn supraspin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ab22ffc1cc0974a1a87c501878fa9424
https://doi.org/10.1136/ard.2011.154229
https://doi.org/10.1136/ard.2011.154229
Autor:
Ashley M. Miller, Shauna C Kerr, Darren L. Asquith, James H. Reilly, Paul Welsh, Iain B. McInnes, Naveed Sattar
Publikováno v:
Annals of the Rheumatic Diseases. 70:2225-2228
Background It has previously been shown that dual activation of the Liver X Receptors (LXRα and LXRβ) by the agonist, GW3965, enhances pathology in a murine model of collagen-induced arthritis. Objective To determine whether LXRα or LXRβ have dis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::41cd9b5579f80ec7501c37856040f01a
https://doi.org/10.1136/ard.2011.152652
https://doi.org/10.1136/ard.2011.152652
Autor:
Christina Böhm, Neal L. Millar, Shauna C Kerr, Regina Gary, James H. Reilly, Foo Y. Liew, Iain B. McInnes, Padraic G. Fallon, Mario M. Zaiss, Bartosz Stolarski, Mariola Kurowska-Stolarska, Carina Scholtysek, Jean-Pierre David, Thomas Kamradt, Georg Schett
Publikováno v:
The Journal of Immunology
The Journal of Immunology; Vol 186
The Journal of Immunology; Vol 186
IL-33 is a new member of the IL-1 family, which plays a crucial role in inflammatory response, enhancing the differentiation of dendritic cells and alternatively activated macrophages (AAM). Based on the evidence of IL-33 expression in bone, we hypot
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::15faf68e03777a785d9ffe4efd6fec97
https://doi.org/10.4049/jimmunol.1003487
https://doi.org/10.4049/jimmunol.1003487
Autor:
Alirio J. Melendez, Jan Leipe, Darren L. Asquith, Axel J. Hueber, Ashley M. Miller, Shauna C Kerr, James H. Reilly, Iain B. McInnes
Publikováno v:
The Journal of Immunology. 184:3336-3340
The proinflammatory cytokine IL-17A is considered a crucial player in rheumatoid arthritis (RA) pathogenesis. In experimental models of autoimmune arthritis, it has been suggested that the cellular source of IL-17A is CD4+ T cells (Th17 cells). Howev
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1fd7a2dc0de1f34c2a914ab58307b335
https://doi.org/10.4049/jimmunol.0903566
https://doi.org/10.4049/jimmunol.0903566