Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Shao-Tung Wu"'
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 173, Iss , Pp 116298- (2024)
Idiopathic pulmonary fibrosis (IPF) is a chronic and progressive interstitial lung disease; its cause is unknown, and it leads to notable health problems. Currently, only two drugs are recommended for IPF treatment. Although these drugs can mitigate
Externí odkaz:
https://doaj.org/article/8ced063e049d4930ae901c0045041520
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 142, Iss , Pp 112028- (2021)
Acute kidney disease due to renal ischemia/reperfusion (I/R) is a major clinical problem without effective therapies. The injured tubular epithelial cells may undergo epithelial–mesenchymal transition (EMT). It will loss epithelial phenotypes and e
Externí odkaz:
https://doaj.org/article/edecf404489847a88e3cf01865c893c1
Soluble Dipeptidyl Peptidase-4 Induces Fibroblast Activation Through Proteinase-Activated Receptor-2
Autor:
Shih-Yi Lee, Shao-Tung Wu, Yao-Jen Liang, Ming-Jai Su, Cheng-Wei Huang, Yu-Hsuan Jao, Hui-Chun Ku
Publikováno v:
Frontiers in Pharmacology, Vol 11 (2020)
Fibroblasts are the chief secretory cells of the extracellular matrix (ECM) responsible for basal deposition and degradation of the ECM under normal conditions. During stress, fibroblasts undergo continuous activation, which is defined as the differe
Externí odkaz:
https://doaj.org/article/75ac1239b09246699fe1d4d736660c68
Background: Oxidative stress is an imbalance between the production of reactive oxygen species (ROS) and the detoxification ability of reactive intermediates. It will lead to mitochondrial damage and dysfunction, resulting in the dysfunction of bioen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5f930e10e463c9c5d989cb5c8b786fad
https://doi.org/10.21203/rs.3.rs-30722/v1
https://doi.org/10.21203/rs.3.rs-30722/v1
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 142, Iss, Pp 112028-(2021)
Acute kidney disease due to renal ischemia/reperfusion (I/R) is a major clinical problem without effective therapies. The injured tubular epithelial cells may undergo epithelial–mesenchymal transition (EMT). It will loss epithelial phenotypes and e