Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Shana L. Skradski"'
Autor:
David C. Henshall, Shana L. Skradski, Robert Meller, Tomohiro Araki, Manabu Minami, Clara K. Schindler, Jing-Quan Lan, David P. Bonislawski, Roger P. Simon
Publikováno v:
Neurobiology of Disease, Vol 10, Iss 2, Pp 71-87 (2002)
The caspase family of cell death proteases has been implicated in the mechanism of neuronal death following seizures. We investigated the expression and processing of caspases 6 and 7, putative executioner caspases. Brief limbic seizures were evoked
Externí odkaz:
https://doaj.org/article/1c6ba07d93bf4282acc9572331c39fc0
Autor:
David C. Henshall, David P. Bonislawski, Shana L. Skradski, Jing-Quan Lan, Robert Meller, Roger P. Simon
Publikováno v:
Neurobiology of Disease, Vol 8, Iss 4, Pp 568-580 (2001)
The mechanism by which seizures induce neuronal death is not completely understood. Caspase-8 is a key initiator of apoptosis via extrinsic, death receptor-mediated pathways; we therefore investigated its role in mediating seizure-induced neuronal de
Externí odkaz:
https://doaj.org/article/49e7bdb751c14911aa25ac48b8f3bb99
Publikováno v:
Neurobiology of Disease, Vol 3, Iss 4, Pp 263-269 (1997)
We examined the expression of the sleep-inducing cytokine interleukin-1α (IL-1α) in the brains of audiogenic seizure-susceptible mice subsequent to the induction of sound-induced seizure. Animal models of epilepsy often require lesioning or trauma
Externí odkaz:
https://doaj.org/article/851662941bf149fba70b1e0524445d52
Autor:
Jing-Quan Lan, Robert Meller, Clara K. Schindler, Roger P. Simon, David C. Henshall, Sachiko Shinoda, Tomohiro Araki, Shana L. Skradski, Waro Taki
Publikováno v:
European Journal of Neuroscience. 17:2065-2076
The consequences of activation of tumour necrosis factor receptor 1 (TNFR1) during neuronal injury remain controversial. The apoptosis signal-regulating kinase 1 (ASK1), a mitogen-activated protein kinase kinase kinase, can mediate cell death downstr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4e24591f619b42485902ac0e585a6ec8
https://doi.org/10.1046/j.1460-9568.2003.02655.x
https://doi.org/10.1046/j.1460-9568.2003.02655.x
Publikováno v:
Neuroscience Letters. 324:33-36
Activation of cysteinyl aspartate-specific proteases (caspases) may underlie apoptotic cell death in brain. Terminal, executioner caspases 3, 6 and 7 likely contribute to such cell death in a stimulus- and cell type-specific manner. Here we investiga
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::17ef070f9169598855e2da1894cdc2ba
https://doi.org/10.1016/s0304-3940(02)00166-0
https://doi.org/10.1016/s0304-3940(02)00166-0
Publikováno v:
Journal of Neurochemistry. 77:886-895
Seizure-induced neuronal death may be under the control of the caspase family of cell death proteases. We examined the role of caspase-2 in a model of focally evoked limbic seizures with continuous EEG recording. Seizures were elicited by microinject
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::acd0deb6a0a7bf53ce6de0e8686b6fcc
https://doi.org/10.1046/j.1471-4159.2001.00291.x
https://doi.org/10.1046/j.1471-4159.2001.00291.x
Autor:
Roger P. Simon, David P. Bonislawski, Robert Meller, Jing-Quan Lan, David C. Henshall, Shana L. Skradski
Publikováno v:
Neurobiology of Disease, Vol 8, Iss 4, Pp 568-580 (2001)
The mechanism by which seizures induce neuronal death is not completely understood. Caspase-8 is a key initiator of apoptosis via extrinsic, death receptor-mediated pathways; we therefore investigated its role in mediating seizure-induced neuronal de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e9a32ec98f1eee4c5c7e6128b373c482
http://www.sciencedirect.com/science/article/pii/S0969996101904152
http://www.sciencedirect.com/science/article/pii/S0969996101904152
Autor:
Sachiko, Shinoda, Shana L, Skradski, Tomohiro, Araki, Clara K, Schindler, Robert, Meller, Jing-Quan, Lan, Waro, Taki, Roger P, Simon, David C, Henshall
Publikováno v:
The European journal of neuroscience. 17(10)
The consequences of activation of tumour necrosis factor receptor 1 (TNFR1) during neuronal injury remain controversial. The apoptosis signal-regulating kinase 1 (ASK1), a mitogen-activated protein kinase kinase kinase, can mediate cell death downstr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::663001124eae9c5d82a2cfb43f484d0a
https://pubmed.ncbi.nlm.nih.gov/12786973
https://pubmed.ncbi.nlm.nih.gov/12786973
Autor:
Robert Meller, Clara K. Schindler, Tomohiro Araki, Shana L. Skradski, Manabu Minami, Roger P. Simon, David C. Henshall, Jing Quan Lan, David P. Bonislawski
Publikováno v:
Neurobiology of Disease, Vol 10, Iss 2, Pp 71-87 (2002)
The caspase family of cell death proteases has been implicated in the mechanism of neuronal death following seizures. We investigated the expression and processing of caspases 6 and 7, putative executioner caspases. Brief limbic seizures were evoked
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fb7bdb93aa3cf57a8b5a8b6c8b98e078
https://pubmed.ncbi.nlm.nih.gov/12127146
https://pubmed.ncbi.nlm.nih.gov/12127146
Publikováno v:
Neuroscience letters. 305(3)
Control of seizure-induced neuronal death may involve members of the Bcl-2 family of cell death regulating proteins. Bcl-w is a newly described anti-apoptotic member of this family that may confer neuroprotective effects. We therefore investigated Bc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c3a7ec129301995d375cb6a7e55fb6c8
https://pubmed.ncbi.nlm.nih.gov/11403928
https://pubmed.ncbi.nlm.nih.gov/11403928