Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Shaleen K. Beck"'
Autor:
Robert Calaluce, David J. Bearss, Jean Barrera, Yu Zhao, Haiyong Han, Shaleen K. Beck, Kathy McDaniel, Ray B. Nagle
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 6, Iss 5, Pp 468-479 (2004)
Interactions between extracellular matrix proteins and prostate carcinoma cells change dramatically during prostate tumor progression. We have concentrated on two key modifications that occur in the hemidesmosome in prostate carcinoma: loss of lamini
Externí odkaz:
https://doaj.org/article/e74844cb44db479a9a0e8dac632e1a68
Autor:
Adam Hoying, Raymond B. Nagle, Kevin A. Greer, David W. Mount, Shaleen K. Beck, Ritu Pandey, Robert Calaluce, James B. Hoying, Elisabeth L. Bair
Publikováno v:
The Prostate. 66:1381-1390
In prostate cancer progression, the basal lamina switches from predominantly laminin-5 to laminin-10. DU-145 prostate cancer cells were treated with either soluble laminin-5 (20 ng/ml) or laminin-10 (1 µg/ml) for 6, 24, and 48 hr. Total RNA was harv
Autor:
Janelle Kennedy, James T. Ryaby, Shaleen K. Beck, Helen Y.S. Chen, Hali Wang, James B. Hoying, Keri B. Vartanian
Publikováno v:
Journal of Cellular Physiology. 206:175-180
Thrombin is a serine protease that promotes platelet aggregation, blood coagulation, and tissue repair. A peptide derived from a non-proteolytically active region of thrombin, TP508, also promotes tissue repair and increased vascularity, yet does not
Autor:
Keri B, Vartanian, Helen Y S, Chen, Janelle, Kennedy, Shaleen K, Beck, James T, Ryaby, Hali, Wang, James B, Hoying
Publikováno v:
Journal of cellular physiology. 206(1)
Thrombin is a serine protease that promotes platelet aggregation, blood coagulation, and tissue repair. A peptide derived from a non-proteolytically active region of thrombin, TP508, also promotes tissue repair and increased vascularity, yet does not
Autor:
Haiyong Han, Kathy McDaniel, Shaleen K. Beck, Yu Zhao, Robert Calaluce, Jean Barrera, David J. Bearss, Ray B. Nagle
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 6, Iss 5, Pp 468-479 (2004)
Interactions between extracellular matrix proteins and prostate carcinoma cells change dramatically during prostate tumor progression. We have concentrated on two key modifications that occur in the hemidesmosome in prostate carcinoma: loss of lamini