Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Shakir D. Al-Sharary"'
Autor:
Othman A. Al-Shabanah, Mohammed M. Attia, Saleh A. Bakheet, Mohammed M. Al-Harbi, Sabry M. Attia, Ali Alhoshani, Ahmed M. Alhuraishi, Faisal Imam, Sheikh F. Ahmad, Khaled A. Al-Hosaini, Naif O. Al-Harbi, Shakir D. Al-Sharary
Publikováno v:
Journal of Biochemical and Molecular Toxicology. 30:559-566
In order to study the mechanisms underlying the alleviation of aflatoxin B1-induced genomic damage by proanthocyanidins (PAs), we examined the modulation of oxidative DNA damage induced by aflatoxin B1 in PAs-pretreated animals. The effects of PAs on
Autor:
Khaled A. Al-Hosaini, Mushtaq A. Ansari, Shakir D. Al-Sharary, Nahid Siddiqui, M. M. Al-Harbi, Khairy M.A. Zoheir, Sheikh F. Ahmad, Ahmed M. El-Sherbeeny, Ahmed Nadeem, Sabry M. Attia, Naif O. Al-Harbi
Publikováno v:
Pharmacological Research. 99:248-257
Psoriasis is one of the most common skin disorders characterized by erythematous plaques that result from hyperproliferative keratinocytes and infiltration of inflammatory leukocytes into dermis and epidermis. Recent studies suggest that IL-23/IL-17A
Autor:
Shakir D. Al-Sharary, Mohamed M. Sayed-Ahmed, Naif O. Al-Harbi, Khaled A. Al-Hosaini, Meshan Lafi Aldelemy, Mohamed M. Hafez, M. M. Al-Harbi, Othman A. Al-Shabanah
Publikováno v:
Cardiovascular Toxicology. 14:232-242
This study investigated whether cyclophosphamide (CP) and ifosfamide (IFO) therapy alters the expression of the key genes engaged in long-chain fatty acid (LCFA) oxidation outside rat heart mitochondria, and if so, whether these alterations should be
Alleviation of Aflatoxin B1-Induced Genomic Damage by Proanthocyanidins via Modulation of DNA Repair
Autor:
Saleh A, Bakheet, Ahmed M, Alhuraishi, Naif O, Al-Harbi, Khaled A, Al-Hosaini, Shakir D, Al-Sharary, Mohammed M, Attia, Ali R, Alhoshani, Othman A, Al-Shabanah, Mohammed M, Al-Harbi, Faisal, Imam, Sheikh F, Ahmad, Sabry M, Attia
Publikováno v:
Journal of biochemical and molecular toxicology. 30(11)
In order to study the mechanisms underlying the alleviation of aflatoxin B1-induced genomic damage by proanthocyanidins (PAs), we examined the modulation of oxidative DNA damage induced by aflatoxin B1 in PAs-pretreated animals. The effects of PAs on