Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Shahla'a Fadhil Sabir"'
Publikováno v:
Al-Mustansiriyah Journal of Pharmaceutical Sciences, Vol 17, Iss 1 (2018)
Objective: chronic myeloid leukemia (CML) is a myeloproliferative confusion characterized by the occurrence of an acquired mutation which affect the hematopoietic stem cell, treatment of CML with tyrosine kinase inhibitors (TKI) has resulted in high
Externí odkaz:
https://doaj.org/article/0faef6c434bb4f69bbedeac9edd2a0ce
Autor:
Shahla′a Fadhil Sabir
Publikováno v:
Iraqi Journal of Hematology, Vol 5, Iss 2, Pp 178-182 (2016)
Background: Myelofibrosis (MF) is largely documented by an abnormal cytokine expression, which in turn could contribute to bone marrow fibrosis, angiogenesis, and constitutional symptoms. To gain additional pathogenetic insight regarding cytokine phe
Externí odkaz:
https://doaj.org/article/c4c3cb4288754ac8bd1976489b8e37f2
Publikováno v:
Immunogenetics. 75:145-153
The key cell population permits cancer cells to avoid immune-surveillance is regulatory T cells (Tregs). This study evaluates the level of Tregs in chronic myeloid leukemia (CML) patients and the effect of Tyrosine kinase inhibitor (TKI) on Treg leve
Publikováno v:
Medico-Legal Update.
Background: Several clinical trials on cancer showed a correlation between elevated levels of regulatoryT cells with either poor prognosis or poor response to some therapies. Hence, in this study we tried tomeasure the regulatory T cells (CD4+CD245+)
Publikováno v:
International Research Journal of Pharmacy. 8:30-34
Autor:
Shahla'a Fadhil Sabir
Publikováno v:
Iraqi Journal of Hematology, Vol 5, Iss 2, Pp 178-182 (2016)
Background: Myelofibrosis (MF) is largely documented by an abnormal cytokine expression, which in turn could contribute to bone marrow fibrosis, angiogenesis, and constitutional symptoms. To gain additional pathogenetic insight regarding cytokine phe
Publikováno v:
Indian Journal of Hematology and Blood Transfusion. 30:247-252
Chronic myeloid leukemia (CML) is a myeloproliferative disorder characterized by the presence of an acquired mutation which affects the hematopoietic stem cell, leading to a striking overproduction of immature granulocytes. The first important clue t