Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Seth M. Schneider"'
Autor:
Tri Komala Sari, Katrina A. Gianopulos, Darin J. Weed, Seth M. Schneider, Suzanne M. Pritchard, Anthony V. Nicola
Publikováno v:
mSphere, Vol 5, Iss 1 (2020)
ABSTRACT Herpes simplex viruses (HSVs) cause significant morbidity and mortality in humans worldwide. Herpesviruses mediate entry by a multicomponent virus-encoded machinery. Herpesviruses enter cells by endosomal low-pH and pH-neutral mechanisms in
Externí odkaz:
https://doaj.org/article/106fc3ec4ff3483b94f755349a82042b
Autor:
Seth M. Schneider, Suzanne M. Pritchard, George A. Wudiri, Chasity E. Trammell, Anthony V. Nicola
Publikováno v:
mBio, Vol 10, Iss 3 (2019)
ABSTRACT Viruses commandeer host cell 26S proteasome activity to promote viral entry, gene expression, replication, assembly, and egress. Proteasomal degradation activity is critical for herpes simplex virus (HSV) infection. The proteasome inhibitor
Externí odkaz:
https://doaj.org/article/009adb537de94edba977771e626954c3
Publikováno v:
Frontiers in Microbiology, Vol 8 (2017)
Methyl beta-cyclodextrin (MβCD) treatment of herpes simplex virus 1 (HSV-1) reduced envelope cholesterol levels and inhibited viral entry and infectivity in several cell types, regardless of the dependence of entry on endocytosis or low pH. Viral pr
Externí odkaz:
https://doaj.org/article/2c6f08bc83ec4edda9b7bb8e44f37253
Publikováno v:
Cellular microbiologyREFERENCES. 23(2)
Viruses confiscate cellular components of the ubiquitin-proteasome system (UPS) to facilitate many aspects of the infectious cycle. The 26S proteasome is an ATP-dependent, multisubunit proteolytic machine present in all eukaryotic cells. The proteaso
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::21f85396fa70aad9c1c6c472546cc899
https://pubmed.ncbi.nlm.nih.gov/33037857
https://pubmed.ncbi.nlm.nih.gov/33037857
Autor:
Seth M. Schneider, Darin J. Weed, Suzanne M. Pritchard, Anthony V. Nicola, Tri Komala Sari, Katrina A. Gianopulos
Publikováno v:
mSphere, Vol 5, Iss 1, p e00826-19 (2020)
mSphere
mSphere, Vol 5, Iss 1 (2020)
mSphere
mSphere, Vol 5, Iss 1 (2020)
Herpesviruses are ubiquitous pathogens that cause lifelong latent infections and that are characterized by multiple entry pathways. We propose that herpes simplex virus (HSV) gC plays a selective role in modulating HSV entry, such as entry into epith
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b150f0c4003c141383691e266b121685
https://doaj.org/article/7817e654fa684d1d9112c03a705b0e02
https://doaj.org/article/7817e654fa684d1d9112c03a705b0e02
Autor:
Tri Komala Sari, Seth M. Schneider, Darin J. Weed, Suzanne M. Pritchard, Anthony V. Nicola, Katrina A. Gianopulos
Herpes simplex viruses (HSVs) cause significant morbidity and mortality in humans worldwide. Herpesviruses mediate entry by a multi-component, virus-encoded machinery. Herpesviruses enter cells by endosomal low pH and pH-neutral mechanisms in a cell-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a71f3f5d44edeaf29375de43b38f3445
Autor:
Chasity E. Trammell, George A. Wudiri, Anthony V. Nicola, Suzanne M. Pritchard, Seth M. Schneider
Publikováno v:
mBio, Vol 10, Iss 3 (2019)
mBio
mBio, Vol 10, Iss 3, p e00732-19 (2019)
mBio
mBio, Vol 10, Iss 3, p e00732-19 (2019)
Viruses usurp host cell functions to advance their replicative agenda. HSV relies on cellular proteasome activity for successful infection. Proteasome inhibitors, such as MG132, block HSV infection at multiple stages of the infectious cycle. Targetin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::39b4b62998a558c8eceb826aace6f67a
https://journals.asm.org/doi/10.1128/mBio.00732-19
https://journals.asm.org/doi/10.1128/mBio.00732-19
Publikováno v:
Frontiers in Microbiology, Vol 8 (2017)
Frontiers in Microbiology
Frontiers in Microbiology
Methyl beta-cyclodextrin (MβCD) treatment of herpes simplex virus 1 (HSV-1) reduced envelope cholesterol levels and inhibited viral entry and infectivity in several cell types, regardless of the dependence of entry on endocytosis or low pH. Viral pr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dd523099c5930709b2d1c429be408bd0
http://journal.frontiersin.org/article/10.3389/fmicb.2017.02383/full
http://journal.frontiersin.org/article/10.3389/fmicb.2017.02383/full